NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTS

Fabry disease (Anderson–Fabry disease) is an X-linked recessive lysosomal storage disorder resulting from deficient activity of lysosomal hydrolase, alpha-galactosidase A, which leads to progressive accumulation of globotriaosylceramide (Gb3) in various cells (predominantly endothelial and vascular...

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Main Authors: A. S. Kotov, K. V. Firsov
Format: Article
Language:Russian
Published: ABV-press 2017-12-01
Series:Русский журнал детской неврологии
Subjects:
Online Access:https://rjdn.abvpress.ru/jour/article/view/227
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author A. S. Kotov
K. V. Firsov
author_facet A. S. Kotov
K. V. Firsov
author_sort A. S. Kotov
collection DOAJ
description Fabry disease (Anderson–Fabry disease) is an X-linked recessive lysosomal storage disorder resulting from deficient activity of lysosomal hydrolase, alpha-galactosidase A, which leads to progressive accumulation of globotriaosylceramide (Gb3) in various cells (predominantly endothelial and vascular smooth muscle cells) with clinical manifestations affecting major organs including the central nervous system. Clinical onset of Fabry disease usually occurs in childhood, but many patients are diagnosed in adulthood. Early recognition of symptoms, enzyme activity levels, concentration of Gb3 in the blood, urine and skin bioptates, as well as genetic testing (GLA gene) enable establishment of early diagnosis and therapeutic intervention with enzyme replacement therapy. Early therapy may prevent complications of the disease.
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publishDate 2017-12-01
publisher ABV-press
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series Русский журнал детской неврологии
spelling doaj-art-cc6e851ca1cc402992252fd1fe56aabb2025-08-20T02:56:07ZrusABV-pressРусский журнал детской неврологии2073-88032412-91782017-12-01123515710.17650/2073-8803-2017-12-3-51-57160NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTSA. S. Kotov0K. V. Firsov1Moscow Regional Research and Clinical Institute named after M.F. VladimirskiyMoscow Regional Research and Clinical Institute named after M.F. VladimirskiyFabry disease (Anderson–Fabry disease) is an X-linked recessive lysosomal storage disorder resulting from deficient activity of lysosomal hydrolase, alpha-galactosidase A, which leads to progressive accumulation of globotriaosylceramide (Gb3) in various cells (predominantly endothelial and vascular smooth muscle cells) with clinical manifestations affecting major organs including the central nervous system. Clinical onset of Fabry disease usually occurs in childhood, but many patients are diagnosed in adulthood. Early recognition of symptoms, enzyme activity levels, concentration of Gb3 in the blood, urine and skin bioptates, as well as genetic testing (GLA gene) enable establishment of early diagnosis and therapeutic intervention with enzyme replacement therapy. Early therapy may prevent complications of the disease.https://rjdn.abvpress.ru/jour/article/view/227anderson–fabry diseaseneurological manifestationsagalsidase alfaagalsidase beta
spellingShingle A. S. Kotov
K. V. Firsov
NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTS
Русский журнал детской неврологии
anderson–fabry disease
neurological manifestations
agalsidase alfa
agalsidase beta
title NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTS
title_full NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTS
title_fullStr NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTS
title_full_unstemmed NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTS
title_short NEUROLOGICAL MANIFESTATIONS OF FABRY DISEASE IN CHILDREN AND ADOLESCENTS
title_sort neurological manifestations of fabry disease in children and adolescents
topic anderson–fabry disease
neurological manifestations
agalsidase alfa
agalsidase beta
url https://rjdn.abvpress.ru/jour/article/view/227
work_keys_str_mv AT askotov neurologicalmanifestationsoffabrydiseaseinchildrenandadolescents
AT kvfirsov neurologicalmanifestationsoffabrydiseaseinchildrenandadolescents