Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal Cancer

Oxidized low-density lipoprotein (oxLDL) exhibits differential expression in microsatellite-stable (MSS) and microsatellite instability-high (MSI) colorectal cancer (CRC), highlighting its potential therapeutic role in immune checkpoint inhibitor (ICI) resistance in MSS CRC. Elevated oxLDL levels in...

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Main Authors: Xiaochun Zhang, Xiaorui Ye, Heiying Jin
Format: Article
Language:English
Published: MDPI AG 2025-06-01
Series:Antioxidants
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Online Access:https://www.mdpi.com/2076-3921/14/6/726
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author Xiaochun Zhang
Xiaorui Ye
Heiying Jin
author_facet Xiaochun Zhang
Xiaorui Ye
Heiying Jin
author_sort Xiaochun Zhang
collection DOAJ
description Oxidized low-density lipoprotein (oxLDL) exhibits differential expression in microsatellite-stable (MSS) and microsatellite instability-high (MSI) colorectal cancer (CRC), highlighting its potential therapeutic role in immune checkpoint inhibitor (ICI) resistance in MSS CRC. Elevated oxLDL levels in MSS CRC contribute to tumor progression and diminish ICI efficacy by modulating metabolic reprogramming and immunosuppressive mechanisms within the tumor microenvironment (TME) by activating receptors such as LOX-1 and CD36. oxLDL triggers signaling pathways, including NF-κB, PI3K/Akt, and MAPK, leading to the expansion of immunosuppressive cells like regulatory T cells (Tregs), myeloid-derived suppressor cells (MDSCs), and M2 macrophages, while concurrently suppressing effector T cell functions. Additionally, oxLDL enhances oxidative stress and promotes fatty acid oxidation (FAO) and glycolytic metabolism, resulting in nutrient competition within the TME and establishing an immunosuppressive milieu, ultimately culminating in ICI resistance. This review systematically examines the disparities in oxLDL expression between MSS and MSI CRC and elucidates the molecular mechanisms through which oxLDL mediates ICI resistance. Furthermore, it explores potential therapeutic strategies targeting oxLDL, offering novel avenues to overcome immunotherapy resistance in MSS CRC.
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spelling doaj-art-cc6cfcb02b40475a99eb4b6e128e69f92025-08-20T03:27:11ZengMDPI AGAntioxidants2076-39212025-06-0114672610.3390/antiox14060726Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal CancerXiaochun Zhang0Xiaorui Ye1Heiying Jin2The Second Clinical Medical College of Nanjing University of Chinese Medicine, Nanjing 210017, ChinaThe Second Affiliated Hospital of Nanjing University of Chinese Medicine, Nanjing 210017, ChinaThe Second Clinical Medical College of Nanjing University of Chinese Medicine, Nanjing 210017, ChinaOxidized low-density lipoprotein (oxLDL) exhibits differential expression in microsatellite-stable (MSS) and microsatellite instability-high (MSI) colorectal cancer (CRC), highlighting its potential therapeutic role in immune checkpoint inhibitor (ICI) resistance in MSS CRC. Elevated oxLDL levels in MSS CRC contribute to tumor progression and diminish ICI efficacy by modulating metabolic reprogramming and immunosuppressive mechanisms within the tumor microenvironment (TME) by activating receptors such as LOX-1 and CD36. oxLDL triggers signaling pathways, including NF-κB, PI3K/Akt, and MAPK, leading to the expansion of immunosuppressive cells like regulatory T cells (Tregs), myeloid-derived suppressor cells (MDSCs), and M2 macrophages, while concurrently suppressing effector T cell functions. Additionally, oxLDL enhances oxidative stress and promotes fatty acid oxidation (FAO) and glycolytic metabolism, resulting in nutrient competition within the TME and establishing an immunosuppressive milieu, ultimately culminating in ICI resistance. This review systematically examines the disparities in oxLDL expression between MSS and MSI CRC and elucidates the molecular mechanisms through which oxLDL mediates ICI resistance. Furthermore, it explores potential therapeutic strategies targeting oxLDL, offering novel avenues to overcome immunotherapy resistance in MSS CRC.https://www.mdpi.com/2076-3921/14/6/726oxidized low-density lipoproteinmicrosatellite-stable colorectal cancerimmune checkpoint inhibitorstumor microenvironmentmetabolic reprogrammingimmunosuppression
spellingShingle Xiaochun Zhang
Xiaorui Ye
Heiying Jin
Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal Cancer
Antioxidants
oxidized low-density lipoprotein
microsatellite-stable colorectal cancer
immune checkpoint inhibitors
tumor microenvironment
metabolic reprogramming
immunosuppression
title Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal Cancer
title_full Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal Cancer
title_fullStr Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal Cancer
title_full_unstemmed Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal Cancer
title_short Oxidized Low-Density Lipoprotein as a Potential Target for Enhancing Immune Checkpoint Inhibitor Therapy in Microsatellite-Stable Colorectal Cancer
title_sort oxidized low density lipoprotein as a potential target for enhancing immune checkpoint inhibitor therapy in microsatellite stable colorectal cancer
topic oxidized low-density lipoprotein
microsatellite-stable colorectal cancer
immune checkpoint inhibitors
tumor microenvironment
metabolic reprogramming
immunosuppression
url https://www.mdpi.com/2076-3921/14/6/726
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AT xiaoruiye oxidizedlowdensitylipoproteinasapotentialtargetforenhancingimmunecheckpointinhibitortherapyinmicrosatellitestablecolorectalcancer
AT heiyingjin oxidizedlowdensitylipoproteinasapotentialtargetforenhancingimmunecheckpointinhibitortherapyinmicrosatellitestablecolorectalcancer