Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor Expression

ABSTRACT Activin‐A is elevated in pulmonary arterial hypertension (PAH) patients, and reportedly suppresses BMPR‐II. This suggests one mechanism of action for PAH drug, sotatercept, an activin‐ligand trap. However, we were unable to confirm that activin‐A reduces BMPR‐II in pulmonary endothelial cel...

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Main Authors: Benjamin J. Dunmore, Nobuhiro Kikuchi, Wei Li, Paul D. Upton, Nicholas W. Morrell
Format: Article
Language:English
Published: Wiley 2025-04-01
Series:Pulmonary Circulation
Online Access:https://doi.org/10.1002/pul2.70095
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author Benjamin J. Dunmore
Nobuhiro Kikuchi
Wei Li
Paul D. Upton
Nicholas W. Morrell
author_facet Benjamin J. Dunmore
Nobuhiro Kikuchi
Wei Li
Paul D. Upton
Nicholas W. Morrell
author_sort Benjamin J. Dunmore
collection DOAJ
description ABSTRACT Activin‐A is elevated in pulmonary arterial hypertension (PAH) patients, and reportedly suppresses BMPR‐II. This suggests one mechanism of action for PAH drug, sotatercept, an activin‐ligand trap. However, we were unable to confirm that activin‐A reduces BMPR‐II in pulmonary endothelial cells. Thus, it seems unlikely that sotatercept influences BMPR‐II or PAH via this mechanism.
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id doaj-art-cb8bce8fca0a4a3ba353bbee4a5e77db
institution OA Journals
issn 2045-8940
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publishDate 2025-04-01
publisher Wiley
record_format Article
series Pulmonary Circulation
spelling doaj-art-cb8bce8fca0a4a3ba353bbee4a5e77db2025-08-20T02:38:14ZengWileyPulmonary Circulation2045-89402025-04-01152n/an/a10.1002/pul2.70095Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor ExpressionBenjamin J. Dunmore0Nobuhiro Kikuchi1Wei Li2Paul D. Upton3Nicholas W. Morrell4Victor Phillip Dahdaleh Heart and Lung Research Institute University of Cambridge Cambridge UKVictor Phillip Dahdaleh Heart and Lung Research Institute University of Cambridge Cambridge UKVictor Phillip Dahdaleh Heart and Lung Research Institute University of Cambridge Cambridge UKVictor Phillip Dahdaleh Heart and Lung Research Institute University of Cambridge Cambridge UKVictor Phillip Dahdaleh Heart and Lung Research Institute University of Cambridge Cambridge UKABSTRACT Activin‐A is elevated in pulmonary arterial hypertension (PAH) patients, and reportedly suppresses BMPR‐II. This suggests one mechanism of action for PAH drug, sotatercept, an activin‐ligand trap. However, we were unable to confirm that activin‐A reduces BMPR‐II in pulmonary endothelial cells. Thus, it seems unlikely that sotatercept influences BMPR‐II or PAH via this mechanism.https://doi.org/10.1002/pul2.70095
spellingShingle Benjamin J. Dunmore
Nobuhiro Kikuchi
Wei Li
Paul D. Upton
Nicholas W. Morrell
Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor Expression
Pulmonary Circulation
title Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor Expression
title_full Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor Expression
title_fullStr Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor Expression
title_full_unstemmed Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor Expression
title_short Activin‐A Regulates Bone Morphogenetic Protein Signaling in Pulmonary Endothelial Cells Without Affecting Bone Morphogenetic Protein Type‐II Receptor Expression
title_sort activin a regulates bone morphogenetic protein signaling in pulmonary endothelial cells without affecting bone morphogenetic protein type ii receptor expression
url https://doi.org/10.1002/pul2.70095
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