SKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegeneration

Abstract Dopamine (DA) plays an essential role in regulating γ-aminobutyric acid (GABA) neurons in the brain. This study aimed to investigate the effects of DA receptor agonists on the expression of glutamic acid decarboxylase 67 (GAD67) in the context of 6-hydroxydopamine (6-OHDA)-induced dopaminer...

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Main Authors: Sijoon Lee, Munki Kim, Soo-Eun Sung, Gonsup Kim, Chungkil Won
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-11672-w
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author Sijoon Lee
Munki Kim
Soo-Eun Sung
Gonsup Kim
Chungkil Won
author_facet Sijoon Lee
Munki Kim
Soo-Eun Sung
Gonsup Kim
Chungkil Won
author_sort Sijoon Lee
collection DOAJ
description Abstract Dopamine (DA) plays an essential role in regulating γ-aminobutyric acid (GABA) neurons in the brain. This study aimed to investigate the effects of DA receptor agonists on the expression of glutamic acid decarboxylase 67 (GAD67) in the context of 6-hydroxydopamine (6-OHDA)-induced dopaminergic neurodegeneration. To explore the potential involvement of DA receptor agonists in modulating GAD67 expression, these agonists were administered to primary cultured neurons and the substantia nigra in a mouse model with 6-OHDA-induced lesions. The GAD67 expression was subsequently assessed via Western blotting and immunohistochemistry analysis. Results revealed an increased GAD67 expression in in vitro and in vivo models induced by 6-OHDA. Interestingly, treatment with the D1-like receptor agonist SKF38393 led to a decrease in the GAD67 expression. Meanwhile, treatment with the D2-like receptor agonist quinpirole resulted in an increase in the GAD67 expression. Further, the inhibitory effect of SKF38393 on GAD67 was negated when the D1 receptor-selective antagonist LE300 was administered. Conversely, the expression of tyrosine hydroxylase was not affected by the DA receptor agonists SKF38393 or quinpirole. Subsequently, the association between heparanase-1 and the increased expression of GAD67 was examined. The GAD67 and heparanase-1 expression levels increased due to 6-OHDA treatment. However, they decreased when SKF38393 was administered. In contrast, treatment with the heparanase inhibitor suramin led to a reduction in the GAD67 expression. Therefore, the DA D1-like receptor agonist SKF38393 can modulate GAD67 expression under DA degenerative conditions by interacting with heparanase-1.
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spelling doaj-art-cb2df482b0aa4a738215fc9c47616fe82025-08-20T03:42:41ZengNature PortfolioScientific Reports2045-23222025-07-0115111110.1038/s41598-025-11672-wSKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegenerationSijoon Lee0Munki Kim1Soo-Eun Sung2Gonsup Kim3Chungkil Won4Preclinical Research Center, Daegu-Gyeongbuk Medical Innovation FoundationDepartment of Veterinary Medicine & Institute of Animal Medicine, Gyeongsang National UniversityPreclinical Research Center, Daegu-Gyeongbuk Medical Innovation FoundationDepartment of Veterinary Medicine & Institute of Animal Medicine, Gyeongsang National UniversityDepartment of Veterinary Medicine & Institute of Animal Medicine, Gyeongsang National UniversityAbstract Dopamine (DA) plays an essential role in regulating γ-aminobutyric acid (GABA) neurons in the brain. This study aimed to investigate the effects of DA receptor agonists on the expression of glutamic acid decarboxylase 67 (GAD67) in the context of 6-hydroxydopamine (6-OHDA)-induced dopaminergic neurodegeneration. To explore the potential involvement of DA receptor agonists in modulating GAD67 expression, these agonists were administered to primary cultured neurons and the substantia nigra in a mouse model with 6-OHDA-induced lesions. The GAD67 expression was subsequently assessed via Western blotting and immunohistochemistry analysis. Results revealed an increased GAD67 expression in in vitro and in vivo models induced by 6-OHDA. Interestingly, treatment with the D1-like receptor agonist SKF38393 led to a decrease in the GAD67 expression. Meanwhile, treatment with the D2-like receptor agonist quinpirole resulted in an increase in the GAD67 expression. Further, the inhibitory effect of SKF38393 on GAD67 was negated when the D1 receptor-selective antagonist LE300 was administered. Conversely, the expression of tyrosine hydroxylase was not affected by the DA receptor agonists SKF38393 or quinpirole. Subsequently, the association between heparanase-1 and the increased expression of GAD67 was examined. The GAD67 and heparanase-1 expression levels increased due to 6-OHDA treatment. However, they decreased when SKF38393 was administered. In contrast, treatment with the heparanase inhibitor suramin led to a reduction in the GAD67 expression. Therefore, the DA D1-like receptor agonist SKF38393 can modulate GAD67 expression under DA degenerative conditions by interacting with heparanase-1.https://doi.org/10.1038/s41598-025-11672-wDopamine receptorsGAD67SKF38393Heparanase-1Tyrosine hydroxylase
spellingShingle Sijoon Lee
Munki Kim
Soo-Eun Sung
Gonsup Kim
Chungkil Won
SKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegeneration
Scientific Reports
Dopamine receptors
GAD67
SKF38393
Heparanase-1
Tyrosine hydroxylase
title SKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegeneration
title_full SKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegeneration
title_fullStr SKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegeneration
title_full_unstemmed SKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegeneration
title_short SKF-38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase-1 in 6-hydroxydopamine-induced neurodegeneration
title_sort skf 38393 regulates the expression of glutamic acid decarboxylase 67 via heparanase 1 in 6 hydroxydopamine induced neurodegeneration
topic Dopamine receptors
GAD67
SKF38393
Heparanase-1
Tyrosine hydroxylase
url https://doi.org/10.1038/s41598-025-11672-w
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