Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in Immunotherapy

Yuhan Chen,1,* Si Yue,1,* Lingyan Yu,1,* Jinghao Cao,1 Yingchao Liu,1 Aoli Deng,1 Yajuan Lu,1 Jing Yang,1 Huanjuan Li,1 Jing Du,1 Jun Xia,1 Yanchun Li,2 Yongming Xia3 1Laboratory Medicine Center, Department of Clinical Laboratory, Zhejiang Provincial People’s Hospital (Affili...

Full description

Saved in:
Bibliographic Details
Main Authors: Chen Y, Yue S, Yu L, Cao J, Liu Y, Deng A, Lu Y, Yang J, Li H, Du J, Xia J, Li Y, Xia Y
Format: Article
Language:English
Published: Dove Medical Press 2025-03-01
Series:Drug Design, Development and Therapy
Subjects:
Online Access:https://www.dovepress.com/regulation-and-function-of-the-cgas-sting-pathway-mechanisms-post-tran-peer-reviewed-fulltext-article-DDDT
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850035668982431744
author Chen Y
Yue S
Yu L
Cao J
Liu Y
Deng A
Lu Y
Yang J
Li H
Du J
Xia J
Li Y
Xia Y
author_facet Chen Y
Yue S
Yu L
Cao J
Liu Y
Deng A
Lu Y
Yang J
Li H
Du J
Xia J
Li Y
Xia Y
author_sort Chen Y
collection DOAJ
description Yuhan Chen,1,* Si Yue,1,* Lingyan Yu,1,* Jinghao Cao,1 Yingchao Liu,1 Aoli Deng,1 Yajuan Lu,1 Jing Yang,1 Huanjuan Li,1 Jing Du,1 Jun Xia,1 Yanchun Li,2 Yongming Xia3 1Laboratory Medicine Center, Department of Clinical Laboratory, Zhejiang Provincial People’s Hospital (Affiliated People’s Hospital), Hangzhou Medical College, Hangzhou, Zhejiang, People’s Republic of China; 2Department of Clinical Laboratory, Affiliated Hangzhou First People’s Hospital, School of Medicine, Westlake University, Hangzhou, Zhejiang, People’s Republic of China; 3Department of Hematology, Yuyao People’s Hospital, Yuyao, Zhejiang, People’s Republic of China*These authors contributed equally to this workCorrespondence: Jun Xia, Email andisky_005@126.com Yongming Xia, Email xym20246@163.comAbstract: Autoimmune diseases arise when the immune system attacks healthy tissues, losing tolerance for self-tissues. Normally, the immune system recognizes and defends against pathogens like bacteria and viruses. The cGAS-STING pathway, activated by pattern-recognition receptors (PRRs), plays a key role in autoimmune responses. The cGAS protein senses pathogenic DNA and synthesizes cGAMP, which induces conformational changes in STING, activating kinases IKK and TBK1 and leading to the expression of interferon genes or inflammatory mediators. This pathway is crucial in immunotherapy, activating innate immunity, enhancing antigen presentation, modulating the tumor microenvironment, and integrating into therapeutic strategies. Modulation strategies include small molecule inhibitors, oligonucleotide therapies, protein and antibody therapies, genetic and epigenetic regulation, cytokine and metabolite modulation, and nanoscale delivery systems. Post-translational modifications (PTMs) of the cGAS-STING pathway, such as phosphorylation, acetylation, ubiquitination, methylation, palmitoylation, and glycosylation, fine-tune immune responses by regulating protein activity, stability, localization, and interactions. These modifications are interconnected and collectively influence pathway functionality. We summarize the functions of cGAS-STING and its PTMs in immune and non-immune cells across various diseases, and explore potential clinical applications.Keywords: cGAS, STING, immunity, immunotherapy, post-transcriptional regulation
format Article
id doaj-art-ca508a59698d4a16b59bbb73f67d5350
institution DOAJ
issn 1177-8881
language English
publishDate 2025-03-01
publisher Dove Medical Press
record_format Article
series Drug Design, Development and Therapy
spelling doaj-art-ca508a59698d4a16b59bbb73f67d53502025-08-20T02:57:25ZengDove Medical PressDrug Design, Development and Therapy1177-88812025-03-01Volume 1917211739100997Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in ImmunotherapyChen YYue SYu LCao JLiu YDeng ALu YYang JLi HDu JXia JLi YXia YYuhan Chen,1,* Si Yue,1,* Lingyan Yu,1,* Jinghao Cao,1 Yingchao Liu,1 Aoli Deng,1 Yajuan Lu,1 Jing Yang,1 Huanjuan Li,1 Jing Du,1 Jun Xia,1 Yanchun Li,2 Yongming Xia3 1Laboratory Medicine Center, Department of Clinical Laboratory, Zhejiang Provincial People’s Hospital (Affiliated People’s Hospital), Hangzhou Medical College, Hangzhou, Zhejiang, People’s Republic of China; 2Department of Clinical Laboratory, Affiliated Hangzhou First People’s Hospital, School of Medicine, Westlake University, Hangzhou, Zhejiang, People’s Republic of China; 3Department of Hematology, Yuyao People’s Hospital, Yuyao, Zhejiang, People’s Republic of China*These authors contributed equally to this workCorrespondence: Jun Xia, Email andisky_005@126.com Yongming Xia, Email xym20246@163.comAbstract: Autoimmune diseases arise when the immune system attacks healthy tissues, losing tolerance for self-tissues. Normally, the immune system recognizes and defends against pathogens like bacteria and viruses. The cGAS-STING pathway, activated by pattern-recognition receptors (PRRs), plays a key role in autoimmune responses. The cGAS protein senses pathogenic DNA and synthesizes cGAMP, which induces conformational changes in STING, activating kinases IKK and TBK1 and leading to the expression of interferon genes or inflammatory mediators. This pathway is crucial in immunotherapy, activating innate immunity, enhancing antigen presentation, modulating the tumor microenvironment, and integrating into therapeutic strategies. Modulation strategies include small molecule inhibitors, oligonucleotide therapies, protein and antibody therapies, genetic and epigenetic regulation, cytokine and metabolite modulation, and nanoscale delivery systems. Post-translational modifications (PTMs) of the cGAS-STING pathway, such as phosphorylation, acetylation, ubiquitination, methylation, palmitoylation, and glycosylation, fine-tune immune responses by regulating protein activity, stability, localization, and interactions. These modifications are interconnected and collectively influence pathway functionality. We summarize the functions of cGAS-STING and its PTMs in immune and non-immune cells across various diseases, and explore potential clinical applications.Keywords: cGAS, STING, immunity, immunotherapy, post-transcriptional regulationhttps://www.dovepress.com/regulation-and-function-of-the-cgas-sting-pathway-mechanisms-post-tran-peer-reviewed-fulltext-article-DDDTcgasstingimmunityimmunotherapypost-transcriptional regulation
spellingShingle Chen Y
Yue S
Yu L
Cao J
Liu Y
Deng A
Lu Y
Yang J
Li H
Du J
Xia J
Li Y
Xia Y
Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in Immunotherapy
Drug Design, Development and Therapy
cgas
sting
immunity
immunotherapy
post-transcriptional regulation
title Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in Immunotherapy
title_full Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in Immunotherapy
title_fullStr Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in Immunotherapy
title_full_unstemmed Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in Immunotherapy
title_short Regulation and Function of the cGAS-STING Pathway: Mechanisms, Post-Translational Modifications, and Therapeutic Potential in Immunotherapy
title_sort regulation and function of the cgas sting pathway mechanisms post translational modifications and therapeutic potential in immunotherapy
topic cgas
sting
immunity
immunotherapy
post-transcriptional regulation
url https://www.dovepress.com/regulation-and-function-of-the-cgas-sting-pathway-mechanisms-post-tran-peer-reviewed-fulltext-article-DDDT
work_keys_str_mv AT cheny regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT yues regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT yul regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT caoj regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT liuy regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT denga regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT luy regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT yangj regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT lih regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT duj regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT xiaj regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT liy regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy
AT xiay regulationandfunctionofthecgasstingpathwaymechanismsposttranslationalmodificationsandtherapeuticpotentialinimmunotherapy