LXR pathway drives hormonal response intensity in polycystic ovary syndrome

Abstract Gonadotropin injections used to stimulate oocyte production during assisted reproductive technology (ART) procedures are associated with the risk of an abnormal response in predisposed patients suffering polycystic ovary syndrome (PCOS). Liver X receptors (LXR) pathway has been identified a...

Full description

Saved in:
Bibliographic Details
Main Authors: Sarah Dallel, Manon Despalles, Margaux Tore, Yoan Renaud, Ayhan Kocer, Christelle Damon-Soubeyrand, Pierre Pouchin, Caroline Vachias, Katia Boutourlinsky, Céline Gonthier-Gueret, Angélique De Haze, Phelipe Sanchez, Jean-Christophe Pointud, Erwan Bouchareb, Marine Vialat, Aurélie Lagarde, Cristina Gulunga, Laure Chaput, Aurélie Vega, Florence Brugnon, Igor Tauveron, Amalia Trousson, Cyrille de Joussineau, Françoise Degoul, Laurent Morel, Jean Marc Lobaccaro, Salwan Maqdasy, Silvère Baron
Format: Article
Language:English
Published: Springer Nature 2025-05-01
Series:EMBO Molecular Medicine
Subjects:
Online Access:https://doi.org/10.1038/s44321-025-00251-1
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849331704934694912
author Sarah Dallel
Manon Despalles
Margaux Tore
Yoan Renaud
Ayhan Kocer
Christelle Damon-Soubeyrand
Pierre Pouchin
Caroline Vachias
Katia Boutourlinsky
Céline Gonthier-Gueret
Angélique De Haze
Phelipe Sanchez
Jean-Christophe Pointud
Erwan Bouchareb
Marine Vialat
Aurélie Lagarde
Cristina Gulunga
Laure Chaput
Aurélie Vega
Florence Brugnon
Igor Tauveron
Amalia Trousson
Cyrille de Joussineau
Françoise Degoul
Laurent Morel
Jean Marc Lobaccaro
Salwan Maqdasy
Silvère Baron
author_facet Sarah Dallel
Manon Despalles
Margaux Tore
Yoan Renaud
Ayhan Kocer
Christelle Damon-Soubeyrand
Pierre Pouchin
Caroline Vachias
Katia Boutourlinsky
Céline Gonthier-Gueret
Angélique De Haze
Phelipe Sanchez
Jean-Christophe Pointud
Erwan Bouchareb
Marine Vialat
Aurélie Lagarde
Cristina Gulunga
Laure Chaput
Aurélie Vega
Florence Brugnon
Igor Tauveron
Amalia Trousson
Cyrille de Joussineau
Françoise Degoul
Laurent Morel
Jean Marc Lobaccaro
Salwan Maqdasy
Silvère Baron
author_sort Sarah Dallel
collection DOAJ
description Abstract Gonadotropin injections used to stimulate oocyte production during assisted reproductive technology (ART) procedures are associated with the risk of an abnormal response in predisposed patients suffering polycystic ovary syndrome (PCOS). Liver X receptors (LXR) pathway has been identified as key regulators during this process. This study explores the integration of the hormonal signals, cellular networks and molecular mechanisms linking sterol signaling with inflammation and immune infiltration. Pharmacological activation of LXR in a wild-type context protects against gonadotropin hyperstimulation mirroring the effect observed in LXR-deficient mice. Ovarian stimulation leads to immune cell infiltration orchestrated by granulosa cells in absence of LXR, resulting in an altered granulosa cell response to gonadotropin and enhanced inflammation. LXR controls inflammasome activity by regulating Thioredoxin Interacting Protein (TXNIP) gene expression in mural granulosa cells, thereby modulating IL1β production. This immune cell infiltration persists throughout ovulation in PCOS patients and is observed in cumulus oocytes complexes, highlighting the pivotal role of LXR path in regulating inflammatory processes during hormonal stimulation in ART procedures.
format Article
id doaj-art-c91c46cf60de48f184a90160fceb7a8a
institution Kabale University
issn 1757-4684
language English
publishDate 2025-05-01
publisher Springer Nature
record_format Article
series EMBO Molecular Medicine
spelling doaj-art-c91c46cf60de48f184a90160fceb7a8a2025-08-20T03:46:25ZengSpringer NatureEMBO Molecular Medicine1757-46842025-05-011771666168510.1038/s44321-025-00251-1LXR pathway drives hormonal response intensity in polycystic ovary syndromeSarah Dallel0Manon Despalles1Margaux Tore2Yoan Renaud3Ayhan Kocer4Christelle Damon-Soubeyrand5Pierre Pouchin6Caroline Vachias7Katia Boutourlinsky8Céline Gonthier-Gueret9Angélique De Haze10Phelipe Sanchez11Jean-Christophe Pointud12Erwan Bouchareb13Marine Vialat14Aurélie Lagarde15Cristina Gulunga16Laure Chaput17Aurélie Vega18Florence Brugnon19Igor Tauveron20Amalia Trousson21Cyrille de Joussineau22Françoise Degoul23Laurent Morel24Jean Marc Lobaccaro25Salwan Maqdasy26Silvère Baron27Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Assistance Médicale à la Procréation, CECOS, CHU Clermont-FerrandAssistance Médicale à la Procréation, CECOS, CHU Clermont-FerrandAssistance Médicale à la Procréation, CECOS, CHU Clermont-FerrandCentre de Recherche en Nutrition Humaine d’AuvergneService d’Endocrinologie, Diabétologie et Maladies Métaboliques, CHU Clermont Ferrand, Hôpital Gabriel MontpiedUniversité Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Université Clermont Auvergne, iGReD, CNRS UMR 6293, INSERM U1103Abstract Gonadotropin injections used to stimulate oocyte production during assisted reproductive technology (ART) procedures are associated with the risk of an abnormal response in predisposed patients suffering polycystic ovary syndrome (PCOS). Liver X receptors (LXR) pathway has been identified as key regulators during this process. This study explores the integration of the hormonal signals, cellular networks and molecular mechanisms linking sterol signaling with inflammation and immune infiltration. Pharmacological activation of LXR in a wild-type context protects against gonadotropin hyperstimulation mirroring the effect observed in LXR-deficient mice. Ovarian stimulation leads to immune cell infiltration orchestrated by granulosa cells in absence of LXR, resulting in an altered granulosa cell response to gonadotropin and enhanced inflammation. LXR controls inflammasome activity by regulating Thioredoxin Interacting Protein (TXNIP) gene expression in mural granulosa cells, thereby modulating IL1β production. This immune cell infiltration persists throughout ovulation in PCOS patients and is observed in cumulus oocytes complexes, highlighting the pivotal role of LXR path in regulating inflammatory processes during hormonal stimulation in ART procedures.https://doi.org/10.1038/s44321-025-00251-1Polycystic Ovary SyndromeGranulosa CellsLiver X ReceptorsHormonal StimulationInflammasome
spellingShingle Sarah Dallel
Manon Despalles
Margaux Tore
Yoan Renaud
Ayhan Kocer
Christelle Damon-Soubeyrand
Pierre Pouchin
Caroline Vachias
Katia Boutourlinsky
Céline Gonthier-Gueret
Angélique De Haze
Phelipe Sanchez
Jean-Christophe Pointud
Erwan Bouchareb
Marine Vialat
Aurélie Lagarde
Cristina Gulunga
Laure Chaput
Aurélie Vega
Florence Brugnon
Igor Tauveron
Amalia Trousson
Cyrille de Joussineau
Françoise Degoul
Laurent Morel
Jean Marc Lobaccaro
Salwan Maqdasy
Silvère Baron
LXR pathway drives hormonal response intensity in polycystic ovary syndrome
EMBO Molecular Medicine
Polycystic Ovary Syndrome
Granulosa Cells
Liver X Receptors
Hormonal Stimulation
Inflammasome
title LXR pathway drives hormonal response intensity in polycystic ovary syndrome
title_full LXR pathway drives hormonal response intensity in polycystic ovary syndrome
title_fullStr LXR pathway drives hormonal response intensity in polycystic ovary syndrome
title_full_unstemmed LXR pathway drives hormonal response intensity in polycystic ovary syndrome
title_short LXR pathway drives hormonal response intensity in polycystic ovary syndrome
title_sort lxr pathway drives hormonal response intensity in polycystic ovary syndrome
topic Polycystic Ovary Syndrome
Granulosa Cells
Liver X Receptors
Hormonal Stimulation
Inflammasome
url https://doi.org/10.1038/s44321-025-00251-1
work_keys_str_mv AT sarahdallel lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT manondespalles lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT margauxtore lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT yoanrenaud lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT ayhankocer lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT christelledamonsoubeyrand lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT pierrepouchin lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT carolinevachias lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT katiaboutourlinsky lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT celinegonthiergueret lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT angeliquedehaze lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT phelipesanchez lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT jeanchristophepointud lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT erwanbouchareb lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT marinevialat lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT aurelielagarde lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT cristinagulunga lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT laurechaput lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT aurelievega lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT florencebrugnon lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT igortauveron lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT amaliatrousson lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT cyrilledejoussineau lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT francoisedegoul lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT laurentmorel lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT jeanmarclobaccaro lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT salwanmaqdasy lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome
AT silverebaron lxrpathwaydriveshormonalresponseintensityinpolycysticovarysyndrome