Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.

To identify new regulators of antiviral innate immunity, we completed the first genome-wide gene silencing screen assessing the transcriptional response at the interferon-β (IFNB1) promoter following Sendai virus (SeV) infection. We now report a novel link between WNT signaling pathway and the modul...

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Main Authors: Martin Baril, Salwa Es-Saad, Laurent Chatel-Chaix, Karin Fink, Tram Pham, Valérie-Ann Raymond, Karine Audette, Anne-Sophie Guenier, Jean Duchaine, Marc Servant, Marc Bilodeau, Eric Cohen, Nathalie Grandvaux, Daniel Lamarre
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2013-01-01
Series:PLoS Pathogens
Online Access:https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1003416&type=printable
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author Martin Baril
Salwa Es-Saad
Laurent Chatel-Chaix
Karin Fink
Tram Pham
Valérie-Ann Raymond
Karine Audette
Anne-Sophie Guenier
Jean Duchaine
Marc Servant
Marc Bilodeau
Eric Cohen
Nathalie Grandvaux
Daniel Lamarre
author_facet Martin Baril
Salwa Es-Saad
Laurent Chatel-Chaix
Karin Fink
Tram Pham
Valérie-Ann Raymond
Karine Audette
Anne-Sophie Guenier
Jean Duchaine
Marc Servant
Marc Bilodeau
Eric Cohen
Nathalie Grandvaux
Daniel Lamarre
author_sort Martin Baril
collection DOAJ
description To identify new regulators of antiviral innate immunity, we completed the first genome-wide gene silencing screen assessing the transcriptional response at the interferon-β (IFNB1) promoter following Sendai virus (SeV) infection. We now report a novel link between WNT signaling pathway and the modulation of retinoic acid-inducible gene I (RIG-I)-like receptor (RLR)-dependent innate immune responses. Here we show that secretion of WNT2B and WNT9B and stabilization of β-catenin (CTNNB1) upon virus infection negatively regulate expression of representative inducible genes IFNB1, IFIT1 and TNF in a CTNNB1-dependent effector mechanism. The antiviral response is drastically reduced by glycogen synthase kinase 3 (GSK3) inhibitors but restored in CTNNB1 knockdown cells. The findings confirm a novel regulation of antiviral innate immunity by a canonical-like WNT/CTNNB1 signaling pathway. The study identifies novel avenues for broad-spectrum antiviral targets and preventing immune-mediated diseases upon viral infection.
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id doaj-art-c8b8b337593f48b0be8e972cb125c37c
institution Kabale University
issn 1553-7366
1553-7374
language English
publishDate 2013-01-01
publisher Public Library of Science (PLoS)
record_format Article
series PLoS Pathogens
spelling doaj-art-c8b8b337593f48b0be8e972cb125c37c2025-08-20T03:26:48ZengPublic Library of Science (PLoS)PLoS Pathogens1553-73661553-73742013-01-0196e100341610.1371/journal.ppat.1003416Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.Martin BarilSalwa Es-SaadLaurent Chatel-ChaixKarin FinkTram PhamValérie-Ann RaymondKarine AudetteAnne-Sophie GuenierJean DuchaineMarc ServantMarc BilodeauEric CohenNathalie GrandvauxDaniel LamarreTo identify new regulators of antiviral innate immunity, we completed the first genome-wide gene silencing screen assessing the transcriptional response at the interferon-β (IFNB1) promoter following Sendai virus (SeV) infection. We now report a novel link between WNT signaling pathway and the modulation of retinoic acid-inducible gene I (RIG-I)-like receptor (RLR)-dependent innate immune responses. Here we show that secretion of WNT2B and WNT9B and stabilization of β-catenin (CTNNB1) upon virus infection negatively regulate expression of representative inducible genes IFNB1, IFIT1 and TNF in a CTNNB1-dependent effector mechanism. The antiviral response is drastically reduced by glycogen synthase kinase 3 (GSK3) inhibitors but restored in CTNNB1 knockdown cells. The findings confirm a novel regulation of antiviral innate immunity by a canonical-like WNT/CTNNB1 signaling pathway. The study identifies novel avenues for broad-spectrum antiviral targets and preventing immune-mediated diseases upon viral infection.https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1003416&type=printable
spellingShingle Martin Baril
Salwa Es-Saad
Laurent Chatel-Chaix
Karin Fink
Tram Pham
Valérie-Ann Raymond
Karine Audette
Anne-Sophie Guenier
Jean Duchaine
Marc Servant
Marc Bilodeau
Eric Cohen
Nathalie Grandvaux
Daniel Lamarre
Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.
PLoS Pathogens
title Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.
title_full Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.
title_fullStr Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.
title_full_unstemmed Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.
title_short Genome-wide RNAi screen reveals a new role of a WNT/CTNNB1 signaling pathway as negative regulator of virus-induced innate immune responses.
title_sort genome wide rnai screen reveals a new role of a wnt ctnnb1 signaling pathway as negative regulator of virus induced innate immune responses
url https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1003416&type=printable
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