Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammation

Serum and BALF (bronchoalveolar lavage fluid) IL-8 levels and serum levels were investigated in Tox ocara canis infected guinea-pigs and the role of IL-5 as a modulator of cytokine secretion was studied. Serum levels increased early in infected animals, exceeding control levels 4 h after infection,...

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Main Authors: L. H. Faccioli, A. I. Medeiros, A. Malheiro, R. C. L. R. Pietro, A. Januário, B. B. Vargaftig
Format: Article
Language:English
Published: Wiley 1998-01-01
Series:Mediators of Inflammation
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Online Access:http://dx.doi.org/10.1080/09629359891379
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author L. H. Faccioli
A. I. Medeiros
A. Malheiro
R. C. L. R. Pietro
A. Januário
B. B. Vargaftig
author_facet L. H. Faccioli
A. I. Medeiros
A. Malheiro
R. C. L. R. Pietro
A. Januário
B. B. Vargaftig
author_sort L. H. Faccioli
collection DOAJ
description Serum and BALF (bronchoalveolar lavage fluid) IL-8 levels and serum levels were investigated in Tox ocara canis infected guinea-pigs and the role of IL-5 as a modulator of cytokine secretion was studied. Serum levels increased early in infected animals, exceeding control levels 4 h after infection, peaked between days 6 and 18, and continued to exceed control levels after 48 days of infection. Serum and BALF IL-8 levels showed the same profile as blood eosinophilia, increasing 6 days post-infection and peaking between days 18 and 24. Treatment of infected animals with anti-IL-5 Ab suppressed eosinophilia with a parallel increase in blood IL-8 levels, whereas no change was found in levels. To support our in vivo observation we carried out experiments in vitro using guinea-pig LPS-stimulated adherent peritoneal cells which release large amounts of IL-8 into the supernatants. When rIL-5 was added to LPS-stimulated cells, 65% inhibition of IL-8 release into the supernatants was observed. Pre-incubation of cells with anti-IL-5 Ab prevented the inhibition of IL-8 release into the supernatants induced by rIL-5. Our results demonstrate for the first time that TNF- α and IL-8 are released concomitant with or after IL-5 in the eosinophilic inflammation induced by T. canis . Moreover, in addition to showing that IL-5 is fundamental for the induction of blood eosinophilia, the present results suggest that this cytokine may play a new biological role by acting as modulator of IL-8 secretion.
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spelling doaj-art-c79c2b6ef1f547859e55f0025bf2ecb22025-08-20T03:05:15ZengWileyMediators of Inflammation0962-93511466-18611998-01-0171414710.1080/09629359891379Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammationL. H. Faccioli0A. I. Medeiros1A. Malheiro2R. C. L. R. Pietro3A. Januário4B. B. Vargaftig5Department of Parasitology, Microbiology and Immunology, School of Medicine of Ribeirão Preto, University of São Paulo, Ribeirão Preto, SP, BrazilDepartment of Parasitology, Microbiology and Immunology, School of Medicine of Ribeirão Preto, University of São Paulo, Ribeirão Preto, SP, BrazilDepartment of Parasitology, Microbiology and Immunology, School of Medicine of Ribeirão Preto, University of São Paulo, Ribeirão Preto, SP, BrazilDepartment of Parasitology, Microbiology and Immunology, School of Medicine of Ribeirão Preto, University of São Paulo, Ribeirão Preto, SP, BrazilDepartment of Parasitology, Microbiology and Immunology, School of Medicine of Ribeirão Preto, University of São Paulo, Ribeirão Preto, SP, BrazilUnité de Pharmacologie Cellulaire, Unité Associée Institut Pasteur/INSERM n. 285, Paris, FranceSerum and BALF (bronchoalveolar lavage fluid) IL-8 levels and serum levels were investigated in Tox ocara canis infected guinea-pigs and the role of IL-5 as a modulator of cytokine secretion was studied. Serum levels increased early in infected animals, exceeding control levels 4 h after infection, peaked between days 6 and 18, and continued to exceed control levels after 48 days of infection. Serum and BALF IL-8 levels showed the same profile as blood eosinophilia, increasing 6 days post-infection and peaking between days 18 and 24. Treatment of infected animals with anti-IL-5 Ab suppressed eosinophilia with a parallel increase in blood IL-8 levels, whereas no change was found in levels. To support our in vivo observation we carried out experiments in vitro using guinea-pig LPS-stimulated adherent peritoneal cells which release large amounts of IL-8 into the supernatants. When rIL-5 was added to LPS-stimulated cells, 65% inhibition of IL-8 release into the supernatants was observed. Pre-incubation of cells with anti-IL-5 Ab prevented the inhibition of IL-8 release into the supernatants induced by rIL-5. Our results demonstrate for the first time that TNF- α and IL-8 are released concomitant with or after IL-5 in the eosinophilic inflammation induced by T. canis . Moreover, in addition to showing that IL-5 is fundamental for the induction of blood eosinophilia, the present results suggest that this cytokine may play a new biological role by acting as modulator of IL-8 secretion.http://dx.doi.org/10.1080/09629359891379InterleukinsEosinophilsInflammationToxocara canis .
spellingShingle L. H. Faccioli
A. I. Medeiros
A. Malheiro
R. C. L. R. Pietro
A. Januário
B. B. Vargaftig
Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammation
Mediators of Inflammation
Interleukins
Eosinophils
Inflammation
Toxocara canis .
title Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammation
title_full Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammation
title_fullStr Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammation
title_full_unstemmed Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammation
title_short Interleukin-5 modulates interleukin-8 secretion in eosinophilic inflammation
title_sort interleukin 5 modulates interleukin 8 secretion in eosinophilic inflammation
topic Interleukins
Eosinophils
Inflammation
Toxocara canis .
url http://dx.doi.org/10.1080/09629359891379
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