Adipokine Contribution to the Pathogenesis of Osteoarthritis

Recent studies have shown that overweight and obesity play an important role in the development of osteoarthritis (OA). However, joint overload is not the only risk factor in this disease. For instance, the presence of OA in non-weight-bearing joints such as the hand suggests that metabolic factors...

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Main Authors: Daniel Azamar-Llamas, Gabriela Hernández-Molina, Bárbara Ramos-Ávalos, Janette Furuzawa-Carballeda
Format: Article
Language:English
Published: Wiley 2017-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2017/5468023
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author Daniel Azamar-Llamas
Gabriela Hernández-Molina
Bárbara Ramos-Ávalos
Janette Furuzawa-Carballeda
author_facet Daniel Azamar-Llamas
Gabriela Hernández-Molina
Bárbara Ramos-Ávalos
Janette Furuzawa-Carballeda
author_sort Daniel Azamar-Llamas
collection DOAJ
description Recent studies have shown that overweight and obesity play an important role in the development of osteoarthritis (OA). However, joint overload is not the only risk factor in this disease. For instance, the presence of OA in non-weight-bearing joints such as the hand suggests that metabolic factors may also contribute to its pathogenesis. Recently, white adipose tissue (WAT) has been recognized not only as an energy reservoir but also as an important secretory organ of adipokines. In this regard, adipokines have been closely associated with obesity and also play an important role in bone and cartilage homeostasis. Furthermore, drugs such as rosuvastatin or rosiglitazone have demonstrated chondroprotective and anti-inflammatory effects in cartilage explants from patients with OA. Thus, it seems that adipokines are important factors linking obesity, adiposity, and inflammation in OA. In this review, we are focused on establishing the physiological mechanisms of adipokines on cartilage homeostasis and evaluating their role in the pathophysiology of OA based on evidence derived from experimental research as well as from clinical-epidemiological studies.
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series Mediators of Inflammation
spelling doaj-art-c6c0f1313e914dd3aa7a7c6acbeef5f22025-08-20T02:19:44ZengWileyMediators of Inflammation0962-93511466-18612017-01-01201710.1155/2017/54680235468023Adipokine Contribution to the Pathogenesis of OsteoarthritisDaniel Azamar-Llamas0Gabriela Hernández-Molina1Bárbara Ramos-Ávalos2Janette Furuzawa-Carballeda3Department of Immunology and Rheumatology, Instituto Nacional de Ciencias Médicas y Nutrición, Salvador Zubirán, Vasco de Quiroga No. 15, Col. Belisario Domínguez, Sección XVI, 14080 Mexico City, MexicoDepartment of Immunology and Rheumatology, Instituto Nacional de Ciencias Médicas y Nutrición, Salvador Zubirán, Vasco de Quiroga No. 15, Col. Belisario Domínguez, Sección XVI, 14080 Mexico City, MexicoDepartment of Immunology and Rheumatology, Instituto Nacional de Ciencias Médicas y Nutrición, Salvador Zubirán, Vasco de Quiroga No. 15, Col. Belisario Domínguez, Sección XVI, 14080 Mexico City, MexicoDepartment of Immunology and Rheumatology, Instituto Nacional de Ciencias Médicas y Nutrición, Salvador Zubirán, Vasco de Quiroga No. 15, Col. Belisario Domínguez, Sección XVI, 14080 Mexico City, MexicoRecent studies have shown that overweight and obesity play an important role in the development of osteoarthritis (OA). However, joint overload is not the only risk factor in this disease. For instance, the presence of OA in non-weight-bearing joints such as the hand suggests that metabolic factors may also contribute to its pathogenesis. Recently, white adipose tissue (WAT) has been recognized not only as an energy reservoir but also as an important secretory organ of adipokines. In this regard, adipokines have been closely associated with obesity and also play an important role in bone and cartilage homeostasis. Furthermore, drugs such as rosuvastatin or rosiglitazone have demonstrated chondroprotective and anti-inflammatory effects in cartilage explants from patients with OA. Thus, it seems that adipokines are important factors linking obesity, adiposity, and inflammation in OA. In this review, we are focused on establishing the physiological mechanisms of adipokines on cartilage homeostasis and evaluating their role in the pathophysiology of OA based on evidence derived from experimental research as well as from clinical-epidemiological studies.http://dx.doi.org/10.1155/2017/5468023
spellingShingle Daniel Azamar-Llamas
Gabriela Hernández-Molina
Bárbara Ramos-Ávalos
Janette Furuzawa-Carballeda
Adipokine Contribution to the Pathogenesis of Osteoarthritis
Mediators of Inflammation
title Adipokine Contribution to the Pathogenesis of Osteoarthritis
title_full Adipokine Contribution to the Pathogenesis of Osteoarthritis
title_fullStr Adipokine Contribution to the Pathogenesis of Osteoarthritis
title_full_unstemmed Adipokine Contribution to the Pathogenesis of Osteoarthritis
title_short Adipokine Contribution to the Pathogenesis of Osteoarthritis
title_sort adipokine contribution to the pathogenesis of osteoarthritis
url http://dx.doi.org/10.1155/2017/5468023
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AT gabrielahernandezmolina adipokinecontributiontothepathogenesisofosteoarthritis
AT barbararamosavalos adipokinecontributiontothepathogenesisofosteoarthritis
AT janettefuruzawacarballeda adipokinecontributiontothepathogenesisofosteoarthritis