Non-epileptic myoclonus and mitochondrial encephalomyopathy

Two brothers presented to us with a progressive myoclonic syndrome with slight cerebellar symptoms. Neurological examination disclosed moderate cerebellar signs and pale optic discs; asymmetric, asynchronous and arhythmic myoclonus, an arthresthesic deficit and no muscular weakness. EEG background a...

Full description

Saved in:
Bibliographic Details
Main Authors: A. Cukiert, F. G. M. Naylor, H. B. Scapolan, M. M. Vilela, F. S. Aloe, J. O. Siffert, Ana M. Tsanaglis, Monica Haddad, Teresa C. Machado, Mary Carvalho-Alegro, J. A. Levy, L. Marques-Assis
Format: Article
Language:English
Published: Thieme Revinter Publicações 1989-09-01
Series:Arquivos de Neuro-Psiquiatria
Online Access:http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X1989000300016&lng=en&tlng=en
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849686476190646272
author A. Cukiert
F. G. M. Naylor
H. B. Scapolan
M. M. Vilela
F. S. Aloe
J. O. Siffert
Ana M. Tsanaglis
Monica Haddad
Teresa C. Machado
Mary Carvalho-Alegro
J. A. Levy
L. Marques-Assis
author_facet A. Cukiert
F. G. M. Naylor
H. B. Scapolan
M. M. Vilela
F. S. Aloe
J. O. Siffert
Ana M. Tsanaglis
Monica Haddad
Teresa C. Machado
Mary Carvalho-Alegro
J. A. Levy
L. Marques-Assis
author_sort A. Cukiert
collection DOAJ
description Two brothers presented to us with a progressive myoclonic syndrome with slight cerebellar symptoms. Neurological examination disclosed moderate cerebellar signs and pale optic discs; asymmetric, asynchronous and arhythmic myoclonus, an arthresthesic deficit and no muscular weakness. EEG background activity was moderately slow with no irritative discharges. CT was normal in both cases, Intermitent photic stimulation increased the frequency of the myoclonic jerks, which became bilateral and synchronous, progressing to a generalized tonic-clonic seizure. EPs and MRI in one case were normal. Anticonvulsant drugs were ineffective. The diagnosis of mitochondrial encephalomyopathy was based on the finding, in muscle specimens, of thickened basement membranes with myofibrillary degeneration and increased number of mitochondria peripherally distributed and with a dense granular matrix and some vacuoles. The clinical and EEG data suggest a. subcortical origin for this type of myoclonic syndrome.
format Article
id doaj-art-c56b778804ab416f9cece5077ddff461
institution DOAJ
issn 1678-4227
language English
publishDate 1989-09-01
publisher Thieme Revinter Publicações
record_format Article
series Arquivos de Neuro-Psiquiatria
spelling doaj-art-c56b778804ab416f9cece5077ddff4612025-08-20T03:22:42ZengThieme Revinter PublicaçõesArquivos de Neuro-Psiquiatria1678-42271989-09-0147334635110.1590/S0004-282X1989000300016S0004-282X1989000300016Non-epileptic myoclonus and mitochondrial encephalomyopathyA. Cukiert0F. G. M. Naylor1H. B. Scapolan2M. M. Vilela3F. S. Aloe4J. O. Siffert5Ana M. Tsanaglis6Monica HaddadTeresa C. Machado7Mary Carvalho-Alegro8J. A. Levy9L. Marques-Assis10Universidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloUniversidade de São PauloTwo brothers presented to us with a progressive myoclonic syndrome with slight cerebellar symptoms. Neurological examination disclosed moderate cerebellar signs and pale optic discs; asymmetric, asynchronous and arhythmic myoclonus, an arthresthesic deficit and no muscular weakness. EEG background activity was moderately slow with no irritative discharges. CT was normal in both cases, Intermitent photic stimulation increased the frequency of the myoclonic jerks, which became bilateral and synchronous, progressing to a generalized tonic-clonic seizure. EPs and MRI in one case were normal. Anticonvulsant drugs were ineffective. The diagnosis of mitochondrial encephalomyopathy was based on the finding, in muscle specimens, of thickened basement membranes with myofibrillary degeneration and increased number of mitochondria peripherally distributed and with a dense granular matrix and some vacuoles. The clinical and EEG data suggest a. subcortical origin for this type of myoclonic syndrome.http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X1989000300016&lng=en&tlng=en
spellingShingle A. Cukiert
F. G. M. Naylor
H. B. Scapolan
M. M. Vilela
F. S. Aloe
J. O. Siffert
Ana M. Tsanaglis
Monica Haddad
Teresa C. Machado
Mary Carvalho-Alegro
J. A. Levy
L. Marques-Assis
Non-epileptic myoclonus and mitochondrial encephalomyopathy
Arquivos de Neuro-Psiquiatria
title Non-epileptic myoclonus and mitochondrial encephalomyopathy
title_full Non-epileptic myoclonus and mitochondrial encephalomyopathy
title_fullStr Non-epileptic myoclonus and mitochondrial encephalomyopathy
title_full_unstemmed Non-epileptic myoclonus and mitochondrial encephalomyopathy
title_short Non-epileptic myoclonus and mitochondrial encephalomyopathy
title_sort non epileptic myoclonus and mitochondrial encephalomyopathy
url http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X1989000300016&lng=en&tlng=en
work_keys_str_mv AT acukiert nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT fgmnaylor nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT hbscapolan nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT mmvilela nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT fsaloe nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT josiffert nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT anamtsanaglis nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT monicahaddad nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT teresacmachado nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT marycarvalhoalegro nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT jalevy nonepilepticmyoclonusandmitochondrialencephalomyopathy
AT lmarquesassis nonepilepticmyoclonusandmitochondrialencephalomyopathy