Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomalies

Summary: Runt-related transcription factor 1 translocated to 1 (RUNX1T1; also known as eight-twenty-one [ETO]) encodes a transcription regulator for hematopoietic genes and is well known for its involvement in hematologic malignancies, particularly acute myeloid leukemia (AML). However, its role in...

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Main Authors: Erfan Aref-Eshghi, Katherine J. Anderson, Lauren Boulay, Kathleen Brown, Jessica Duis, Christine A. Giummo, Jessica Ogawa, Deanna Alexis Carere, Elizabeth A. Normand, Yaping Qian, Kirsty McWalter, Erin Torti
Format: Article
Language:English
Published: Elsevier 2025-01-01
Series:HGG Advances
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Online Access:http://www.sciencedirect.com/science/article/pii/S2666247724001246
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author Erfan Aref-Eshghi
Katherine J. Anderson
Lauren Boulay
Kathleen Brown
Jessica Duis
Christine A. Giummo
Jessica Ogawa
Deanna Alexis Carere
Elizabeth A. Normand
Yaping Qian
Kirsty McWalter
Erin Torti
author_facet Erfan Aref-Eshghi
Katherine J. Anderson
Lauren Boulay
Kathleen Brown
Jessica Duis
Christine A. Giummo
Jessica Ogawa
Deanna Alexis Carere
Elizabeth A. Normand
Yaping Qian
Kirsty McWalter
Erin Torti
author_sort Erfan Aref-Eshghi
collection DOAJ
description Summary: Runt-related transcription factor 1 translocated to 1 (RUNX1T1; also known as eight-twenty-one [ETO]) encodes a transcription regulator for hematopoietic genes and is well known for its involvement in hematologic malignancies, particularly acute myeloid leukemia (AML). However, its role in congenital disease is less understood. This study provides detailed clinical and molecular information on three cases exhibiting neurodevelopmental and congenital anomalies with germline de novo alterations in RUNX1T1. One case features a de novo nonsense variant in the 5′ region of the gene (c.106C>T p.Gln36Ter), while the other two harbor de novo missense variants in the C terminus end (c.1234G>A p.Gly412Arg and c.1561C>T p.His521Tyr). Common features across cases include craniofacial dysmorphism and neurodevelopmental issues, including developmental delay, learning disabilities, attention-deficit hyperactivity disorder, and autism. This study, in conjunction with previously reported germline disruptions of RUNX1T1, provides evidence supporting the role of germline RUNX1T1 variation in human congenital neurodevelopmental disorders.
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spelling doaj-art-c2f349dfe2e048f4801d494abc5fbe232025-08-20T02:39:28ZengElsevierHGG Advances2666-24772025-01-016110038410.1016/j.xhgg.2024.100384Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomaliesErfan Aref-Eshghi0Katherine J. Anderson1Lauren Boulay2Kathleen Brown3Jessica Duis4Christine A. Giummo5Jessica Ogawa6Deanna Alexis Carere7Elizabeth A. Normand8Yaping Qian9Kirsty McWalter10Erin Torti11GeneDx, LLC, Gaithersburg, MD 20877, USAUniversity of Vermont Larner College of Medicine, Department of Pediatrics, Division of Clinical Genetics, Burlington, VT 05401, USADepartment of Pediatrics, Section of Genetics and Metabolism, University of Colorado School of Medicine Anschutz Medical Campus, Aurora, CO 80045, USADepartment of Pediatrics, Section of Genetics and Metabolism, University of Colorado School of Medicine Anschutz Medical Campus, Aurora, CO 80045, USARareDiseaseDoc, LLC, Aurora, CO, USAUniversity of Vermont Larner College of Medicine, Department of Pediatrics, Division of Clinical Genetics, Burlington, VT 05401, USACase Western Reserve University School of Medicine/Rainbow Babies and Children’s Hospital, Department of Genetics and Genome Sciences, Cleveland, OH 44106, USAGeneDx, LLC, Gaithersburg, MD 20877, USAGeneDx, LLC, Gaithersburg, MD 20877, USAGeneDx, LLC, Gaithersburg, MD 20877, USAGeneDx, LLC, Gaithersburg, MD 20877, USAGeneDx, LLC, Gaithersburg, MD 20877, USA; Corresponding authorSummary: Runt-related transcription factor 1 translocated to 1 (RUNX1T1; also known as eight-twenty-one [ETO]) encodes a transcription regulator for hematopoietic genes and is well known for its involvement in hematologic malignancies, particularly acute myeloid leukemia (AML). However, its role in congenital disease is less understood. This study provides detailed clinical and molecular information on three cases exhibiting neurodevelopmental and congenital anomalies with germline de novo alterations in RUNX1T1. One case features a de novo nonsense variant in the 5′ region of the gene (c.106C>T p.Gln36Ter), while the other two harbor de novo missense variants in the C terminus end (c.1234G>A p.Gly412Arg and c.1561C>T p.His521Tyr). Common features across cases include craniofacial dysmorphism and neurodevelopmental issues, including developmental delay, learning disabilities, attention-deficit hyperactivity disorder, and autism. This study, in conjunction with previously reported germline disruptions of RUNX1T1, provides evidence supporting the role of germline RUNX1T1 variation in human congenital neurodevelopmental disorders.http://www.sciencedirect.com/science/article/pii/S2666247724001246RUNX1T1clinical exome sequencingneurodevelopmental disorderautismcongenital anomaliescandidate gene
spellingShingle Erfan Aref-Eshghi
Katherine J. Anderson
Lauren Boulay
Kathleen Brown
Jessica Duis
Christine A. Giummo
Jessica Ogawa
Deanna Alexis Carere
Elizabeth A. Normand
Yaping Qian
Kirsty McWalter
Erin Torti
Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomalies
HGG Advances
RUNX1T1
clinical exome sequencing
neurodevelopmental disorder
autism
congenital anomalies
candidate gene
title Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomalies
title_full Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomalies
title_fullStr Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomalies
title_full_unstemmed Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomalies
title_short Germline de novo alterations of RUNX1T1 in individuals with neurodevelopmental and congenital anomalies
title_sort germline de novo alterations of runx1t1 in individuals with neurodevelopmental and congenital anomalies
topic RUNX1T1
clinical exome sequencing
neurodevelopmental disorder
autism
congenital anomalies
candidate gene
url http://www.sciencedirect.com/science/article/pii/S2666247724001246
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