Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et al

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Main Authors: Lei Huang, Hui Yang, Kaidi Chen, Jing Yuan, Jie Li, Guanghai Dai, Mancang Gu, Yan Shi
Format: Article
Language:English
Published: Wiley 2025-01-01
Series:Clinical and Translational Medicine
Online Access:https://doi.org/10.1002/ctm2.70158
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author Lei Huang
Hui Yang
Kaidi Chen
Jing Yuan
Jie Li
Guanghai Dai
Mancang Gu
Yan Shi
author_facet Lei Huang
Hui Yang
Kaidi Chen
Jing Yuan
Jie Li
Guanghai Dai
Mancang Gu
Yan Shi
author_sort Lei Huang
collection DOAJ
format Article
id doaj-art-c27e1306048d454f82b49dd675e9ab12
institution Kabale University
issn 2001-1326
language English
publishDate 2025-01-01
publisher Wiley
record_format Article
series Clinical and Translational Medicine
spelling doaj-art-c27e1306048d454f82b49dd675e9ab122025-01-25T04:00:38ZengWileyClinical and Translational Medicine2001-13262025-01-01151n/an/a10.1002/ctm2.70158Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et alLei Huang0Hui Yang1Kaidi Chen2Jing Yuan3Jie Li4Guanghai Dai5Mancang Gu6Yan Shi7Department of Oncology Ruijin Hospital, Shanghai Jiao Tong University School of Medicine Shanghai ChinaDepartment of Oncology Ruijin Hospital, Shanghai Jiao Tong University School of Medicine Shanghai ChinaSchool of Pharmaceutical Science Zhejiang Chinese Medical University Hangzhou ChinaDepartment of Pathology Chinese PLA General Hospital Beijing ChinaDepartment of Pathology Chinese PLA General Hospital Beijing ChinaDepartment of Medical Oncology Chinese PLA General Hospital Beijing ChinaSchool of Pharmaceutical Science Zhejiang Chinese Medical University Hangzhou ChinaDepartment of General Surgery Shanghai Seventh People's Hospital Shanghai University of Traditional Chinese Medicine Shanghai Chinahttps://doi.org/10.1002/ctm2.70158
spellingShingle Lei Huang
Hui Yang
Kaidi Chen
Jing Yuan
Jie Li
Guanghai Dai
Mancang Gu
Yan Shi
Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et al
Clinical and Translational Medicine
title Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et al
title_full Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et al
title_fullStr Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et al
title_full_unstemmed Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et al
title_short Erratum for the research article “The suppressive efficacy of THZ1 depends on KRAS mutation subtype and is associated with super‐enhancer activity and the PI3K/AKT/mTOR signalling in pancreatic ductal adenocarcinoma: A hypothesis‐generating study” by Huang et al
title_sort erratum for the research article the suppressive efficacy of thz1 depends on kras mutation subtype and is associated with super enhancer activity and the pi3k akt mtor signalling in pancreatic ductal adenocarcinoma a hypothesis generating study by huang et al
url https://doi.org/10.1002/ctm2.70158
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