Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in Mycobacteria

Cold Shock Proteins (Csps) are multifunctional regulators critical for bacterial stress adaptation. While Csps are known to regulate biofilm formation and low-temperature growth in some species, their roles in mycobacteria remain unclear. Here, we explored the functions of three Csps (CspA1, CspA2,...

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Main Authors: Jiachen Zheng, Linzhao He, Yizhang Wei, Jie Lu, Xiaolin Liu, Weihui Li
Format: Article
Language:English
Published: MDPI AG 2025-07-01
Series:Microorganisms
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Online Access:https://www.mdpi.com/2076-2607/13/7/1597
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author Jiachen Zheng
Linzhao He
Yizhang Wei
Jie Lu
Xiaolin Liu
Weihui Li
author_facet Jiachen Zheng
Linzhao He
Yizhang Wei
Jie Lu
Xiaolin Liu
Weihui Li
author_sort Jiachen Zheng
collection DOAJ
description Cold Shock Proteins (Csps) are multifunctional regulators critical for bacterial stress adaptation. While Csps are known to regulate biofilm formation and low-temperature growth in some species, their roles in mycobacteria remain unclear. Here, we explored the functions of three Csps (CspA1, CspA2, and CspB) in <i>Mycobacterium smegmatis</i>. We found that CspA1 promotes biofilm formation and isoniazid (INH) resistance but negatively affects oxidative stress resistance. In contrast, CspB promotes biofilm formation, whereas CspA2 appears functionally redundant in this process. Notably, CspB and CspA2 do not contribute redundantly to oxidative stress resistance. Proteomic analysis revealed that CspA1 significantly modulates the expression of key metabolic and stress-response proteins, including WhiB3 and KatG. Our findings establish CspA1 as a key regulatory factor in mycobacteria, linking metabolic adaptation to biofilm-associated drug resistance and oxidative defense.
format Article
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institution Kabale University
issn 2076-2607
language English
publishDate 2025-07-01
publisher MDPI AG
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series Microorganisms
spelling doaj-art-c261d4bf20234d4ea0fe336d07a1cf8d2025-08-20T03:35:38ZengMDPI AGMicroorganisms2076-26072025-07-01137159710.3390/microorganisms13071597Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in MycobacteriaJiachen Zheng0Linzhao He1Yizhang Wei2Jie Lu3Xiaolin Liu4Weihui Li5State Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, College of Life Science and Technology, Guangxi University, Nanning 530004, ChinaState Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, College of Life Science and Technology, Guangxi University, Nanning 530004, ChinaState Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, College of Life Science and Technology, Guangxi University, Nanning 530004, ChinaState Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, College of Life Science and Technology, Guangxi University, Nanning 530004, ChinaState Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, College of Life Science and Technology, Guangxi University, Nanning 530004, ChinaState Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, College of Life Science and Technology, Guangxi University, Nanning 530004, ChinaCold Shock Proteins (Csps) are multifunctional regulators critical for bacterial stress adaptation. While Csps are known to regulate biofilm formation and low-temperature growth in some species, their roles in mycobacteria remain unclear. Here, we explored the functions of three Csps (CspA1, CspA2, and CspB) in <i>Mycobacterium smegmatis</i>. We found that CspA1 promotes biofilm formation and isoniazid (INH) resistance but negatively affects oxidative stress resistance. In contrast, CspB promotes biofilm formation, whereas CspA2 appears functionally redundant in this process. Notably, CspB and CspA2 do not contribute redundantly to oxidative stress resistance. Proteomic analysis revealed that CspA1 significantly modulates the expression of key metabolic and stress-response proteins, including WhiB3 and KatG. Our findings establish CspA1 as a key regulatory factor in mycobacteria, linking metabolic adaptation to biofilm-associated drug resistance and oxidative defense.https://www.mdpi.com/2076-2607/13/7/1597cold shock proteinbiofilmsoxidative stressisoniazid resistancetranscriptional regulation
spellingShingle Jiachen Zheng
Linzhao He
Yizhang Wei
Jie Lu
Xiaolin Liu
Weihui Li
Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in Mycobacteria
Microorganisms
cold shock protein
biofilms
oxidative stress
isoniazid resistance
transcriptional regulation
title Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in Mycobacteria
title_full Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in Mycobacteria
title_fullStr Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in Mycobacteria
title_full_unstemmed Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in Mycobacteria
title_short Cold Shock Proteins Balance Biofilm-Associated Antibiotic Resistance and Oxidative Vulnerability in Mycobacteria
title_sort cold shock proteins balance biofilm associated antibiotic resistance and oxidative vulnerability in mycobacteria
topic cold shock protein
biofilms
oxidative stress
isoniazid resistance
transcriptional regulation
url https://www.mdpi.com/2076-2607/13/7/1597
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AT yizhangwei coldshockproteinsbalancebiofilmassociatedantibioticresistanceandoxidativevulnerabilityinmycobacteria
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