The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles

IntroductionUlcerative colitis (UC) is a chronic gastrointestinal disease characterized by symptoms of abdominal pain and diarrhea. Chymase is a serine protease released by the mast cells and is highly expressed in patients with UC. However, its role in disease pathogenesis remains poorly understoo...

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Main Authors: Zhiqiang Li, Xiaoyuan Kuang, Dashuang Mo, Magnus Åbrink, Ge Shan, Jin-ping Li, Can Yang, Yuexi Wang, Tao Shen, Weiwei Yu
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-07-01
Series:Frontiers in Cellular and Infection Microbiology
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Online Access:https://www.frontiersin.org/articles/10.3389/fcimb.2025.1481927/full
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author Zhiqiang Li
Zhiqiang Li
Zhiqiang Li
Xiaoyuan Kuang
Dashuang Mo
Magnus Åbrink
Ge Shan
Jin-ping Li
Can Yang
Yuexi Wang
Tao Shen
Weiwei Yu
author_facet Zhiqiang Li
Zhiqiang Li
Zhiqiang Li
Xiaoyuan Kuang
Dashuang Mo
Magnus Åbrink
Ge Shan
Jin-ping Li
Can Yang
Yuexi Wang
Tao Shen
Weiwei Yu
author_sort Zhiqiang Li
collection DOAJ
description IntroductionUlcerative colitis (UC) is a chronic gastrointestinal disease characterized by symptoms of abdominal pain and diarrhea. Chymase is a serine protease released by the mast cells and is highly expressed in patients with UC. However, its role in disease pathogenesis remains poorly understood. In mice, mast cell protease-4 (MCP-4) is considered as the functional homolog of human chymase, sharing similar enzymatic activity and biological roles.MethodsTo investigate the role of MCP-4 in UC, we induced colitis in Mcpt-4-deficient (Mcpt-4∆Cre) and littermate control (Mcpt-4fl/fl) mice using 2% dextran sulfate sodium salt (DSS) for 8 days followed by 2 days of water. After sacrifice, colon length was measured, and tissue samples were analyzed by histology (H&E and toluidine blue staining), qPCR, and protein assays. Colonic contents were collected for microbiota and metabolomics analysis.ResultsThe results show that the Mcpt-4-deficiency exacerbated colitis, as reflected by the significantly greater weight loss, higher histological scores, elevated levels of myeloperoxidase and most of determined cytokines. Furthermore, the Mcpt-4∆Cre colitis mice displayed a distinct shift in colonic microbiota composition, notably with significantly increased abundance of bacteria (Akkermansia and Turicibacter), associated with potentially worsened inflammation in colitis models. In addition, metabolomic profiling revealed alterations in colonic metabolites involved in key Kyoto Encyclopedia of Genes and Genomes (KEGG) pathways, including NF-kappa B signaling, Th1 and Th2 cell differentiation.DiscussionThese findings reveal that the mouse chymase MCPT-4 plays important roles in maintaining the intestinal homeostasis during colitis, potentially through regulation of colonic cytokines, microbial and metabolic networks.
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spelling doaj-art-c166fa57d22d40c4b411b6dd75ee773f2025-08-20T03:17:39ZengFrontiers Media S.A.Frontiers in Cellular and Infection Microbiology2235-29882025-07-011510.3389/fcimb.2025.14819271481927The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profilesZhiqiang Li0Zhiqiang Li1Zhiqiang Li2Xiaoyuan Kuang3Dashuang Mo4Magnus Åbrink5Ge Shan6Jin-ping Li7Can Yang8Yuexi Wang9Tao Shen10Weiwei Yu11Department of Immunology, College of Basic Medicine, Guizhou Medical University, Guiyang, ChinaThe Key and Characteristic Laboratory of Modern Pathogen Biology, College of Basic Medicine, Guizhou Medical University; Department of Medical Parasitology, College of Basic Medicine, Guizhou Medical University, Guiyang, ChinaDepartment of Medical Biochemistry and Microbiology, Uppsala University, Uppsala, SwedenThe Key and Characteristic Laboratory of Modern Pathogen Biology, College of Basic Medicine, Guizhou Medical University; Department of Medical Parasitology, College of Basic Medicine, Guizhou Medical University, Guiyang, ChinaDepartment of Immunology, College of Basic Medicine, Guizhou Medical University, Guiyang, ChinaDivision of Anatomy, Physiology, Immunology and Patholog, Department of Animal Biosciences, Swedish University of Agricultural Sciences, Uppsala, SwedenDepartment of Immunology, College of Basic Medicine, Guizhou Medical University, Guiyang, ChinaDepartment of Medical Biochemistry and Microbiology, Uppsala University, Uppsala, SwedenUrologic Surgery Clinic, Tongren Municipal People’s Hospital, Tongren, ChinaDepartment of Immunology, College of Basic Medicine, Guizhou Medical University, Guiyang, ChinaDepartment of Immunology, College of Basic Medicine, Guizhou Medical University, Guiyang, ChinaDepartment of Hepatobiliary Surgery, The Affiliated Drum Tower Hospital of Nanjing University Medical School, Nanjing, ChinaIntroductionUlcerative colitis (UC) is a chronic gastrointestinal disease characterized by symptoms of abdominal pain and diarrhea. Chymase is a serine protease released by the mast cells and is highly expressed in patients with UC. However, its role in disease pathogenesis remains poorly understood. In mice, mast cell protease-4 (MCP-4) is considered as the functional homolog of human chymase, sharing similar enzymatic activity and biological roles.MethodsTo investigate the role of MCP-4 in UC, we induced colitis in Mcpt-4-deficient (Mcpt-4∆Cre) and littermate control (Mcpt-4fl/fl) mice using 2% dextran sulfate sodium salt (DSS) for 8 days followed by 2 days of water. After sacrifice, colon length was measured, and tissue samples were analyzed by histology (H&E and toluidine blue staining), qPCR, and protein assays. Colonic contents were collected for microbiota and metabolomics analysis.ResultsThe results show that the Mcpt-4-deficiency exacerbated colitis, as reflected by the significantly greater weight loss, higher histological scores, elevated levels of myeloperoxidase and most of determined cytokines. Furthermore, the Mcpt-4∆Cre colitis mice displayed a distinct shift in colonic microbiota composition, notably with significantly increased abundance of bacteria (Akkermansia and Turicibacter), associated with potentially worsened inflammation in colitis models. In addition, metabolomic profiling revealed alterations in colonic metabolites involved in key Kyoto Encyclopedia of Genes and Genomes (KEGG) pathways, including NF-kappa B signaling, Th1 and Th2 cell differentiation.DiscussionThese findings reveal that the mouse chymase MCPT-4 plays important roles in maintaining the intestinal homeostasis during colitis, potentially through regulation of colonic cytokines, microbial and metabolic networks.https://www.frontiersin.org/articles/10.3389/fcimb.2025.1481927/fullulcerative colitismicrobiotachymaseexacerbationmetabolism
spellingShingle Zhiqiang Li
Zhiqiang Li
Zhiqiang Li
Xiaoyuan Kuang
Dashuang Mo
Magnus Åbrink
Ge Shan
Jin-ping Li
Can Yang
Yuexi Wang
Tao Shen
Weiwei Yu
The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles
Frontiers in Cellular and Infection Microbiology
ulcerative colitis
microbiota
chymase
exacerbation
metabolism
title The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles
title_full The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles
title_fullStr The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles
title_full_unstemmed The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles
title_short The deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt-induced colitis in mice and is associated with altered microbiota and metabolome profiles
title_sort deficiency of chymase mast cell protease 4 exacerbates dextran sulfate sodium salt induced colitis in mice and is associated with altered microbiota and metabolome profiles
topic ulcerative colitis
microbiota
chymase
exacerbation
metabolism
url https://www.frontiersin.org/articles/10.3389/fcimb.2025.1481927/full
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