High-throughput discovery of genetic determinants of circadian misalignment.

Circadian systems provide a fitness advantage to organisms by allowing them to adapt to daily changes of environmental cues, such as light/dark cycles. The molecular mechanism underlying the circadian clock has been well characterized. However, how internal circadian clocks are entrained with regula...

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Main Authors: Tao Zhang, Pancheng Xie, Yingying Dong, Zhiwei Liu, Fei Zhou, Dejing Pan, Zhengyun Huang, Qiaocheng Zhai, Yue Gu, Qingyu Wu, Nobuhiko Tanaka, Yuichi Obata, Allan Bradley, Christopher J Lelliott, Sanger Institute Mouse Genetics Project, Lauryl M J Nutter, Colin McKerlie, Ann M Flenniken, Marie-France Champy, Tania Sorg, Yann Herault, Martin Hrabe De Angelis, Valerie Gailus Durner, Ann-Marie Mallon, Steve D M Brown, Terry Meehan, Helen E Parkinson, Damian Smedley, K C Kent Lloyd, Jun Yan, Xiang Gao, Je Kyung Seong, Chi-Kuang Leo Wang, Radislav Sedlacek, Yi Liu, Jan Rozman, Ling Yang, Ying Xu
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2020-01-01
Series:PLoS Genetics
Online Access:https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1008577&type=printable
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author Tao Zhang
Pancheng Xie
Yingying Dong
Zhiwei Liu
Fei Zhou
Dejing Pan
Zhengyun Huang
Qiaocheng Zhai
Yue Gu
Qingyu Wu
Nobuhiko Tanaka
Yuichi Obata
Allan Bradley
Christopher J Lelliott
Sanger Institute Mouse Genetics Project
Lauryl M J Nutter
Colin McKerlie
Ann M Flenniken
Marie-France Champy
Tania Sorg
Yann Herault
Martin Hrabe De Angelis
Valerie Gailus Durner
Ann-Marie Mallon
Steve D M Brown
Terry Meehan
Helen E Parkinson
Damian Smedley
K C Kent Lloyd
Jun Yan
Xiang Gao
Je Kyung Seong
Chi-Kuang Leo Wang
Radislav Sedlacek
Yi Liu
Jan Rozman
Ling Yang
Ying Xu
author_facet Tao Zhang
Pancheng Xie
Yingying Dong
Zhiwei Liu
Fei Zhou
Dejing Pan
Zhengyun Huang
Qiaocheng Zhai
Yue Gu
Qingyu Wu
Nobuhiko Tanaka
Yuichi Obata
Allan Bradley
Christopher J Lelliott
Sanger Institute Mouse Genetics Project
Lauryl M J Nutter
Colin McKerlie
Ann M Flenniken
Marie-France Champy
Tania Sorg
Yann Herault
Martin Hrabe De Angelis
Valerie Gailus Durner
Ann-Marie Mallon
Steve D M Brown
Terry Meehan
Helen E Parkinson
Damian Smedley
K C Kent Lloyd
Jun Yan
Xiang Gao
Je Kyung Seong
Chi-Kuang Leo Wang
Radislav Sedlacek
Yi Liu
Jan Rozman
Ling Yang
Ying Xu
author_sort Tao Zhang
collection DOAJ
description Circadian systems provide a fitness advantage to organisms by allowing them to adapt to daily changes of environmental cues, such as light/dark cycles. The molecular mechanism underlying the circadian clock has been well characterized. However, how internal circadian clocks are entrained with regular daily light/dark cycles remains unclear. By collecting and analyzing indirect calorimetry (IC) data from more than 2000 wild-type mice available from the International Mouse Phenotyping Consortium (IMPC), we show that the onset time and peak phase of activity and food intake rhythms are reliable parameters for screening defects of circadian misalignment. We developed a machine learning algorithm to quantify these two parameters in our misalignment screen (SyncScreener) with existing datasets and used it to screen 750 mutant mouse lines from five IMPC phenotyping centres. Mutants of five genes (Slc7a11, Rhbdl1, Spop, Ctc1 and Oxtr) were found to be associated with altered patterns of activity or food intake. By further studying the Slc7a11tm1a/tm1a mice, we confirmed its advanced activity phase phenotype in response to a simulated jetlag and skeleton photoperiod stimuli. Disruption of Slc7a11 affected the intercellular communication in the suprachiasmatic nucleus, suggesting a defect in synchronization of clock neurons. Our study has established a systematic phenotype analysis approach that can be used to uncover the mechanism of circadian entrainment in mice.
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language English
publishDate 2020-01-01
publisher Public Library of Science (PLoS)
record_format Article
series PLoS Genetics
spelling doaj-art-c12d6bf50c5146379916d239a99b9e3e2025-08-20T02:11:01ZengPublic Library of Science (PLoS)PLoS Genetics1553-73901553-74042020-01-01161e100857710.1371/journal.pgen.1008577High-throughput discovery of genetic determinants of circadian misalignment.Tao ZhangPancheng XieYingying DongZhiwei LiuFei ZhouDejing PanZhengyun HuangQiaocheng ZhaiYue GuQingyu WuNobuhiko TanakaYuichi ObataAllan BradleyChristopher J LelliottSanger Institute Mouse Genetics ProjectLauryl M J NutterColin McKerlieAnn M FlennikenMarie-France ChampyTania SorgYann HeraultMartin Hrabe De AngelisValerie Gailus DurnerAnn-Marie MallonSteve D M BrownTerry MeehanHelen E ParkinsonDamian SmedleyK C Kent LloydJun YanXiang GaoJe Kyung SeongChi-Kuang Leo WangRadislav SedlacekYi LiuJan RozmanLing YangYing XuCircadian systems provide a fitness advantage to organisms by allowing them to adapt to daily changes of environmental cues, such as light/dark cycles. The molecular mechanism underlying the circadian clock has been well characterized. However, how internal circadian clocks are entrained with regular daily light/dark cycles remains unclear. By collecting and analyzing indirect calorimetry (IC) data from more than 2000 wild-type mice available from the International Mouse Phenotyping Consortium (IMPC), we show that the onset time and peak phase of activity and food intake rhythms are reliable parameters for screening defects of circadian misalignment. We developed a machine learning algorithm to quantify these two parameters in our misalignment screen (SyncScreener) with existing datasets and used it to screen 750 mutant mouse lines from five IMPC phenotyping centres. Mutants of five genes (Slc7a11, Rhbdl1, Spop, Ctc1 and Oxtr) were found to be associated with altered patterns of activity or food intake. By further studying the Slc7a11tm1a/tm1a mice, we confirmed its advanced activity phase phenotype in response to a simulated jetlag and skeleton photoperiod stimuli. Disruption of Slc7a11 affected the intercellular communication in the suprachiasmatic nucleus, suggesting a defect in synchronization of clock neurons. Our study has established a systematic phenotype analysis approach that can be used to uncover the mechanism of circadian entrainment in mice.https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1008577&type=printable
spellingShingle Tao Zhang
Pancheng Xie
Yingying Dong
Zhiwei Liu
Fei Zhou
Dejing Pan
Zhengyun Huang
Qiaocheng Zhai
Yue Gu
Qingyu Wu
Nobuhiko Tanaka
Yuichi Obata
Allan Bradley
Christopher J Lelliott
Sanger Institute Mouse Genetics Project
Lauryl M J Nutter
Colin McKerlie
Ann M Flenniken
Marie-France Champy
Tania Sorg
Yann Herault
Martin Hrabe De Angelis
Valerie Gailus Durner
Ann-Marie Mallon
Steve D M Brown
Terry Meehan
Helen E Parkinson
Damian Smedley
K C Kent Lloyd
Jun Yan
Xiang Gao
Je Kyung Seong
Chi-Kuang Leo Wang
Radislav Sedlacek
Yi Liu
Jan Rozman
Ling Yang
Ying Xu
High-throughput discovery of genetic determinants of circadian misalignment.
PLoS Genetics
title High-throughput discovery of genetic determinants of circadian misalignment.
title_full High-throughput discovery of genetic determinants of circadian misalignment.
title_fullStr High-throughput discovery of genetic determinants of circadian misalignment.
title_full_unstemmed High-throughput discovery of genetic determinants of circadian misalignment.
title_short High-throughput discovery of genetic determinants of circadian misalignment.
title_sort high throughput discovery of genetic determinants of circadian misalignment
url https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1008577&type=printable
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