NAMPT regulates mitochondria and oxidative stress level for mouse early embryo development

Abstract Background Nicotinamide phosphoribosyltransferase (NAMPT) is an enzyme that involves into NMN-NAD + synthesis which involves into cellular metabolism related with aging, immune function, and neurodegeneration. However, its roles in early embryo development are still unclear. Methods In pres...

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Main Authors: Mei-Hua Liao, Xin Liu, Xiao-Ting Yu, Shun Zhang, You-Zhu Li, Lin-Lin Hu, Shao-Chen Sun, Jun-Li Wang
Format: Article
Language:English
Published: BMC 2025-05-01
Series:Biological Research
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Online Access:https://doi.org/10.1186/s40659-025-00608-3
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author Mei-Hua Liao
Xin Liu
Xiao-Ting Yu
Shun Zhang
You-Zhu Li
Lin-Lin Hu
Shao-Chen Sun
Jun-Li Wang
author_facet Mei-Hua Liao
Xin Liu
Xiao-Ting Yu
Shun Zhang
You-Zhu Li
Lin-Lin Hu
Shao-Chen Sun
Jun-Li Wang
author_sort Mei-Hua Liao
collection DOAJ
description Abstract Background Nicotinamide phosphoribosyltransferase (NAMPT) is an enzyme that involves into NMN-NAD + synthesis which involves into cellular metabolism related with aging, immune function, and neurodegeneration. However, its roles in early embryo development are still unclear. Methods In present study we disturbed the NAMPT activity and employed immunofluorescence staining and live cell imaging to explore its roles during early embryo development. Results We showed that NAMPT mRNA level was stable during mouse early embryo development, and NAMPT accumulated in the nucleus of blastomeres in mouse embryos. The loss of NAMPT activity disturbed the early cleavage from zygote to 2-cell, 4-cell to morula formation in the dose-dependent manner. We found that NAMPT inhibition disrupted mitochondria function in 2-cell embryos, showing decreased mitochondria number and aberrant accumulation in the blastomeres, which further disturb mitochondrial membrane potential level and elevated ROS level in embryos, indicating the occurrence of oxidative stress. Moreover, NAMPT inhibition also increased the apoptotic index, showing with increased Annexin-V signals and apoptotic gene expression. Conclusions Taken together, our study provided the evidence that NAMPT was essential for the mitochondria function to control oxidative stress and apoptosis during mouse early embryo development.
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publishDate 2025-05-01
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spelling doaj-art-c0b8eea6cff44d1e86d506dfd26fe3082025-08-20T01:49:46ZengBMCBiological Research0717-62872025-05-0158111010.1186/s40659-025-00608-3NAMPT regulates mitochondria and oxidative stress level for mouse early embryo developmentMei-Hua Liao0Xin Liu1Xiao-Ting Yu2Shun Zhang3You-Zhu Li4Lin-Lin Hu5Shao-Chen Sun6Jun-Li Wang7Key Laboratory of Research on Clinical Molecular Diagnosis for High Incidence Diseases in Western Guangxi of Guangxi Higher Education Institutions, Reproductive Medicine of Guangxi Medical and Health Key Discipline Construction Project, Affiliated Hospital of Youjiang Medical University for NationalitiesCollege of Animal Science and Technology, Nanjing Agricultural UniversityKey Laboratory of Research on Clinical Molecular Diagnosis for High Incidence Diseases in Western Guangxi of Guangxi Higher Education Institutions, Reproductive Medicine of Guangxi Medical and Health Key Discipline Construction Project, Affiliated Hospital of Youjiang Medical University for NationalitiesDepartment of Reproductive Medical Center, The Affiliated Hospital of Guilin Medical UniversityDepartment of Reproductive Medicine, School of Medicine, The First Affiliated Hospital of Xiamen University, Xiamen UniversityKey Laboratory of Research on Clinical Molecular Diagnosis for High Incidence Diseases in Western Guangxi of Guangxi Higher Education Institutions, Reproductive Medicine of Guangxi Medical and Health Key Discipline Construction Project, Affiliated Hospital of Youjiang Medical University for NationalitiesKey Laboratory of Research on Clinical Molecular Diagnosis for High Incidence Diseases in Western Guangxi of Guangxi Higher Education Institutions, Reproductive Medicine of Guangxi Medical and Health Key Discipline Construction Project, Affiliated Hospital of Youjiang Medical University for NationalitiesKey Laboratory of Research on Clinical Molecular Diagnosis for High Incidence Diseases in Western Guangxi of Guangxi Higher Education Institutions, Reproductive Medicine of Guangxi Medical and Health Key Discipline Construction Project, Affiliated Hospital of Youjiang Medical University for NationalitiesAbstract Background Nicotinamide phosphoribosyltransferase (NAMPT) is an enzyme that involves into NMN-NAD + synthesis which involves into cellular metabolism related with aging, immune function, and neurodegeneration. However, its roles in early embryo development are still unclear. Methods In present study we disturbed the NAMPT activity and employed immunofluorescence staining and live cell imaging to explore its roles during early embryo development. Results We showed that NAMPT mRNA level was stable during mouse early embryo development, and NAMPT accumulated in the nucleus of blastomeres in mouse embryos. The loss of NAMPT activity disturbed the early cleavage from zygote to 2-cell, 4-cell to morula formation in the dose-dependent manner. We found that NAMPT inhibition disrupted mitochondria function in 2-cell embryos, showing decreased mitochondria number and aberrant accumulation in the blastomeres, which further disturb mitochondrial membrane potential level and elevated ROS level in embryos, indicating the occurrence of oxidative stress. Moreover, NAMPT inhibition also increased the apoptotic index, showing with increased Annexin-V signals and apoptotic gene expression. Conclusions Taken together, our study provided the evidence that NAMPT was essential for the mitochondria function to control oxidative stress and apoptosis during mouse early embryo development.https://doi.org/10.1186/s40659-025-00608-3OocyteActinMitochondriaOxidative stressParkinson’s disease
spellingShingle Mei-Hua Liao
Xin Liu
Xiao-Ting Yu
Shun Zhang
You-Zhu Li
Lin-Lin Hu
Shao-Chen Sun
Jun-Li Wang
NAMPT regulates mitochondria and oxidative stress level for mouse early embryo development
Biological Research
Oocyte
Actin
Mitochondria
Oxidative stress
Parkinson’s disease
title NAMPT regulates mitochondria and oxidative stress level for mouse early embryo development
title_full NAMPT regulates mitochondria and oxidative stress level for mouse early embryo development
title_fullStr NAMPT regulates mitochondria and oxidative stress level for mouse early embryo development
title_full_unstemmed NAMPT regulates mitochondria and oxidative stress level for mouse early embryo development
title_short NAMPT regulates mitochondria and oxidative stress level for mouse early embryo development
title_sort nampt regulates mitochondria and oxidative stress level for mouse early embryo development
topic Oocyte
Actin
Mitochondria
Oxidative stress
Parkinson’s disease
url https://doi.org/10.1186/s40659-025-00608-3
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