Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic Neuropathy

T-type Ca2+ channels are known as important participants of nociception and their remodeling contributes to diabetes-induced alterations of pain sensation. In this work we have established that about 30% of rat nonpeptidergic thermal C-type nociceptive (NTCN) neurons of segments L4–L6 express a slow...

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Main Authors: Eugen V. Khomula, Anya L. Borisyuk, Viacheslav Y. Viatchenko-Karpinski, Andrea Briede, Pavel V. Belan, Nana V. Voitenko
Format: Article
Language:English
Published: Wiley 2014-01-01
Series:Neural Plasticity
Online Access:http://dx.doi.org/10.1155/2014/938235
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author Eugen V. Khomula
Anya L. Borisyuk
Viacheslav Y. Viatchenko-Karpinski
Andrea Briede
Pavel V. Belan
Nana V. Voitenko
author_facet Eugen V. Khomula
Anya L. Borisyuk
Viacheslav Y. Viatchenko-Karpinski
Andrea Briede
Pavel V. Belan
Nana V. Voitenko
author_sort Eugen V. Khomula
collection DOAJ
description T-type Ca2+ channels are known as important participants of nociception and their remodeling contributes to diabetes-induced alterations of pain sensation. In this work we have established that about 30% of rat nonpeptidergic thermal C-type nociceptive (NTCN) neurons of segments L4–L6 express a slow T-type Ca2+ current (T-current) while a fast T-current is expressed in the other 70% of these neurons. Streptozotocin-induced diabetes in young rats resulted in thermal hyperalgesia, hypoalgesia, or normalgesia 5-6 weeks after the induction. Our results show that NTCN neurons obtained from hyperalgesic animals do not express the slow T-current. Meanwhile, the fraction of neurons expressing the slow T-current did not significantly change in the hypo- and normalgesic diabetic groups. Moreover, the peak current density of fast T-current was significantly increased only in the neurons of hyperalgesic group. In contrast, the peak current density of slow T-current was significantly decreased in the hypo- and normalgesic groups. Experimental diabetes also resulted in a depolarizing shift of steady-state inactivation of fast T-current in the hyperalgesic group and slow T-current in the hypo- and normalgesic groups. We suggest that the observed changes may contribute to expression of different types of peripheral diabetic neuropathy occurring during the development of diabetes mellitus.
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spelling doaj-art-bf17dd1a7e3347edbedc21dffbb5c8f02025-08-20T03:23:07ZengWileyNeural Plasticity2090-59041687-54432014-01-01201410.1155/2014/938235938235Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic NeuropathyEugen V. Khomula0Anya L. Borisyuk1Viacheslav Y. Viatchenko-Karpinski2Andrea Briede3Pavel V. Belan4Nana V. Voitenko5International Center of Molecular Physiology of National Academy of Sciences of Ukraine, 4 Bogomoletz Street., Kyiv 01024, UkraineState Key Laboratory of Molecular and Cellular Biology, Bogomoletz Institute of Physiology of National Academy of Sciences of Ukraine, 4 Bogomoletz Street, Kyiv 01024, UkraineState Key Laboratory of Molecular and Cellular Biology, Bogomoletz Institute of Physiology of National Academy of Sciences of Ukraine, 4 Bogomoletz Street, Kyiv 01024, UkraineState Key Laboratory of Molecular and Cellular Biology, Bogomoletz Institute of Physiology of National Academy of Sciences of Ukraine, 4 Bogomoletz Street, Kyiv 01024, UkraineInternational Center of Molecular Physiology of National Academy of Sciences of Ukraine, 4 Bogomoletz Street., Kyiv 01024, UkraineInternational Center of Molecular Physiology of National Academy of Sciences of Ukraine, 4 Bogomoletz Street., Kyiv 01024, UkraineT-type Ca2+ channels are known as important participants of nociception and their remodeling contributes to diabetes-induced alterations of pain sensation. In this work we have established that about 30% of rat nonpeptidergic thermal C-type nociceptive (NTCN) neurons of segments L4–L6 express a slow T-type Ca2+ current (T-current) while a fast T-current is expressed in the other 70% of these neurons. Streptozotocin-induced diabetes in young rats resulted in thermal hyperalgesia, hypoalgesia, or normalgesia 5-6 weeks after the induction. Our results show that NTCN neurons obtained from hyperalgesic animals do not express the slow T-current. Meanwhile, the fraction of neurons expressing the slow T-current did not significantly change in the hypo- and normalgesic diabetic groups. Moreover, the peak current density of fast T-current was significantly increased only in the neurons of hyperalgesic group. In contrast, the peak current density of slow T-current was significantly decreased in the hypo- and normalgesic groups. Experimental diabetes also resulted in a depolarizing shift of steady-state inactivation of fast T-current in the hyperalgesic group and slow T-current in the hypo- and normalgesic groups. We suggest that the observed changes may contribute to expression of different types of peripheral diabetic neuropathy occurring during the development of diabetes mellitus.http://dx.doi.org/10.1155/2014/938235
spellingShingle Eugen V. Khomula
Anya L. Borisyuk
Viacheslav Y. Viatchenko-Karpinski
Andrea Briede
Pavel V. Belan
Nana V. Voitenko
Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic Neuropathy
Neural Plasticity
title Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic Neuropathy
title_full Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic Neuropathy
title_fullStr Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic Neuropathy
title_full_unstemmed Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic Neuropathy
title_short Nociceptive Neurons Differentially Express Fast and Slow T-Type Ca2+ Currents in Different Types of Diabetic Neuropathy
title_sort nociceptive neurons differentially express fast and slow t type ca2 currents in different types of diabetic neuropathy
url http://dx.doi.org/10.1155/2014/938235
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