Diabetes causal attributions among affected and unaffected individuals
Objective The present study aims to describe and compare causal attributions for type 1 diabetes (T1D) and type 2 diabetes (T2D) among affected and unaffected individuals and to investigate the relationships among attributions, attitudes, and beliefs.Research design and methods Adults with no diabet...
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| Format: | Article |
| Language: | English |
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BMJ Publishing Group
2019-05-01
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| Series: | BMJ Open Diabetes Research & Care |
| Online Access: | https://drc.bmj.com/content/7/1/e000708.full |
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| author | Margaret K Rose Kristi A Costabile Sarah E Boland Rachel W Cohen Susan Persky |
| author_facet | Margaret K Rose Kristi A Costabile Sarah E Boland Rachel W Cohen Susan Persky |
| author_sort | Margaret K Rose |
| collection | DOAJ |
| description | Objective The present study aims to describe and compare causal attributions for type 1 diabetes (T1D) and type 2 diabetes (T2D) among affected and unaffected individuals and to investigate the relationships among attributions, attitudes, and beliefs.Research design and methods Adults with no diabetes (N=458), T1D (N=192), or T2D (N=207) completed an online survey. Measures assessed diabetes conceptual knowledge, causal attributions for T1D and T2D, perceived control over diabetes onset, and favorability judgements of individuals affected by each type.Results Results indicate general agreement on causal attributions for T1D and T2D among all respondent groups, with some divergences by disease status. All respondents attributed both T1D and T2D to genetics, and genetic attributions were positively associated with favorability judgements of individuals with T2D, but not those with T1D.Conclusions This report sets the stage for investigations into how and why attributions for T1D and T2D differ and the implications of these differences including stigmatization of individuals with diabetes and diabetes-related self-concept. Additionally, this work can inform efforts towards clinical and public health education to prevent and optimize treatment of T1D and T2D. |
| format | Article |
| id | doaj-art-bd3cf27f6b2147eaad6273f845c4e05a |
| institution | Kabale University |
| issn | 2052-4897 |
| language | English |
| publishDate | 2019-05-01 |
| publisher | BMJ Publishing Group |
| record_format | Article |
| series | BMJ Open Diabetes Research & Care |
| spelling | doaj-art-bd3cf27f6b2147eaad6273f845c4e05a2024-12-15T18:45:09ZengBMJ Publishing GroupBMJ Open Diabetes Research & Care2052-48972019-05-017110.1136/bmjdrc-2019-000708Diabetes causal attributions among affected and unaffected individualsMargaret K Rose0Kristi A Costabile1Sarah E Boland2Rachel W Cohen3Susan Persky4Social and Behavioral Research Branch, National Human Genome Research Institute, Bethesda, Maryland, USADepartment of Psychology, Iowa State University, Ames, Iowa, USASocial and Behavioral Research Branch, National Human Genome Research Institute, Bethesda, Maryland, USASocial and Behavioral Research Branch, National Human Genome Research Institute, Bethesda, Maryland, USA3 Social and Behavioral Research Branch, National Human Genome Research Institute Division of Intramural Research, National Institutes of Health, Bethesda, Maryland, USAObjective The present study aims to describe and compare causal attributions for type 1 diabetes (T1D) and type 2 diabetes (T2D) among affected and unaffected individuals and to investigate the relationships among attributions, attitudes, and beliefs.Research design and methods Adults with no diabetes (N=458), T1D (N=192), or T2D (N=207) completed an online survey. Measures assessed diabetes conceptual knowledge, causal attributions for T1D and T2D, perceived control over diabetes onset, and favorability judgements of individuals affected by each type.Results Results indicate general agreement on causal attributions for T1D and T2D among all respondent groups, with some divergences by disease status. All respondents attributed both T1D and T2D to genetics, and genetic attributions were positively associated with favorability judgements of individuals with T2D, but not those with T1D.Conclusions This report sets the stage for investigations into how and why attributions for T1D and T2D differ and the implications of these differences including stigmatization of individuals with diabetes and diabetes-related self-concept. Additionally, this work can inform efforts towards clinical and public health education to prevent and optimize treatment of T1D and T2D.https://drc.bmj.com/content/7/1/e000708.full |
| spellingShingle | Margaret K Rose Kristi A Costabile Sarah E Boland Rachel W Cohen Susan Persky Diabetes causal attributions among affected and unaffected individuals BMJ Open Diabetes Research & Care |
| title | Diabetes causal attributions among affected and unaffected individuals |
| title_full | Diabetes causal attributions among affected and unaffected individuals |
| title_fullStr | Diabetes causal attributions among affected and unaffected individuals |
| title_full_unstemmed | Diabetes causal attributions among affected and unaffected individuals |
| title_short | Diabetes causal attributions among affected and unaffected individuals |
| title_sort | diabetes causal attributions among affected and unaffected individuals |
| url | https://drc.bmj.com/content/7/1/e000708.full |
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