Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer Cells

Interleukin-10 (IL10) is best studied for its inhibitory action on immune cells and ability to suppress an antitumour immune response. But IL10 also exerts direct effects on nonimmune cells such as prostate cancer epithelial cells. Elevated serum levels of IL10 observed in prostate and other cancer...

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Main Authors: Abrar Samiea, Jeff S. J. Yoon, Christopher J. Ong, Amina Zoubeidi, Thomas C. Chamberlain, Alice L.-F. Mui
Format: Article
Language:English
Published: Wiley 2020-01-01
Series:Prostate Cancer
Online Access:http://dx.doi.org/10.1155/2020/5305306
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author Abrar Samiea
Jeff S. J. Yoon
Christopher J. Ong
Amina Zoubeidi
Thomas C. Chamberlain
Alice L.-F. Mui
author_facet Abrar Samiea
Jeff S. J. Yoon
Christopher J. Ong
Amina Zoubeidi
Thomas C. Chamberlain
Alice L.-F. Mui
author_sort Abrar Samiea
collection DOAJ
description Interleukin-10 (IL10) is best studied for its inhibitory action on immune cells and ability to suppress an antitumour immune response. But IL10 also exerts direct effects on nonimmune cells such as prostate cancer epithelial cells. Elevated serum levels of IL10 observed in prostate and other cancer patients are associated with poor prognosis. After first-line androgen-deprivation therapy, prostate cancer patients are treated with androgen receptor antagonists such as enzalutamide to inhibit androgen-dependent prostate cancer cell growth. However, development of resistance inevitably occurs and this is associated with tumour differentiation to more aggressive forms such as a neuroendocrine phenotype characterized by expression of neuron specific enolase and synaptophysin. We found that treatment of prostate cancer cell lines in vitro with IL10 or enzalutamide induced markers of neuroendocrine differentiation and inhibited androgen receptor reporter activity. Both also upregulated the levels of PDL1, which could promote tumour survival in vivo through its interaction with the immune cell inhibitory receptor PD1 to suppress antitumour immunity. These findings suggest that IL10’s direct action on prostate cancer cells could contribute to prostate cancer progression independent of IL10’s suppression of host immune cells.
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issn 2090-3111
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language English
publishDate 2020-01-01
publisher Wiley
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series Prostate Cancer
spelling doaj-art-bc6b7ee61bac411b9c6eba4813cc37ea2025-08-20T03:23:19ZengWileyProstate Cancer2090-31112090-312X2020-01-01202010.1155/2020/53053065305306Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer CellsAbrar Samiea0Jeff S. J. Yoon1Christopher J. Ong2Amina Zoubeidi3Thomas C. Chamberlain4Alice L.-F. Mui5Immunity and Infection Research Centre, Vancouver Coastal Health Research Institute, Vancouver, CanadaImmunity and Infection Research Centre, Vancouver Coastal Health Research Institute, Vancouver, CanadaDepartment of Surgery, University of British Columbia, Vancouver, CanadaVancouver Prostate Centre and Department of Urologic Sciences, University of British Columbia, Vancouver, BC, CanadaImmunity and Infection Research Centre, Vancouver Coastal Health Research Institute, Vancouver, CanadaImmunity and Infection Research Centre, Vancouver Coastal Health Research Institute, Vancouver, CanadaInterleukin-10 (IL10) is best studied for its inhibitory action on immune cells and ability to suppress an antitumour immune response. But IL10 also exerts direct effects on nonimmune cells such as prostate cancer epithelial cells. Elevated serum levels of IL10 observed in prostate and other cancer patients are associated with poor prognosis. After first-line androgen-deprivation therapy, prostate cancer patients are treated with androgen receptor antagonists such as enzalutamide to inhibit androgen-dependent prostate cancer cell growth. However, development of resistance inevitably occurs and this is associated with tumour differentiation to more aggressive forms such as a neuroendocrine phenotype characterized by expression of neuron specific enolase and synaptophysin. We found that treatment of prostate cancer cell lines in vitro with IL10 or enzalutamide induced markers of neuroendocrine differentiation and inhibited androgen receptor reporter activity. Both also upregulated the levels of PDL1, which could promote tumour survival in vivo through its interaction with the immune cell inhibitory receptor PD1 to suppress antitumour immunity. These findings suggest that IL10’s direct action on prostate cancer cells could contribute to prostate cancer progression independent of IL10’s suppression of host immune cells.http://dx.doi.org/10.1155/2020/5305306
spellingShingle Abrar Samiea
Jeff S. J. Yoon
Christopher J. Ong
Amina Zoubeidi
Thomas C. Chamberlain
Alice L.-F. Mui
Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer Cells
Prostate Cancer
title Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer Cells
title_full Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer Cells
title_fullStr Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer Cells
title_full_unstemmed Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer Cells
title_short Interleukin-10 Induces Expression of Neuroendocrine Markers and PDL1 in Prostate Cancer Cells
title_sort interleukin 10 induces expression of neuroendocrine markers and pdl1 in prostate cancer cells
url http://dx.doi.org/10.1155/2020/5305306
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