Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.

Evolution of the highly successful and multidrug resistant clone ST111 in Pseudomonas aeruginosa involves serotype switching from O-antigen O4 to O12. How expression of a different O-antigen serotype alters pathogen physiology to enable global dissemination of this high-risk clone-type is not unders...

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Main Authors: Mikkel Anbo, Mahbuba Akter Lubna, Dina A Moustafa, Telmo O Paiva, Laura Serioli, Kinga Zor, Claus Sternberg, Katy Jeannot, Oana Ciofu, Yves F Dufrêne, Joanna B Goldberg, Lars Jelsbak
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2024-12-01
Series:PLoS Pathogens
Online Access:https://doi.org/10.1371/journal.ppat.1012221
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author Mikkel Anbo
Mahbuba Akter Lubna
Dina A Moustafa
Telmo O Paiva
Laura Serioli
Kinga Zor
Claus Sternberg
Katy Jeannot
Oana Ciofu
Yves F Dufrêne
Joanna B Goldberg
Lars Jelsbak
author_facet Mikkel Anbo
Mahbuba Akter Lubna
Dina A Moustafa
Telmo O Paiva
Laura Serioli
Kinga Zor
Claus Sternberg
Katy Jeannot
Oana Ciofu
Yves F Dufrêne
Joanna B Goldberg
Lars Jelsbak
author_sort Mikkel Anbo
collection DOAJ
description Evolution of the highly successful and multidrug resistant clone ST111 in Pseudomonas aeruginosa involves serotype switching from O-antigen O4 to O12. How expression of a different O-antigen serotype alters pathogen physiology to enable global dissemination of this high-risk clone-type is not understood. Here, we engineered isogenic laboratory and clinical P. aeruginosa strains that express the different O-antigen gene clusters to assess the correlation of structural differences of O4 and O12 O-antigens to pathogen-relevant phenotypic traits. We show that serotype O12 is associated with enhanced adhesion, type IV pili dependent twitching motility, and tolerance to host defense molecules and serum. Moreover, we find that serotype O4 is less virulent compared to O12 in an acute murine pneumonia infection in terms of both colonization and survival rate. Finally, we find that these O-antigen effects may be explained by specific biophysical properties of the serotype repeat unit found in O4 and O12, and by differences in membrane stability between O4 and O12 expressing cells. The results demonstrate that differences in O-antigen sugar composition can affect P. aeruginosa pathogenicity traits, and provide a better understanding of the potential selective advantages that underlie serotype switching and emergence of serotype O12 ST111.
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spelling doaj-art-bc2c724bdab94b2d950c2e4840242ad42025-08-20T02:49:01ZengPublic Library of Science (PLoS)PLoS Pathogens1553-73661553-73742024-12-012012e101222110.1371/journal.ppat.1012221Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.Mikkel AnboMahbuba Akter LubnaDina A MoustafaTelmo O PaivaLaura SerioliKinga ZorClaus SternbergKaty JeannotOana CiofuYves F DufrêneJoanna B GoldbergLars JelsbakEvolution of the highly successful and multidrug resistant clone ST111 in Pseudomonas aeruginosa involves serotype switching from O-antigen O4 to O12. How expression of a different O-antigen serotype alters pathogen physiology to enable global dissemination of this high-risk clone-type is not understood. Here, we engineered isogenic laboratory and clinical P. aeruginosa strains that express the different O-antigen gene clusters to assess the correlation of structural differences of O4 and O12 O-antigens to pathogen-relevant phenotypic traits. We show that serotype O12 is associated with enhanced adhesion, type IV pili dependent twitching motility, and tolerance to host defense molecules and serum. Moreover, we find that serotype O4 is less virulent compared to O12 in an acute murine pneumonia infection in terms of both colonization and survival rate. Finally, we find that these O-antigen effects may be explained by specific biophysical properties of the serotype repeat unit found in O4 and O12, and by differences in membrane stability between O4 and O12 expressing cells. The results demonstrate that differences in O-antigen sugar composition can affect P. aeruginosa pathogenicity traits, and provide a better understanding of the potential selective advantages that underlie serotype switching and emergence of serotype O12 ST111.https://doi.org/10.1371/journal.ppat.1012221
spellingShingle Mikkel Anbo
Mahbuba Akter Lubna
Dina A Moustafa
Telmo O Paiva
Laura Serioli
Kinga Zor
Claus Sternberg
Katy Jeannot
Oana Ciofu
Yves F Dufrêne
Joanna B Goldberg
Lars Jelsbak
Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.
PLoS Pathogens
title Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.
title_full Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.
title_fullStr Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.
title_full_unstemmed Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.
title_short Serotype switching in Pseudomonas aeruginosa ST111 enhances adhesion and virulence.
title_sort serotype switching in pseudomonas aeruginosa st111 enhances adhesion and virulence
url https://doi.org/10.1371/journal.ppat.1012221
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