Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch Activation

Epsins are part of the internalization machinery pivotal to control clathrin-mediated endocytosis. Here, we report that epsin family members are expressed in mouse embryonic stem cells (mESCs) and that epsin1/2 knockdown alters both mESC exits from pluripotency and their differentiation. Furthermore...

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Main Authors: Marina Cardano, Jacopo Zasso, Luca Ruggiero, Giuseppina Di Giacomo, Matteo Marcatili, Ottavio Cremona, Luciano Conti
Format: Article
Language:English
Published: Wiley 2019-01-01
Series:Stem Cells International
Online Access:http://dx.doi.org/10.1155/2019/4084351
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author Marina Cardano
Jacopo Zasso
Luca Ruggiero
Giuseppina Di Giacomo
Matteo Marcatili
Ottavio Cremona
Luciano Conti
author_facet Marina Cardano
Jacopo Zasso
Luca Ruggiero
Giuseppina Di Giacomo
Matteo Marcatili
Ottavio Cremona
Luciano Conti
author_sort Marina Cardano
collection DOAJ
description Epsins are part of the internalization machinery pivotal to control clathrin-mediated endocytosis. Here, we report that epsin family members are expressed in mouse embryonic stem cells (mESCs) and that epsin1/2 knockdown alters both mESC exits from pluripotency and their differentiation. Furthermore, we show that epsin1/2 knockdown compromises the correct polarization and division of mESC-derived neural progenitors and their conversion into expandable radial glia-like neural stem cells. Finally, we provide evidence that Notch signaling is impaired following epsin1/2 knockdown and that experimental restoration of Notch signaling rescues the epsin-mediated phenotypes. We conclude that epsins contribute to control mESC exit from pluripotency and allow their neural differentiation by appropriate modulation of Notch signaling.
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publishDate 2019-01-01
publisher Wiley
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spelling doaj-art-bc1037637f26433b879ca7cf2cfec26d2025-08-20T02:18:43ZengWileyStem Cells International1687-966X1687-96782019-01-01201910.1155/2019/40843514084351Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch ActivationMarina Cardano0Jacopo Zasso1Luca Ruggiero2Giuseppina Di Giacomo3Matteo Marcatili4Ottavio Cremona5Luciano Conti6Laboratory of Stem Cell Biology, Department of Cellular, Computational and Integrative Biology (CIBIO), Università degli Studi di Trento, Trento, ItalyLaboratory of Stem Cell Biology, Department of Cellular, Computational and Integrative Biology (CIBIO), Università degli Studi di Trento, Trento, ItalyUniversità Vita Salute San Raffaele, Ospedale San Raffaele, Division of Neuroscience, Milan, ItalyUniversità Vita Salute San Raffaele, Ospedale San Raffaele, Division of Neuroscience, Milan, ItalyLaboratory of Stem Cell Biology, Department of Cellular, Computational and Integrative Biology (CIBIO), Università degli Studi di Trento, Trento, ItalyUniversità Vita Salute San Raffaele, Ospedale San Raffaele, Division of Neuroscience, Milan, ItalyLaboratory of Stem Cell Biology, Department of Cellular, Computational and Integrative Biology (CIBIO), Università degli Studi di Trento, Trento, ItalyEpsins are part of the internalization machinery pivotal to control clathrin-mediated endocytosis. Here, we report that epsin family members are expressed in mouse embryonic stem cells (mESCs) and that epsin1/2 knockdown alters both mESC exits from pluripotency and their differentiation. Furthermore, we show that epsin1/2 knockdown compromises the correct polarization and division of mESC-derived neural progenitors and their conversion into expandable radial glia-like neural stem cells. Finally, we provide evidence that Notch signaling is impaired following epsin1/2 knockdown and that experimental restoration of Notch signaling rescues the epsin-mediated phenotypes. We conclude that epsins contribute to control mESC exit from pluripotency and allow their neural differentiation by appropriate modulation of Notch signaling.http://dx.doi.org/10.1155/2019/4084351
spellingShingle Marina Cardano
Jacopo Zasso
Luca Ruggiero
Giuseppina Di Giacomo
Matteo Marcatili
Ottavio Cremona
Luciano Conti
Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch Activation
Stem Cells International
title Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch Activation
title_full Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch Activation
title_fullStr Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch Activation
title_full_unstemmed Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch Activation
title_short Epsins Regulate Mouse Embryonic Stem Cell Exit from Pluripotency and Neural Commitment by Controlling Notch Activation
title_sort epsins regulate mouse embryonic stem cell exit from pluripotency and neural commitment by controlling notch activation
url http://dx.doi.org/10.1155/2019/4084351
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