The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagy

Acute kidney injury (AKI) is a significant issue in public health, displaying a high occurrence rate and mortality rate. Ferroptosis, a form of programmed cell death (PCD), is characterized by iron accumulation and intensified lipid peroxidation. Recent studies have demonstrated the pivotal signific...

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Main Authors: Chunyu Pan, Hairui Zhao, Xiaojing Cai, Manyi Wu, Bowen Qin, Junhua Li
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Renal Failure
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Online Access:https://www.tandfonline.com/doi/10.1080/0886022X.2024.2379601
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author Chunyu Pan
Hairui Zhao
Xiaojing Cai
Manyi Wu
Bowen Qin
Junhua Li
author_facet Chunyu Pan
Hairui Zhao
Xiaojing Cai
Manyi Wu
Bowen Qin
Junhua Li
author_sort Chunyu Pan
collection DOAJ
description Acute kidney injury (AKI) is a significant issue in public health, displaying a high occurrence rate and mortality rate. Ferroptosis, a form of programmed cell death (PCD), is characterized by iron accumulation and intensified lipid peroxidation. Recent studies have demonstrated the pivotal significance of ferroptosis in AKI caused by diverse stimuli, including ischemia-reperfusion injury (IRI), sepsis and toxins. Autophagy, a multistep process that targets damaged organelles and macromolecules for degradation and recycling, also plays an essential role in AKI. Previous research has demonstrated that autophagy deletion in proximal tubules could aggravate tubular injury and renal function loss, indicating the protective function of autophagy in AKI. Consequently, finding ways to stimulate autophagy has become a crucial therapeutic strategy. The recent discovery of the role of selective autophagy in influencing ferroptosis has identified new therapeutic targets for AKI and has highlighted the importance of understanding the cross-talk between autophagy and ferroptosis. This study aims to provide an overview of the signaling pathways involved in ferroptosis and autophagy, focusing on the mechanisms and functions of selective autophagy and autophagy-dependent ferroptosis. We hope to establish a foundation for future investigations into the interaction between autophagy and ferroptosis in AKI as well as other diseases.
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issn 0886-022X
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publishDate 2024-12-01
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series Renal Failure
spelling doaj-art-bb292e45f14641c48bf441efe1e822da2025-08-20T03:05:26ZengTaylor & Francis GroupRenal Failure0886-022X1525-60492024-12-0146210.1080/0886022X.2024.2379601The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagyChunyu Pan0Hairui Zhao1Xiaojing Cai2Manyi Wu3Bowen Qin4Junhua Li5Department of Nephrology, Tongji Hospital Affiliated to Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaDepartment of Nephrology, Tianyou Hospital, Wuhan University of Science and Technology, Wuhan, ChinaDepartment of Nephrology, Tongji Hospital Affiliated to Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaDepartment of Nephrology, Tongji Hospital Affiliated to Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaDepartment of Nephrology, Tianyou Hospital, Wuhan University of Science and Technology, Wuhan, ChinaDepartment of Nephrology, Tongji Hospital Affiliated to Tongji Medical College, Huazhong University of Science and Technology, Wuhan, ChinaAcute kidney injury (AKI) is a significant issue in public health, displaying a high occurrence rate and mortality rate. Ferroptosis, a form of programmed cell death (PCD), is characterized by iron accumulation and intensified lipid peroxidation. Recent studies have demonstrated the pivotal significance of ferroptosis in AKI caused by diverse stimuli, including ischemia-reperfusion injury (IRI), sepsis and toxins. Autophagy, a multistep process that targets damaged organelles and macromolecules for degradation and recycling, also plays an essential role in AKI. Previous research has demonstrated that autophagy deletion in proximal tubules could aggravate tubular injury and renal function loss, indicating the protective function of autophagy in AKI. Consequently, finding ways to stimulate autophagy has become a crucial therapeutic strategy. The recent discovery of the role of selective autophagy in influencing ferroptosis has identified new therapeutic targets for AKI and has highlighted the importance of understanding the cross-talk between autophagy and ferroptosis. This study aims to provide an overview of the signaling pathways involved in ferroptosis and autophagy, focusing on the mechanisms and functions of selective autophagy and autophagy-dependent ferroptosis. We hope to establish a foundation for future investigations into the interaction between autophagy and ferroptosis in AKI as well as other diseases.https://www.tandfonline.com/doi/10.1080/0886022X.2024.2379601Autophagyferroptosisselective autophagyAKIferritinophagy
spellingShingle Chunyu Pan
Hairui Zhao
Xiaojing Cai
Manyi Wu
Bowen Qin
Junhua Li
The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagy
Renal Failure
Autophagy
ferroptosis
selective autophagy
AKI
ferritinophagy
title The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagy
title_full The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagy
title_fullStr The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagy
title_full_unstemmed The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagy
title_short The connection between autophagy and ferroptosis in AKI: recent advances regarding selective autophagy
title_sort connection between autophagy and ferroptosis in aki recent advances regarding selective autophagy
topic Autophagy
ferroptosis
selective autophagy
AKI
ferritinophagy
url https://www.tandfonline.com/doi/10.1080/0886022X.2024.2379601
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