PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research

Abstract Attention‐deficit/hyperactivity disorder (ADHD) is a developmental disorder characterized by hyperactivity, inattention, and impulsive behaviors and has significant societal impact. ADHD is recognized as a heterogeneous disease, and genetic and/or environmental factors underlying pathogenes...

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Main Authors: Emmanuel Darcq, Brigitte L Kieffer
Format: Article
Language:English
Published: Springer Nature 2015-04-01
Series:EMBO Molecular Medicine
Online Access:https://doi.org/10.15252/emmm.201505266
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author Emmanuel Darcq
Brigitte L Kieffer
author_facet Emmanuel Darcq
Brigitte L Kieffer
author_sort Emmanuel Darcq
collection DOAJ
description Abstract Attention‐deficit/hyperactivity disorder (ADHD) is a developmental disorder characterized by hyperactivity, inattention, and impulsive behaviors and has significant societal impact. ADHD is recognized as a heterogeneous disease, and genetic and/or environmental factors underlying pathogenesis remain largely unknown. There is an obvious need to increase knowledge on molecular signaling and brain pathways underlying disease development, and genetic mouse models are key to this goal. In this issue of EMBO Molecular Medicine, D'Andrea et al (2015) combine state‐of‐the‐art genetic and behavioral approaches in the mouse to demonstrate an essential role for PI3Kγ and cAMP homeostasis in ADHD‐related behaviors, through signaling mechanisms operating at the level of the locus coeruleus, the main source of noradrenaline in the brain. Furthermore, the study posits PI3Kγ knockout mice as a novel tool of high interest for modeling ADHD endophenotypes.
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spelling doaj-art-ba6bc9a03ff34a70a637e456e9b44c012025-08-20T03:05:55ZengSpringer NatureEMBO Molecular Medicine1757-46761757-46842015-04-017785986110.15252/emmm.201505266PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD researchEmmanuel Darcq0Brigitte L Kieffer1Department of Psychiatry, Faculty of Medicine, Douglas Hospital Research Center, McGill UniversityDepartment of Psychiatry, Faculty of Medicine, Douglas Hospital Research Center, McGill UniversityAbstract Attention‐deficit/hyperactivity disorder (ADHD) is a developmental disorder characterized by hyperactivity, inattention, and impulsive behaviors and has significant societal impact. ADHD is recognized as a heterogeneous disease, and genetic and/or environmental factors underlying pathogenesis remain largely unknown. There is an obvious need to increase knowledge on molecular signaling and brain pathways underlying disease development, and genetic mouse models are key to this goal. In this issue of EMBO Molecular Medicine, D'Andrea et al (2015) combine state‐of‐the‐art genetic and behavioral approaches in the mouse to demonstrate an essential role for PI3Kγ and cAMP homeostasis in ADHD‐related behaviors, through signaling mechanisms operating at the level of the locus coeruleus, the main source of noradrenaline in the brain. Furthermore, the study posits PI3Kγ knockout mice as a novel tool of high interest for modeling ADHD endophenotypes.https://doi.org/10.15252/emmm.201505266
spellingShingle Emmanuel Darcq
Brigitte L Kieffer
PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research
EMBO Molecular Medicine
title PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research
title_full PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research
title_fullStr PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research
title_full_unstemmed PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research
title_short PI3K signaling in the locus coeruleus: a new molecular pathway for ADHD research
title_sort pi3k signaling in the locus coeruleus a new molecular pathway for adhd research
url https://doi.org/10.15252/emmm.201505266
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