Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophages

Metabolic diseases have gradually become one of the most significant global medical burdens. Diseases such as obesity, diabetes, and metabolic syndrome, along with their complications, are clinically categorized as metabolic diseases. Long-term oral medication significantly reduces patient complianc...

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Main Authors: Liang Zhang, Kecheng Lou, Yunmeng Zhang, Yuanjing Leng, Yuqing Huang, Xinxin Liao, Xiaoliang Liu, Shangzhi Feng, Guoqiang Feng
Format: Article
Language:English
Published: Frontiers Media S.A. 2024-12-01
Series:Frontiers in Endocrinology
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Online Access:https://www.frontiersin.org/articles/10.3389/fendo.2024.1510712/full
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author Liang Zhang
Kecheng Lou
Yunmeng Zhang
Yuanjing Leng
Yuqing Huang
Xinxin Liao
Xiaoliang Liu
Shangzhi Feng
Guoqiang Feng
author_facet Liang Zhang
Kecheng Lou
Yunmeng Zhang
Yuanjing Leng
Yuqing Huang
Xinxin Liao
Xiaoliang Liu
Shangzhi Feng
Guoqiang Feng
author_sort Liang Zhang
collection DOAJ
description Metabolic diseases have gradually become one of the most significant global medical burdens. Diseases such as obesity, diabetes, and metabolic syndrome, along with their complications, are clinically categorized as metabolic diseases. Long-term oral medication significantly reduces patient compliance and quality of life. Therefore, alternative therapies that intervene at the cellular level or target the root causes of metabolic diseases might help change this predicament. Research has found that extracellular vesicles derived from adipose macrophages can effectively regulate metabolic diseases by influencing the disease’s development. This regulation is likely related to the role of these extracellular vesicles as important mediators in modulating adipose tissue function and insulin sensitivity, and their involvement in the crosstalk between adipocytes and macrophages. This review aims to describe the regulation of metabolic diseases mediated by adipose macrophage-derived extracellular vesicles, with a focus on their involvement in adipocyte crosstalk, the regulation of metabolism-related autoimmunity, and their potential as therapeutic agents for metabolic diseases, providing new avenues for diagnosis and treatment.
format Article
id doaj-art-b9a21e3f49ae48fea2dac8809d39152e
institution DOAJ
issn 1664-2392
language English
publishDate 2024-12-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Endocrinology
spelling doaj-art-b9a21e3f49ae48fea2dac8809d39152e2025-08-20T02:56:58ZengFrontiers Media S.A.Frontiers in Endocrinology1664-23922024-12-011510.3389/fendo.2024.15107121510712Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophagesLiang Zhang0Kecheng Lou1Yunmeng Zhang2Yuanjing Leng3Yuqing Huang4Xinxin Liao5Xiaoliang Liu6Shangzhi Feng7Guoqiang Feng8Department of Urology, Jiujiang University Clinic College/Hospital, Jiujiang, Jiangxi, ChinaDepartment of Urology, Lanxi People’s Hospital, Jinhua, Zhejiang, ChinaDepartment of Anesthesiology, Jiujiang College Hospital, Jiujiang, Jiangxi, ChinaDepartment of Urology, Jiujiang University Clinic College/Hospital, Jiujiang, Jiangxi, ChinaDepartment of Urology, Jiujiang University Clinic College/Hospital, Jiujiang, Jiangxi, ChinaDepartment of Urology, Jiujiang University Clinic College/Hospital, Jiujiang, Jiangxi, ChinaDepartment of Urology, Jiujiang University Clinic College/Hospital, Jiujiang, Jiangxi, ChinaDepartment of Urology, Jiujiang University Clinic College/Hospital, Jiujiang, Jiangxi, ChinaDepartment of Rehabilitation, Jiujiang College Hospital, Jiujiang, Jiangxi, ChinaMetabolic diseases have gradually become one of the most significant global medical burdens. Diseases such as obesity, diabetes, and metabolic syndrome, along with their complications, are clinically categorized as metabolic diseases. Long-term oral medication significantly reduces patient compliance and quality of life. Therefore, alternative therapies that intervene at the cellular level or target the root causes of metabolic diseases might help change this predicament. Research has found that extracellular vesicles derived from adipose macrophages can effectively regulate metabolic diseases by influencing the disease’s development. This regulation is likely related to the role of these extracellular vesicles as important mediators in modulating adipose tissue function and insulin sensitivity, and their involvement in the crosstalk between adipocytes and macrophages. This review aims to describe the regulation of metabolic diseases mediated by adipose macrophage-derived extracellular vesicles, with a focus on their involvement in adipocyte crosstalk, the regulation of metabolism-related autoimmunity, and their potential as therapeutic agents for metabolic diseases, providing new avenues for diagnosis and treatment.https://www.frontiersin.org/articles/10.3389/fendo.2024.1510712/fullobesitymetabolic diseaseadipose tissue macrophageextracellular vesiclesnano-targeted therapy
spellingShingle Liang Zhang
Kecheng Lou
Yunmeng Zhang
Yuanjing Leng
Yuqing Huang
Xinxin Liao
Xiaoliang Liu
Shangzhi Feng
Guoqiang Feng
Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophages
Frontiers in Endocrinology
obesity
metabolic disease
adipose tissue macrophage
extracellular vesicles
nano-targeted therapy
title Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophages
title_full Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophages
title_fullStr Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophages
title_full_unstemmed Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophages
title_short Tools for regulating metabolic diseases: extracellular vesicles from adipose macrophages
title_sort tools for regulating metabolic diseases extracellular vesicles from adipose macrophages
topic obesity
metabolic disease
adipose tissue macrophage
extracellular vesicles
nano-targeted therapy
url https://www.frontiersin.org/articles/10.3389/fendo.2024.1510712/full
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