Urinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal function

Abstract Type 2 diabetes mellitus (T2DM) is closely associated with an increased risk and adverse event of acute coronary syndrome (ACS). The present study aims to investigate the association between differential urinary metabolites and major adverse cardiovascular events (MACEs) in patients with AC...

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Main Authors: Mingyu Duan, Zhihan Zhang, Ruhua He, Yameng Zhang, Xueying Zhang, Miradil Dilshat, Jun He
Format: Article
Language:English
Published: Nature Portfolio 2025-04-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-96010-w
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author Mingyu Duan
Zhihan Zhang
Ruhua He
Yameng Zhang
Xueying Zhang
Miradil Dilshat
Jun He
author_facet Mingyu Duan
Zhihan Zhang
Ruhua He
Yameng Zhang
Xueying Zhang
Miradil Dilshat
Jun He
author_sort Mingyu Duan
collection DOAJ
description Abstract Type 2 diabetes mellitus (T2DM) is closely associated with an increased risk and adverse event of acute coronary syndrome (ACS). The present study aims to investigate the association between differential urinary metabolites and major adverse cardiovascular events (MACEs) in patients with ACS co-morbid T2DM with preserved renal function, and to explore the potential value of the metabolites as prognostic biomarkers in this population. Ultra-high performance liquid chromatography-mass spectrometry (UHPLC/MS) was used to analyze urine samples from ACS co-morbid T2DM. Spearman’s correlation was used to examine the association between differential metabolites and serum fasting blood glucose (FBG), glycated hemoglobin (HbA1c), Syntax score I, and MACE. The Cox proportional hazards models and Kaplan–Meier survival curves were used to identify MACE risk factors. A total of 101 differential urinary metabolites were identified, of which seven showed a correlation with FBG, HbA1c, Syntax score I and MACE. In particular, myo-inositol and (E)-Monocrotophos emerged as significant indicators of poor prognosis in ACS co-morbid with T2DM. Urinary metabolomic alteration is closely associated with clinical manifestation of ACS co-morbid T2DM. Urinary myo-inositol and (E)-Monocrotophos may be considered as prognostic biomarkers of ACS co-morbid T2DM.
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spelling doaj-art-b94eb29d2a5a4e8bada0b1bb3af410972025-08-20T02:27:53ZengNature PortfolioScientific Reports2045-23222025-04-0115111410.1038/s41598-025-96010-wUrinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal functionMingyu Duan0Zhihan Zhang1Ruhua He2Yameng Zhang3Xueying Zhang4Miradil Dilshat5Jun He6Department of Ultrasound of Jiangbei Campus, The First Affiliated Hospital of Army Medical University (The 958th Hospital of Chinese People‘s Liberation Army)Department of Cardiology, Hanzhong Central HospitalDepartment of Cardiology, General Hospital of Ningxia Medical UniversityDepartment of Cardiology, The Second Affiliated Hospital of Henan University of Science and TechnologyNingxia Medical UniversityNingxia Medical UniversityDepartment of Cardiology, General Hospital of Ningxia Medical UniversityAbstract Type 2 diabetes mellitus (T2DM) is closely associated with an increased risk and adverse event of acute coronary syndrome (ACS). The present study aims to investigate the association between differential urinary metabolites and major adverse cardiovascular events (MACEs) in patients with ACS co-morbid T2DM with preserved renal function, and to explore the potential value of the metabolites as prognostic biomarkers in this population. Ultra-high performance liquid chromatography-mass spectrometry (UHPLC/MS) was used to analyze urine samples from ACS co-morbid T2DM. Spearman’s correlation was used to examine the association between differential metabolites and serum fasting blood glucose (FBG), glycated hemoglobin (HbA1c), Syntax score I, and MACE. The Cox proportional hazards models and Kaplan–Meier survival curves were used to identify MACE risk factors. A total of 101 differential urinary metabolites were identified, of which seven showed a correlation with FBG, HbA1c, Syntax score I and MACE. In particular, myo-inositol and (E)-Monocrotophos emerged as significant indicators of poor prognosis in ACS co-morbid with T2DM. Urinary metabolomic alteration is closely associated with clinical manifestation of ACS co-morbid T2DM. Urinary myo-inositol and (E)-Monocrotophos may be considered as prognostic biomarkers of ACS co-morbid T2DM.https://doi.org/10.1038/s41598-025-96010-wAcute coronary syndromeType 2 diabetes mellitusMetabolomicsMajor adverse cardiovascular eventsPrognosis
spellingShingle Mingyu Duan
Zhihan Zhang
Ruhua He
Yameng Zhang
Xueying Zhang
Miradil Dilshat
Jun He
Urinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal function
Scientific Reports
Acute coronary syndrome
Type 2 diabetes mellitus
Metabolomics
Major adverse cardiovascular events
Prognosis
title Urinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal function
title_full Urinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal function
title_fullStr Urinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal function
title_full_unstemmed Urinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal function
title_short Urinary metabolomics reveals myo-inositol and (E)-Monocrotophos associate with prognosis in ACS comorbid with T2DM and preserved renal function
title_sort urinary metabolomics reveals myo inositol and e monocrotophos associate with prognosis in acs comorbid with t2dm and preserved renal function
topic Acute coronary syndrome
Type 2 diabetes mellitus
Metabolomics
Major adverse cardiovascular events
Prognosis
url https://doi.org/10.1038/s41598-025-96010-w
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