Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.

The essential mammalian gene TACC3 is frequently mutated and amplified in cancers and its fusion products exhibit oncogenic activity in glioblastomas. TACC3 functions in mitotic spindle assembly and chromosome segregation. In particular, phosphorylation on S558 by the mitotic kinase, Aurora-A, promo...

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Main Authors: Selena G Burgess, Isabel Peset, Nimesh Joseph, Tommaso Cavazza, Isabelle Vernos, Mark Pfuhl, Fanni Gergely, Richard Bayliss
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2015-07-01
Series:PLoS Genetics
Online Access:https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1005345&type=printable
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author Selena G Burgess
Isabel Peset
Nimesh Joseph
Tommaso Cavazza
Isabelle Vernos
Mark Pfuhl
Fanni Gergely
Richard Bayliss
author_facet Selena G Burgess
Isabel Peset
Nimesh Joseph
Tommaso Cavazza
Isabelle Vernos
Mark Pfuhl
Fanni Gergely
Richard Bayliss
author_sort Selena G Burgess
collection DOAJ
description The essential mammalian gene TACC3 is frequently mutated and amplified in cancers and its fusion products exhibit oncogenic activity in glioblastomas. TACC3 functions in mitotic spindle assembly and chromosome segregation. In particular, phosphorylation on S558 by the mitotic kinase, Aurora-A, promotes spindle recruitment of TACC3 and triggers the formation of a complex with ch-TOG-clathrin that crosslinks and stabilises kinetochore microtubules. Here we map the Aurora-A-binding interface in TACC3 and show that TACC3 potently activates Aurora-A through a domain centered on F525. Vertebrate cells carrying homozygous F525A mutation in the endogenous TACC3 loci exhibit defects in TACC3 function, namely perturbed localization, reduced phosphorylation and weakened interaction with clathrin. The most striking feature of the F525A cells however is a marked shortening of mitosis, at least in part due to rapid spindle assembly. F525A cells do not exhibit chromosome missegregation, indicating that they undergo fast yet apparently faithful mitosis. By contrast, mutating the phosphorylation site S558 to alanine in TACC3 causes aneuploidy without a significant change in mitotic duration. Our work has therefore defined a regulatory role for the Aurora-A-TACC3 interaction beyond the act of phosphorylation at S558. We propose that the regulatory relationship between Aurora-A and TACC3 enables the transition from the microtubule-polymerase activity of TACC3-ch-TOG to the microtubule-crosslinking activity of TACC3-ch-TOG-clathrin complexes as mitosis progresses. Aurora-A-dependent control of TACC3 could determine the balance between these activities, thereby influencing not only spindle length and stability but also the speed of spindle formation with vital consequences for chromosome alignment and segregation.
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spelling doaj-art-b5677532a490461bba5d2a583ccf56d72025-08-20T02:22:40ZengPublic Library of Science (PLoS)PLoS Genetics1553-73901553-74042015-07-01117e100534510.1371/journal.pgen.1005345Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.Selena G BurgessIsabel PesetNimesh JosephTommaso CavazzaIsabelle VernosMark PfuhlFanni GergelyRichard BaylissThe essential mammalian gene TACC3 is frequently mutated and amplified in cancers and its fusion products exhibit oncogenic activity in glioblastomas. TACC3 functions in mitotic spindle assembly and chromosome segregation. In particular, phosphorylation on S558 by the mitotic kinase, Aurora-A, promotes spindle recruitment of TACC3 and triggers the formation of a complex with ch-TOG-clathrin that crosslinks and stabilises kinetochore microtubules. Here we map the Aurora-A-binding interface in TACC3 and show that TACC3 potently activates Aurora-A through a domain centered on F525. Vertebrate cells carrying homozygous F525A mutation in the endogenous TACC3 loci exhibit defects in TACC3 function, namely perturbed localization, reduced phosphorylation and weakened interaction with clathrin. The most striking feature of the F525A cells however is a marked shortening of mitosis, at least in part due to rapid spindle assembly. F525A cells do not exhibit chromosome missegregation, indicating that they undergo fast yet apparently faithful mitosis. By contrast, mutating the phosphorylation site S558 to alanine in TACC3 causes aneuploidy without a significant change in mitotic duration. Our work has therefore defined a regulatory role for the Aurora-A-TACC3 interaction beyond the act of phosphorylation at S558. We propose that the regulatory relationship between Aurora-A and TACC3 enables the transition from the microtubule-polymerase activity of TACC3-ch-TOG to the microtubule-crosslinking activity of TACC3-ch-TOG-clathrin complexes as mitosis progresses. Aurora-A-dependent control of TACC3 could determine the balance between these activities, thereby influencing not only spindle length and stability but also the speed of spindle formation with vital consequences for chromosome alignment and segregation.https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1005345&type=printable
spellingShingle Selena G Burgess
Isabel Peset
Nimesh Joseph
Tommaso Cavazza
Isabelle Vernos
Mark Pfuhl
Fanni Gergely
Richard Bayliss
Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.
PLoS Genetics
title Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.
title_full Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.
title_fullStr Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.
title_full_unstemmed Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.
title_short Aurora-A-Dependent Control of TACC3 Influences the Rate of Mitotic Spindle Assembly.
title_sort aurora a dependent control of tacc3 influences the rate of mitotic spindle assembly
url https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1005345&type=printable
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