Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 cases

Introduction: Out of all non-communicable diseases, cancer is the leading cause of death in the 21st century. Oral cancer ranks in top ten cancers in the world and oncogenic viruses contribute to its development and are a major preventable risk factor for it. Material and methods: In the present stu...

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Main Authors: Shahi Vanshika, Agarwal Preeti, Qayoom Sumaira, Kumar Vijay, Tewari Shikha, Raghuvanshi Shivanjali, Singh Uma Shankar, Goel Madhu Mati
Format: Article
Language:English
Published: Elsevier 2021-04-01
Series:Journal of Oral Biology and Craniofacial Research
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Online Access:http://www.sciencedirect.com/science/article/pii/S2212426821000075
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author Shahi Vanshika
Agarwal Preeti
Qayoom Sumaira
Kumar Vijay
Tewari Shikha
Raghuvanshi Shivanjali
Singh Uma Shankar
Goel Madhu Mati
author_facet Shahi Vanshika
Agarwal Preeti
Qayoom Sumaira
Kumar Vijay
Tewari Shikha
Raghuvanshi Shivanjali
Singh Uma Shankar
Goel Madhu Mati
author_sort Shahi Vanshika
collection DOAJ
description Introduction: Out of all non-communicable diseases, cancer is the leading cause of death in the 21st century. Oral cancer ranks in top ten cancers in the world and oncogenic viruses contribute to its development and are a major preventable risk factor for it. Material and methods: In the present study we intend to find the incidence of HPV and EBV by PCR in 108 sporadic oral cancer patients and study the clinico-pathological variables in agreement to their presence or absence. Results: We found that the incidence of EBV in oral cancer is much higher (30/108; 27.8%) than HPV 16 (14/108; 13%) and, a complete absence of HPV 18 (0/108) was reported by real time PCR. Co-infection by EBV and HPV was reported in 5.6% cases (6/108). However, on comparing this data with the corresponding clinico-pathological cofactors (age, gender, grade of tumour, tumour size, nodal status, metastasis, DOI, TIL, ENE) it was found that there was no statistical significance between the two. Conclusion: Hence, we conclude that these oncogenic viruses are independent cofactors to oral cancer development and none of the clinico-morphologic variable is associated with the viral etiology.
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issn 2212-4268
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publishDate 2021-04-01
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spelling doaj-art-b53c659585fa425b8786071bd19ad2cc2025-08-20T02:50:26ZengElsevierJournal of Oral Biology and Craniofacial Research2212-42682021-04-0111218018410.1016/j.jobcr.2021.01.007Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 casesShahi Vanshika0Agarwal Preeti1Qayoom Sumaira2Kumar Vijay3Tewari Shikha4Raghuvanshi Shivanjali5Singh Uma Shankar6Goel Madhu Mati7Department of Pathology, King George’s Medical University, Lucknow, UP, IndiaDepartment of Pathology, King George’s Medical University, Lucknow, UP, India; Corresponding author. Department of Pathology , KGMU, Lucknow, 226003, UP, India.Department of Pathology, King George’s Medical University, Lucknow, UP, IndiaDepartment of Surgical Oncology, King George’s Medical University, Lucknow, UP, IndiaDepartment of Laboratory Medicine and Pathology, Medanta Hospital, Lucknow, UP, IndiaDepartment of Pathology, King George’s Medical University, Lucknow, UP, IndiaDepartment of Pathology, King George’s Medical University, Lucknow, UP, IndiaDepartment of Laboratory Medicine and Pathology, Medanta Hospital, Lucknow, UP, IndiaIntroduction: Out of all non-communicable diseases, cancer is the leading cause of death in the 21st century. Oral cancer ranks in top ten cancers in the world and oncogenic viruses contribute to its development and are a major preventable risk factor for it. Material and methods: In the present study we intend to find the incidence of HPV and EBV by PCR in 108 sporadic oral cancer patients and study the clinico-pathological variables in agreement to their presence or absence. Results: We found that the incidence of EBV in oral cancer is much higher (30/108; 27.8%) than HPV 16 (14/108; 13%) and, a complete absence of HPV 18 (0/108) was reported by real time PCR. Co-infection by EBV and HPV was reported in 5.6% cases (6/108). However, on comparing this data with the corresponding clinico-pathological cofactors (age, gender, grade of tumour, tumour size, nodal status, metastasis, DOI, TIL, ENE) it was found that there was no statistical significance between the two. Conclusion: Hence, we conclude that these oncogenic viruses are independent cofactors to oral cancer development and none of the clinico-morphologic variable is associated with the viral etiology.http://www.sciencedirect.com/science/article/pii/S2212426821000075Oncogenic virusesEBVHPVReal time PCROral cancer
spellingShingle Shahi Vanshika
Agarwal Preeti
Qayoom Sumaira
Kumar Vijay
Tewari Shikha
Raghuvanshi Shivanjali
Singh Uma Shankar
Goel Madhu Mati
Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 cases
Journal of Oral Biology and Craniofacial Research
Oncogenic viruses
EBV
HPV
Real time PCR
Oral cancer
title Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 cases
title_full Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 cases
title_fullStr Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 cases
title_full_unstemmed Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 cases
title_short Incidence OF HPV and EBV in oral cancer and their clinico-pathological correlation– a pilot study of 108 cases
title_sort incidence of hpv and ebv in oral cancer and their clinico pathological correlation a pilot study of 108 cases
topic Oncogenic viruses
EBV
HPV
Real time PCR
Oral cancer
url http://www.sciencedirect.com/science/article/pii/S2212426821000075
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