Review on Trypanosoma cruzi: Host Cell Interaction

Trypanosoma cruzi, the causative agent of Chagas' disease, which affects a large number of individuals in Central and South America, is transmitted to vertebrate hosts by blood-sucking insects. This protozoan is an obligate intracellular parasite. The infective forms of the parasite are metacyc...

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Main Authors: Wanderley de Souza, Tecia Maria Ulisses de Carvalho, Emile Santos Barrias
Format: Article
Language:English
Published: Wiley 2010-01-01
Series:International Journal of Cell Biology
Online Access:http://dx.doi.org/10.1155/2010/295394
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author Wanderley de Souza
Tecia Maria Ulisses de Carvalho
Emile Santos Barrias
author_facet Wanderley de Souza
Tecia Maria Ulisses de Carvalho
Emile Santos Barrias
author_sort Wanderley de Souza
collection DOAJ
description Trypanosoma cruzi, the causative agent of Chagas' disease, which affects a large number of individuals in Central and South America, is transmitted to vertebrate hosts by blood-sucking insects. This protozoan is an obligate intracellular parasite. The infective forms of the parasite are metacyclic and bloodstream trypomastigote and amastigote. Metacyclic trypomastigotes are released with the feces of the insect while amastigotes and bloodstream trypomastigotes are released from the infected host cells of the vertebrate host after a complex intracellular life cycle. The recognition between parasite and mammalian host cell involves numerous molecules present in both cell types. Here, we present a brief review of the interaction between Trypanosoma cruzi and its host cells, mainly emphasizing the mechanisms and molecules that participate in the T. cruzi invasion process of the mammalian cells.
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spelling doaj-art-b4f069a490e349eb9d96d984629181fd2025-08-20T02:23:31ZengWileyInternational Journal of Cell Biology1687-88761687-88842010-01-01201010.1155/2010/295394295394Review on Trypanosoma cruzi: Host Cell InteractionWanderley de Souza0Tecia Maria Ulisses de Carvalho1Emile Santos Barrias2Laboratório de Ultraestrutura Celular Hertha Meyer, CCS, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Bloco G, Ilha do Fundão, RJ 21941-902, BrazilLaboratório de Ultraestrutura Celular Hertha Meyer, CCS, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Bloco G, Ilha do Fundão, RJ 21941-902, BrazilLaboratório de Ultraestrutura Celular Hertha Meyer, CCS, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, Bloco G, Ilha do Fundão, RJ 21941-902, BrazilTrypanosoma cruzi, the causative agent of Chagas' disease, which affects a large number of individuals in Central and South America, is transmitted to vertebrate hosts by blood-sucking insects. This protozoan is an obligate intracellular parasite. The infective forms of the parasite are metacyclic and bloodstream trypomastigote and amastigote. Metacyclic trypomastigotes are released with the feces of the insect while amastigotes and bloodstream trypomastigotes are released from the infected host cells of the vertebrate host after a complex intracellular life cycle. The recognition between parasite and mammalian host cell involves numerous molecules present in both cell types. Here, we present a brief review of the interaction between Trypanosoma cruzi and its host cells, mainly emphasizing the mechanisms and molecules that participate in the T. cruzi invasion process of the mammalian cells.http://dx.doi.org/10.1155/2010/295394
spellingShingle Wanderley de Souza
Tecia Maria Ulisses de Carvalho
Emile Santos Barrias
Review on Trypanosoma cruzi: Host Cell Interaction
International Journal of Cell Biology
title Review on Trypanosoma cruzi: Host Cell Interaction
title_full Review on Trypanosoma cruzi: Host Cell Interaction
title_fullStr Review on Trypanosoma cruzi: Host Cell Interaction
title_full_unstemmed Review on Trypanosoma cruzi: Host Cell Interaction
title_short Review on Trypanosoma cruzi: Host Cell Interaction
title_sort review on trypanosoma cruzi host cell interaction
url http://dx.doi.org/10.1155/2010/295394
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