Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative Stress

Background. Inflammation and oxidative stress (OxS) contribute to the pathogenesis of diabetic kidney disease (DKD) and contrast-induced nephropathy (CIN). Patients with DKD were found to be more prone to CIN. Interleukin-33 (IL-33) is a proinflammatory cytokine, but its role in DKD and CIN is unkno...

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Main Authors: Didem Onk, Oruc Alper Onk, Kultigin Turkmen, Huseyin Serkan Erol, Tulin Akarsu Ayazoglu, Osman Nuri Keles, Mesut Halici, Ergun Topal
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2016/9050828
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author Didem Onk
Oruc Alper Onk
Kultigin Turkmen
Huseyin Serkan Erol
Tulin Akarsu Ayazoglu
Osman Nuri Keles
Mesut Halici
Ergun Topal
author_facet Didem Onk
Oruc Alper Onk
Kultigin Turkmen
Huseyin Serkan Erol
Tulin Akarsu Ayazoglu
Osman Nuri Keles
Mesut Halici
Ergun Topal
author_sort Didem Onk
collection DOAJ
description Background. Inflammation and oxidative stress (OxS) contribute to the pathogenesis of diabetic kidney disease (DKD) and contrast-induced nephropathy (CIN). Patients with DKD were found to be more prone to CIN. Interleukin-33 (IL-33) is a proinflammatory cytokine, but its role in DKD and CIN is unknown. Methods. Thirty male Sprague-Dawley rats were enrolled. The first group was comprised of healthy rats (HRs), whereas the other four groups were made up of diabetic rats (DRs), diabetic rats with contrast-induced nephropathy (CIN + DRs), melatonin-treated diabetic rats (MTDRs), and melatonin-treated CIN + DRs (MTCIN + DRs). All groups except the HRs received 50 mg/kg/day streptozotocin (STZ). CIN + DRs were constituted by administrating 1.5 mg/kg of intravenous radiocontrast dye on the 35th day. MTDRs and MTCIN + DRs were given 20 mg/kg/day of intraperitoneal injection of melatonin (MT) from the 28th day for the constitutive seven days. Results. We observed increased IL-33 in the kidney tissue following induction of CIN in DRs. To determine whether MT is effective in preventing CIN, we administered MT in CIN + DRs and demonstrated that kidney tissue levels of OxS markers, inflammatory cytokines, and IL-33 were significantly diminished in MTCIN + DRs compared with other groups without MT treatment (p<0.05). Conclusion. Inhibition of IL-33 with MT provides therapeutic potential in DKD with CIN.
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spelling doaj-art-b4e0d3d34f8d43b8a70f749dbef9d5d62025-08-20T02:23:31ZengWileyMediators of Inflammation0962-93511466-18612016-01-01201610.1155/2016/90508289050828Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative StressDidem Onk0Oruc Alper Onk1Kultigin Turkmen2Huseyin Serkan Erol3Tulin Akarsu Ayazoglu4Osman Nuri Keles5Mesut Halici6Ergun Topal7Department of Anesthesiology and Reanimation, Faculty of Medicine, Erzincan University, Erzincan, TurkeyDepartment of Cardiovascular Surgery, Faculty of Medicine, Erzincan University, Erzincan, TurkeyDivision of Nephrology, Department of Internal Medicine, Meram School of Medicine, Necmettin Erbakan University, Meram Tıp Fakültesi No. 215, Meram, 42090 Konya, TurkeyDepartment of Biochemistry, Faculty of Veterinary, Ataturk University, Erzurum, TurkeyDepartment of Anesthesiology and Reanimation, Medeniyet University, Goztepe Training and Research Hospital, Istanbul, TurkeyDepartment of Histology and Embryology, Faculty of Medicine, Ataturk University, Erzurum, TurkeyDepartment of Biochemistry, Faculty of Veterinary, Ataturk University, Erzurum, TurkeyDepartment of Cardiology, Faculty of Medicine, Erzincan University, Erzincan, TurkeyBackground. Inflammation and oxidative stress (OxS) contribute to the pathogenesis of diabetic kidney disease (DKD) and contrast-induced nephropathy (CIN). Patients with DKD were found to be more prone to CIN. Interleukin-33 (IL-33) is a proinflammatory cytokine, but its role in DKD and CIN is unknown. Methods. Thirty male Sprague-Dawley rats were enrolled. The first group was comprised of healthy rats (HRs), whereas the other four groups were made up of diabetic rats (DRs), diabetic rats with contrast-induced nephropathy (CIN + DRs), melatonin-treated diabetic rats (MTDRs), and melatonin-treated CIN + DRs (MTCIN + DRs). All groups except the HRs received 50 mg/kg/day streptozotocin (STZ). CIN + DRs were constituted by administrating 1.5 mg/kg of intravenous radiocontrast dye on the 35th day. MTDRs and MTCIN + DRs were given 20 mg/kg/day of intraperitoneal injection of melatonin (MT) from the 28th day for the constitutive seven days. Results. We observed increased IL-33 in the kidney tissue following induction of CIN in DRs. To determine whether MT is effective in preventing CIN, we administered MT in CIN + DRs and demonstrated that kidney tissue levels of OxS markers, inflammatory cytokines, and IL-33 were significantly diminished in MTCIN + DRs compared with other groups without MT treatment (p<0.05). Conclusion. Inhibition of IL-33 with MT provides therapeutic potential in DKD with CIN.http://dx.doi.org/10.1155/2016/9050828
spellingShingle Didem Onk
Oruc Alper Onk
Kultigin Turkmen
Huseyin Serkan Erol
Tulin Akarsu Ayazoglu
Osman Nuri Keles
Mesut Halici
Ergun Topal
Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative Stress
Mediators of Inflammation
title Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative Stress
title_full Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative Stress
title_fullStr Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative Stress
title_full_unstemmed Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative Stress
title_short Melatonin Attenuates Contrast-Induced Nephropathy in Diabetic Rats: The Role of Interleukin-33 and Oxidative Stress
title_sort melatonin attenuates contrast induced nephropathy in diabetic rats the role of interleukin 33 and oxidative stress
url http://dx.doi.org/10.1155/2016/9050828
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