Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in rats

Background and purpose: The Klotho (Klo) gene, an aging suppressor in rats, accelerates aging when disrupted and extends lifespan when overexpressed. It encodes a transmembrane protein primarily expressed in renal tubules. This study investigated the protective effects of central Klo, both alone and...

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Main Authors: Fatemeh Ahmadi, Elahe Amohashemi, Mohammad Kazemi, Hossein Salehi, Parham Reisi
Format: Article
Language:English
Published: Wolters Kluwer Medknow Publications 2025-05-01
Series:Research in Pharmaceutical Sciences
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Online Access:https://journals.lww.com/10.4103/RPS.RPS_46_24
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author Fatemeh Ahmadi
Elahe Amohashemi
Mohammad Kazemi
Hossein Salehi
Parham Reisi
author_facet Fatemeh Ahmadi
Elahe Amohashemi
Mohammad Kazemi
Hossein Salehi
Parham Reisi
author_sort Fatemeh Ahmadi
collection DOAJ
description Background and purpose: The Klotho (Klo) gene, an aging suppressor in rats, accelerates aging when disrupted and extends lifespan when overexpressed. It encodes a transmembrane protein primarily expressed in renal tubules. This study investigated the protective effects of central Klo, both alone and in combination with cholinergic anti-inflammatory pathway (CAP) inhibition, against ischemia-reperfusion injury (IRI)- induced acute kidney injury. The current study evaluated the expression of inflammatory and anti-inflammatory genes (including Illb, Tnfa, Tgfb, Trem2, and Il10) in the kidney, alongside plasma levels of creatinine (Cr), blood urea nitrogen (BUN), and signs of acute tubular injury. Experimental approach: Klo was microinjected into the rostral ventrolateral medulla, and CAP inhibition was achieved through intraperitoneal administration of mecamylamine (Mec). Real-time RT-PCR and hematoxylin and eosin staining were used for gene expression analysis and histopathological examination, respectively. Findings/Results: The results showed elevated Cr and BUN levels, tubular injury, and increased inflammatory gene expression in IRI and IRI + Mec groups, as well as reduced Il10 in the IRI + Mec group. Klo exhibited protective effects. Elevated Tgfb expression was seen in IRI + Klo and IRI + Mec + Klo groups one week post-surgery. Conclusion and implications: These findings indicated Klo potential to extend lifespan and protect against age-related diseases, including kidney disease and inflammation, via neural modulation of peripheral immunity.
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spelling doaj-art-b3cd3a93b828472b81be91186ab5e1992025-08-20T03:31:27ZengWolters Kluwer Medknow PublicationsResearch in Pharmaceutical Sciences1735-53621735-94142025-05-0120334335510.4103/RPS.RPS_46_24Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in ratsFatemeh AhmadiElahe AmohashemiMohammad KazemiHossein SalehiParham ReisiBackground and purpose: The Klotho (Klo) gene, an aging suppressor in rats, accelerates aging when disrupted and extends lifespan when overexpressed. It encodes a transmembrane protein primarily expressed in renal tubules. This study investigated the protective effects of central Klo, both alone and in combination with cholinergic anti-inflammatory pathway (CAP) inhibition, against ischemia-reperfusion injury (IRI)- induced acute kidney injury. The current study evaluated the expression of inflammatory and anti-inflammatory genes (including Illb, Tnfa, Tgfb, Trem2, and Il10) in the kidney, alongside plasma levels of creatinine (Cr), blood urea nitrogen (BUN), and signs of acute tubular injury. Experimental approach: Klo was microinjected into the rostral ventrolateral medulla, and CAP inhibition was achieved through intraperitoneal administration of mecamylamine (Mec). Real-time RT-PCR and hematoxylin and eosin staining were used for gene expression analysis and histopathological examination, respectively. Findings/Results: The results showed elevated Cr and BUN levels, tubular injury, and increased inflammatory gene expression in IRI and IRI + Mec groups, as well as reduced Il10 in the IRI + Mec group. Klo exhibited protective effects. Elevated Tgfb expression was seen in IRI + Klo and IRI + Mec + Klo groups one week post-surgery. Conclusion and implications: These findings indicated Klo potential to extend lifespan and protect against age-related diseases, including kidney disease and inflammation, via neural modulation of peripheral immunity.https://journals.lww.com/10.4103/RPS.RPS_46_24acute kidney injurycholinergic anti-inflammatory pathwayklothomecamylaminerostral ventrolateral medulla
spellingShingle Fatemeh Ahmadi
Elahe Amohashemi
Mohammad Kazemi
Hossein Salehi
Parham Reisi
Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in rats
Research in Pharmaceutical Sciences
acute kidney injury
cholinergic anti-inflammatory pathway
klotho
mecamylamine
rostral ventrolateral medulla
title Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in rats
title_full Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in rats
title_fullStr Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in rats
title_full_unstemmed Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in rats
title_short Klotho microinjection into the RVLM attenuates acute kidney injury via interaction with the cholinergic anti-inflammatory pathway in rats
title_sort klotho microinjection into the rvlm attenuates acute kidney injury via interaction with the cholinergic anti inflammatory pathway in rats
topic acute kidney injury
cholinergic anti-inflammatory pathway
klotho
mecamylamine
rostral ventrolateral medulla
url https://journals.lww.com/10.4103/RPS.RPS_46_24
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