HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD Patients

STAT3 is highly expressed in aGVHD CD4+ T cells and plays a critical role in inducing or worsening aGVHD. In our preceding studies, DNA hypomethylation in STAT3 promoter was shown to cause high expression of STAT3 in aGVHD CD4+ T cells, and the process could be modulated by HMGB1, but the underlying...

Full description

Saved in:
Bibliographic Details
Main Authors: Xuejun Xu, Yan Chen, Enyi Liu, Bin Fu, Juan Hua, Xu Chen, Yajing Xu
Format: Article
Language:English
Published: Wiley 2020-01-01
Series:Journal of Immunology Research
Online Access:http://dx.doi.org/10.1155/2020/7165230
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849396405695676416
author Xuejun Xu
Yan Chen
Enyi Liu
Bin Fu
Juan Hua
Xu Chen
Yajing Xu
author_facet Xuejun Xu
Yan Chen
Enyi Liu
Bin Fu
Juan Hua
Xu Chen
Yajing Xu
author_sort Xuejun Xu
collection DOAJ
description STAT3 is highly expressed in aGVHD CD4+ T cells and plays a critical role in inducing or worsening aGVHD. In our preceding studies, DNA hypomethylation in STAT3 promoter was shown to cause high expression of STAT3 in aGVHD CD4+ T cells, and the process could be modulated by HMGB1, but the underlying mechanism remains unclear. TET2, AID, and TDG are indispensable in DNA demethylation; meanwhile, TET2 and AID also serve extremely important roles in immune response. So, we speculated these enzymes involved in the STAT3 promoter hypomethylation induced by HMGB1 in aGVHD CD4+ T cells. In this study, we found that the binding levels of TET2/AID/TDG to STAT3 promoter were remarkably increased in CD4+T cells from aGVHD patients and were significantly negatively correlated with the STAT3 promoter methylation level. Simultaneously, we revealed that HMGB1 could recruit TET2, AID, and TDG to form a complex in the STAT3 promoter region. Interference with the expression of TET2/AID/TDG inhibited the overexpression of STAT3 caused by HMGB1 downregulation of the STAT3 promoter DNA methylation. These data demonstrated a new molecular mechanism of how HMGB1 promoted the expression of STAT3 in CD4+ T cells from aGVHD patients.
format Article
id doaj-art-b2992bf76c444d9699daee5815d4b18d
institution Kabale University
issn 2314-8861
2314-7156
language English
publishDate 2020-01-01
publisher Wiley
record_format Article
series Journal of Immunology Research
spelling doaj-art-b2992bf76c444d9699daee5815d4b18d2025-08-20T03:39:21ZengWileyJournal of Immunology Research2314-88612314-71562020-01-01202010.1155/2020/71652307165230HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD PatientsXuejun Xu0Yan Chen1Enyi Liu2Bin Fu3Juan Hua4Xu Chen5Yajing Xu6Department of Hematology, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaDepartment of Hematology, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaDepartment of Hematology, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaDepartment of Hematology, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaDepartment of Hematology, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaDepartment of Hematology, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaDepartment of Hematology, Xiangya Hospital, Central South University, Changsha, Hunan, ChinaSTAT3 is highly expressed in aGVHD CD4+ T cells and plays a critical role in inducing or worsening aGVHD. In our preceding studies, DNA hypomethylation in STAT3 promoter was shown to cause high expression of STAT3 in aGVHD CD4+ T cells, and the process could be modulated by HMGB1, but the underlying mechanism remains unclear. TET2, AID, and TDG are indispensable in DNA demethylation; meanwhile, TET2 and AID also serve extremely important roles in immune response. So, we speculated these enzymes involved in the STAT3 promoter hypomethylation induced by HMGB1 in aGVHD CD4+ T cells. In this study, we found that the binding levels of TET2/AID/TDG to STAT3 promoter were remarkably increased in CD4+T cells from aGVHD patients and were significantly negatively correlated with the STAT3 promoter methylation level. Simultaneously, we revealed that HMGB1 could recruit TET2, AID, and TDG to form a complex in the STAT3 promoter region. Interference with the expression of TET2/AID/TDG inhibited the overexpression of STAT3 caused by HMGB1 downregulation of the STAT3 promoter DNA methylation. These data demonstrated a new molecular mechanism of how HMGB1 promoted the expression of STAT3 in CD4+ T cells from aGVHD patients.http://dx.doi.org/10.1155/2020/7165230
spellingShingle Xuejun Xu
Yan Chen
Enyi Liu
Bin Fu
Juan Hua
Xu Chen
Yajing Xu
HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD Patients
Journal of Immunology Research
title HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD Patients
title_full HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD Patients
title_fullStr HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD Patients
title_full_unstemmed HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD Patients
title_short HMGB1 Recruits TET2/AID/TDG to Induce DNA Demethylation in STAT3 Promoter in CD4+ T Cells from aGVHD Patients
title_sort hmgb1 recruits tet2 aid tdg to induce dna demethylation in stat3 promoter in cd4 t cells from agvhd patients
url http://dx.doi.org/10.1155/2020/7165230
work_keys_str_mv AT xuejunxu hmgb1recruitstet2aidtdgtoinducednademethylationinstat3promoterincd4tcellsfromagvhdpatients
AT yanchen hmgb1recruitstet2aidtdgtoinducednademethylationinstat3promoterincd4tcellsfromagvhdpatients
AT enyiliu hmgb1recruitstet2aidtdgtoinducednademethylationinstat3promoterincd4tcellsfromagvhdpatients
AT binfu hmgb1recruitstet2aidtdgtoinducednademethylationinstat3promoterincd4tcellsfromagvhdpatients
AT juanhua hmgb1recruitstet2aidtdgtoinducednademethylationinstat3promoterincd4tcellsfromagvhdpatients
AT xuchen hmgb1recruitstet2aidtdgtoinducednademethylationinstat3promoterincd4tcellsfromagvhdpatients
AT yajingxu hmgb1recruitstet2aidtdgtoinducednademethylationinstat3promoterincd4tcellsfromagvhdpatients