A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency Anemia

Background: The efficacy of intravenous iron preparations for chronic heart failure with iron deficiency has been reported, but the efficacy of oral iron preparations has not been demonstrated. In this study, we conducted a prospective clinical study using ferric citrate hydrate tablets in patients...

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Main Authors: Akira Sezai, Hisakuni Sekino, Makoto Taoka, Kazuaki Obata, Sakie Kanno, Masashi Tanaka
Format: Article
Language:English
Published: MDPI AG 2025-04-01
Series:Life
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Online Access:https://www.mdpi.com/2075-1729/15/4/598
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author Akira Sezai
Hisakuni Sekino
Makoto Taoka
Kazuaki Obata
Sakie Kanno
Masashi Tanaka
author_facet Akira Sezai
Hisakuni Sekino
Makoto Taoka
Kazuaki Obata
Sakie Kanno
Masashi Tanaka
author_sort Akira Sezai
collection DOAJ
description Background: The efficacy of intravenous iron preparations for chronic heart failure with iron deficiency has been reported, but the efficacy of oral iron preparations has not been demonstrated. In this study, we conducted a prospective clinical study using ferric citrate hydrate tablets in patients with chronic heart failure complicated by iron deficiency anemia. Methods and Results: A prospective study was conducted using ferric citrate hydrate in patients with chronic heart failure complicated by iron deficiency anemia. The registered patients were divided into two groups: those administered ferric citrate hydrate and those switched from iron sulfate sustained-release to ferric citrate hydrate. The primary endpoint was hemoglobin level. The secondary endpoints included hematocrit, serum iron, saturation, ferritin, and cardiac-, renal-, and hepatic-related biomarkers. A total of 141 patients were enrolled in this study, including 95 patients who were newly administered ferric citrate hydrate and 46 patients who were switched from iron sulfate sustained-release to ferric citrate hydrate. Conclusions: Ferric citrate hydrate significantly increased hemoglobin, serum iron, transferrin saturation (TSAT), and ferritin levels, and decreased atrial natriuretic peptide (ANP), brain natriuretic peptide (BNP), and N-terminal pro-brain natriuretic peptide (NT-proBNP) levels. Ferric citrate hydrate could be continued without side effects such as gastrointestinal symptoms. Improvement in iron metabolism and anemia due to iron supplementation with ferric citrate hydrate led to improvement in heart failure biomarkers.
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spelling doaj-art-b1f170a99f9641fca45d72d9e2ba0baf2025-08-20T02:18:14ZengMDPI AGLife2075-17292025-04-0115459810.3390/life15040598A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency AnemiaAkira Sezai0Hisakuni Sekino1Makoto Taoka2Kazuaki Obata3Sakie Kanno4Masashi Tanaka5Department of Cardiovascular Surgery, Nihon University School of Medicine, Tokyo 173-8610, JapanSekino Hospital, Tokyo 171-0014, JapanDepartment of Cardiovascular Surgery, Nihon University School of Medicine, Tokyo 173-8610, JapanSekino Hospital, Tokyo 171-0014, JapanSekino Hospital, Tokyo 171-0014, JapanDepartment of Cardiovascular Surgery, Nihon University School of Medicine, Tokyo 173-8610, JapanBackground: The efficacy of intravenous iron preparations for chronic heart failure with iron deficiency has been reported, but the efficacy of oral iron preparations has not been demonstrated. In this study, we conducted a prospective clinical study using ferric citrate hydrate tablets in patients with chronic heart failure complicated by iron deficiency anemia. Methods and Results: A prospective study was conducted using ferric citrate hydrate in patients with chronic heart failure complicated by iron deficiency anemia. The registered patients were divided into two groups: those administered ferric citrate hydrate and those switched from iron sulfate sustained-release to ferric citrate hydrate. The primary endpoint was hemoglobin level. The secondary endpoints included hematocrit, serum iron, saturation, ferritin, and cardiac-, renal-, and hepatic-related biomarkers. A total of 141 patients were enrolled in this study, including 95 patients who were newly administered ferric citrate hydrate and 46 patients who were switched from iron sulfate sustained-release to ferric citrate hydrate. Conclusions: Ferric citrate hydrate significantly increased hemoglobin, serum iron, transferrin saturation (TSAT), and ferritin levels, and decreased atrial natriuretic peptide (ANP), brain natriuretic peptide (BNP), and N-terminal pro-brain natriuretic peptide (NT-proBNP) levels. Ferric citrate hydrate could be continued without side effects such as gastrointestinal symptoms. Improvement in iron metabolism and anemia due to iron supplementation with ferric citrate hydrate led to improvement in heart failure biomarkers.https://www.mdpi.com/2075-1729/15/4/598iron-deficiency anemiaanemiaheart failureferric citrate hydratechronic heart failure
spellingShingle Akira Sezai
Hisakuni Sekino
Makoto Taoka
Kazuaki Obata
Sakie Kanno
Masashi Tanaka
A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency Anemia
Life
iron-deficiency anemia
anemia
heart failure
ferric citrate hydrate
chronic heart failure
title A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency Anemia
title_full A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency Anemia
title_fullStr A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency Anemia
title_full_unstemmed A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency Anemia
title_short A Prospective Clinical Study of Ferric Citrate Hydrate for Chronic Heart Failure with Iron Deficiency Anemia
title_sort prospective clinical study of ferric citrate hydrate for chronic heart failure with iron deficiency anemia
topic iron-deficiency anemia
anemia
heart failure
ferric citrate hydrate
chronic heart failure
url https://www.mdpi.com/2075-1729/15/4/598
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