Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In Vivo

Immune disorder is considered the main pathogenesis of autoimmune diseases, such as rheumatoid arthritis (RA). The balance of the two special subsets of CD4+T cells, T helper cell 17 (Th17), and Regulator T cell (Treg) is the key factor of maintaining a normal immune response. Dendritic cells (DCs),...

Full description

Saved in:
Bibliographic Details
Main Authors: Xiaoyang Yu, Caihong Wang, Jing Luo, Xiangcong Zhao, Lixing Wang, Xiaofeng Li
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:Clinical and Developmental Immunology
Online Access:http://dx.doi.org/10.1155/2013/238035
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850177083481784320
author Xiaoyang Yu
Caihong Wang
Jing Luo
Xiangcong Zhao
Lixing Wang
Xiaofeng Li
author_facet Xiaoyang Yu
Caihong Wang
Jing Luo
Xiangcong Zhao
Lixing Wang
Xiaofeng Li
author_sort Xiaoyang Yu
collection DOAJ
description Immune disorder is considered the main pathogenesis of autoimmune diseases, such as rheumatoid arthritis (RA). The balance of the two special subsets of CD4+T cells, T helper cell 17 (Th17), and Regulator T cell (Treg) is the key factor of maintaining a normal immune response. Dendritic cells (DCs), which are the most powerful antigen-presenting cells, play an important role in regulating the balance of Th17 and Treg. The combination of disease modifying antirheumatic drugs (DMARDs) is an important strategy of RA therapy. In this study, we investigated the effect of MTX and CTX on DC maturation in ovalbumin (OVA) immunized mice. Th17 inflammatory response is stronger, while the level of DCs maturity is higher. In contrast, the immunosuppression of Treg is stronger. We found that MTX combined with CTX significantly inhibited the DCs maturity and downregulated the antigen presenting capacity of DCs. As a result, it reestablished a balance of Th17 and Treg. Our study adds a novel mechanism and therapeutic target of MTX combined with CTX for autoimmune disease treatment.
format Article
id doaj-art-af3a4fdf09af4bb880b1e89a61d2da88
institution OA Journals
issn 1740-2522
1740-2530
language English
publishDate 2013-01-01
publisher Wiley
record_format Article
series Clinical and Developmental Immunology
spelling doaj-art-af3a4fdf09af4bb880b1e89a61d2da882025-08-20T02:19:06ZengWileyClinical and Developmental Immunology1740-25221740-25302013-01-01201310.1155/2013/238035238035Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In VivoXiaoyang Yu0Caihong Wang1Jing Luo2Xiangcong Zhao3Lixing Wang4Xiaofeng Li5Department of Rheumatology, The Second Hospital of Shanxi Medical University, 382 Wu Yi Road, Taiyuan 030001, ChinaDepartment of Rheumatology, The Second Hospital of Shanxi Medical University, 382 Wu Yi Road, Taiyuan 030001, ChinaDepartment of Rheumatology, The Second Hospital of Shanxi Medical University, 382 Wu Yi Road, Taiyuan 030001, ChinaDepartment of Rheumatology, The Second Hospital of Shanxi Medical University, 382 Wu Yi Road, Taiyuan 030001, ChinaDepartment of Rheumatology, The Second Hospital of Shanxi Medical University, 382 Wu Yi Road, Taiyuan 030001, ChinaDepartment of Rheumatology, The Second Hospital of Shanxi Medical University, 382 Wu Yi Road, Taiyuan 030001, ChinaImmune disorder is considered the main pathogenesis of autoimmune diseases, such as rheumatoid arthritis (RA). The balance of the two special subsets of CD4+T cells, T helper cell 17 (Th17), and Regulator T cell (Treg) is the key factor of maintaining a normal immune response. Dendritic cells (DCs), which are the most powerful antigen-presenting cells, play an important role in regulating the balance of Th17 and Treg. The combination of disease modifying antirheumatic drugs (DMARDs) is an important strategy of RA therapy. In this study, we investigated the effect of MTX and CTX on DC maturation in ovalbumin (OVA) immunized mice. Th17 inflammatory response is stronger, while the level of DCs maturity is higher. In contrast, the immunosuppression of Treg is stronger. We found that MTX combined with CTX significantly inhibited the DCs maturity and downregulated the antigen presenting capacity of DCs. As a result, it reestablished a balance of Th17 and Treg. Our study adds a novel mechanism and therapeutic target of MTX combined with CTX for autoimmune disease treatment.http://dx.doi.org/10.1155/2013/238035
spellingShingle Xiaoyang Yu
Caihong Wang
Jing Luo
Xiangcong Zhao
Lixing Wang
Xiaofeng Li
Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In Vivo
Clinical and Developmental Immunology
title Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In Vivo
title_full Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In Vivo
title_fullStr Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In Vivo
title_full_unstemmed Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In Vivo
title_short Combination with Methotrexate and Cyclophosphamide Attenuated Maturation of Dendritic Cells: Inducing Treg Skewing and Th17 Suppression In Vivo
title_sort combination with methotrexate and cyclophosphamide attenuated maturation of dendritic cells inducing treg skewing and th17 suppression in vivo
url http://dx.doi.org/10.1155/2013/238035
work_keys_str_mv AT xiaoyangyu combinationwithmethotrexateandcyclophosphamideattenuatedmaturationofdendriticcellsinducingtregskewingandth17suppressioninvivo
AT caihongwang combinationwithmethotrexateandcyclophosphamideattenuatedmaturationofdendriticcellsinducingtregskewingandth17suppressioninvivo
AT jingluo combinationwithmethotrexateandcyclophosphamideattenuatedmaturationofdendriticcellsinducingtregskewingandth17suppressioninvivo
AT xiangcongzhao combinationwithmethotrexateandcyclophosphamideattenuatedmaturationofdendriticcellsinducingtregskewingandth17suppressioninvivo
AT lixingwang combinationwithmethotrexateandcyclophosphamideattenuatedmaturationofdendriticcellsinducingtregskewingandth17suppressioninvivo
AT xiaofengli combinationwithmethotrexateandcyclophosphamideattenuatedmaturationofdendriticcellsinducingtregskewingandth17suppressioninvivo