Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of Losartan

<b>Background/Objectives:</b> Hypertension is a pathological condition characterized by elevated systolic and/or diastolic blood pressure. A range of pharmacotherapeutic agents are available to treat this condition and prevent complications, including the angiotensin II AT1-receptor bloc...

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Main Authors: Dmitry Babaev, Elena Kutumova, Fedor Kolpakov
Format: Article
Language:English
Published: MDPI AG 2025-07-01
Series:Pharmaceutics
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Online Access:https://www.mdpi.com/1999-4923/17/7/935
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author Dmitry Babaev
Elena Kutumova
Fedor Kolpakov
author_facet Dmitry Babaev
Elena Kutumova
Fedor Kolpakov
author_sort Dmitry Babaev
collection DOAJ
description <b>Background/Objectives:</b> Hypertension is a pathological condition characterized by elevated systolic and/or diastolic blood pressure. A range of pharmacotherapeutic agents are available to treat this condition and prevent complications, including the angiotensin II AT1-receptor blocker losartan. Following oral administration, losartan is exposed to a variety of enzymes that facilitate its metabolism or transportation. The structural characteristics of the genes that encode the enzymes may potentially impact the pharmacokinetics and pharmacodynamics of losartan, thereby modulating its effects on the treatment process. <b>Methods</b>: In this study, a computational model of losartan pharmacokinetics was developed, taking into account the influence of different alleles of the <i>CYP2C9</i> gene, which plays a pivotal role in losartan metabolism, and the <i>ABCB1</i> gene, which is responsible for losartan transport. <b>Results</b>: Alterations in the modeled activities of the enzymes encoded by <i>CYP2C9</i> and <i>ABCB1</i> result in changes in the losartan and its metabolite profiles that are consistent with known experimental data in real patients with different <i>CYP2C9</i> and <i>ABCB1</i> genotypes. <b>Conclusions</b>: The findings of the modeling can potentially be used to personalize drug therapy for arterial hypertension.
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spelling doaj-art-add4088ae5584a60892c911a1f2cb9bc2025-08-20T03:56:46ZengMDPI AGPharmaceutics1999-49232025-07-0117793510.3390/pharmaceutics17070935Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of LosartanDmitry Babaev0Elena Kutumova1Fedor Kolpakov2Department of Computational Biology, Sirius University of Science and Technology, 354340 Sirius, RussiaDepartment of Computational Biology, Sirius University of Science and Technology, 354340 Sirius, RussiaDepartment of Computational Biology, Sirius University of Science and Technology, 354340 Sirius, Russia<b>Background/Objectives:</b> Hypertension is a pathological condition characterized by elevated systolic and/or diastolic blood pressure. A range of pharmacotherapeutic agents are available to treat this condition and prevent complications, including the angiotensin II AT1-receptor blocker losartan. Following oral administration, losartan is exposed to a variety of enzymes that facilitate its metabolism or transportation. The structural characteristics of the genes that encode the enzymes may potentially impact the pharmacokinetics and pharmacodynamics of losartan, thereby modulating its effects on the treatment process. <b>Methods</b>: In this study, a computational model of losartan pharmacokinetics was developed, taking into account the influence of different alleles of the <i>CYP2C9</i> gene, which plays a pivotal role in losartan metabolism, and the <i>ABCB1</i> gene, which is responsible for losartan transport. <b>Results</b>: Alterations in the modeled activities of the enzymes encoded by <i>CYP2C9</i> and <i>ABCB1</i> result in changes in the losartan and its metabolite profiles that are consistent with known experimental data in real patients with different <i>CYP2C9</i> and <i>ABCB1</i> genotypes. <b>Conclusions</b>: The findings of the modeling can potentially be used to personalize drug therapy for arterial hypertension.https://www.mdpi.com/1999-4923/17/7/935arterial hypertensionlosartanpharmacokineticspharmacodynamics<i>ABCB1</i><i>CYP2C9</i>
spellingShingle Dmitry Babaev
Elena Kutumova
Fedor Kolpakov
Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of Losartan
Pharmaceutics
arterial hypertension
losartan
pharmacokinetics
pharmacodynamics
<i>ABCB1</i>
<i>CYP2C9</i>
title Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of Losartan
title_full Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of Losartan
title_fullStr Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of Losartan
title_full_unstemmed Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of Losartan
title_short Modeling the Influence of <i>CYP2C9</i> and <i>ABCB1</i> Gene Polymorphisms on the Pharmacokinetics and Pharmacodynamics of Losartan
title_sort modeling the influence of i cyp2c9 i and i abcb1 i gene polymorphisms on the pharmacokinetics and pharmacodynamics of losartan
topic arterial hypertension
losartan
pharmacokinetics
pharmacodynamics
<i>ABCB1</i>
<i>CYP2C9</i>
url https://www.mdpi.com/1999-4923/17/7/935
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AT fedorkolpakov modelingtheinfluenceoficyp2c9iandiabcb1igenepolymorphismsonthepharmacokineticsandpharmacodynamicsoflosartan