Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZ

Background and purpose F. nucleatum, a gram-negative oral bacteria, is abundant in laryngeal cancer (LC). While specific 14-3-3 proteins act as LC oncogenes, the link between F. nucleatum and 14-3-3 proteins dysregulation remains unknown.Materials and methods Transcriptome data from 520 HNSC patient...

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Main Authors: Yiyue Ren, Jing Ye, Liuhong Shi, Kehao Le, Yifan Wang
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Journal of Oral Microbiology
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Online Access:https://www.tandfonline.com/doi/10.1080/20002297.2024.2442245
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author Yiyue Ren
Jing Ye
Liuhong Shi
Kehao Le
Yifan Wang
author_facet Yiyue Ren
Jing Ye
Liuhong Shi
Kehao Le
Yifan Wang
author_sort Yiyue Ren
collection DOAJ
description Background and purpose F. nucleatum, a gram-negative oral bacteria, is abundant in laryngeal cancer (LC). While specific 14-3-3 proteins act as LC oncogenes, the link between F. nucleatum and 14-3-3 proteins dysregulation remains unknown.Materials and methods Transcriptome data from 520 HNSC patients were obtained from TCGA. DEGs were evaluated by Wilcoxon test, and survival analysis done by KM plotter. In 41 untreated LC patients, F. nucleatum was detected by PCR. GEO data assessed 14-3-3 protein changes post F. nucleatum infection, verified in HuLa-PC and LC cells (Tu686, LCC). YWHAZ oncogenic role was tested in vitro and in xenografts. YWHAZ knockdown confirmed F. nucleatum’s oncogenic effects are YWHAZ-dependent.Results F. nucleatum positive tumors have increased YWAHZ. F. nucleatum infection upregulates the expression of YWHAZ in HuLa-PC, Tu686 and Hep-2 cells. YWAHZ overexpression promotes proliferation, migration and colony formation but inhibits apoptosis of HuLa-PC and LCa cells via activating Akt signaling. F. nucleatum infection promotes the tumor growth. Knockdown YWAHZ in LCa cells totally rescued the phenotypes of F. nucleatum infection.Conclusions We identified for the first time that F. nucleatum infection promotes the development of LCa via upregulating YWHAZ, which provides a new clue for LCa treatment.
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spelling doaj-art-adabe16d623a42adaae40a5f767c05162025-08-20T02:35:36ZengTaylor & Francis GroupJournal of Oral Microbiology2000-22972025-12-0117110.1080/20002297.2024.2442245Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZYiyue Ren0Jing Ye1Liuhong Shi2Kehao Le3Yifan Wang4Department of Head and Neck Surgery, Sir Run Run Shaw Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, ChinaDepartment of Otolaryngology Head and Neck Surgery, Sir Run Run Shaw Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, ChinaDepartment of Head and Neck Surgery, Sir Run Run Shaw Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, ChinaDepartment of Head and Neck Surgery, Sir Run Run Shaw Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang, ChinaSchool of Medicine, Zhejiang University, Hangzhou, Zhejiang, ChinaBackground and purpose F. nucleatum, a gram-negative oral bacteria, is abundant in laryngeal cancer (LC). While specific 14-3-3 proteins act as LC oncogenes, the link between F. nucleatum and 14-3-3 proteins dysregulation remains unknown.Materials and methods Transcriptome data from 520 HNSC patients were obtained from TCGA. DEGs were evaluated by Wilcoxon test, and survival analysis done by KM plotter. In 41 untreated LC patients, F. nucleatum was detected by PCR. GEO data assessed 14-3-3 protein changes post F. nucleatum infection, verified in HuLa-PC and LC cells (Tu686, LCC). YWHAZ oncogenic role was tested in vitro and in xenografts. YWHAZ knockdown confirmed F. nucleatum’s oncogenic effects are YWHAZ-dependent.Results F. nucleatum positive tumors have increased YWAHZ. F. nucleatum infection upregulates the expression of YWHAZ in HuLa-PC, Tu686 and Hep-2 cells. YWAHZ overexpression promotes proliferation, migration and colony formation but inhibits apoptosis of HuLa-PC and LCa cells via activating Akt signaling. F. nucleatum infection promotes the tumor growth. Knockdown YWAHZ in LCa cells totally rescued the phenotypes of F. nucleatum infection.Conclusions We identified for the first time that F. nucleatum infection promotes the development of LCa via upregulating YWHAZ, which provides a new clue for LCa treatment.https://www.tandfonline.com/doi/10.1080/20002297.2024.2442245Laryngeal cancerFusobacterium nucleatumYWAHZHNSCAkt
spellingShingle Yiyue Ren
Jing Ye
Liuhong Shi
Kehao Le
Yifan Wang
Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZ
Journal of Oral Microbiology
Laryngeal cancer
Fusobacterium nucleatum
YWAHZ
HNSC
Akt
title Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZ
title_full Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZ
title_fullStr Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZ
title_full_unstemmed Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZ
title_short Fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating YWHAZ
title_sort fusobacterium nucleatum as an oral bacteria promotes the carcinogenesis of laryngeal cancer via upregulating ywhaz
topic Laryngeal cancer
Fusobacterium nucleatum
YWAHZ
HNSC
Akt
url https://www.tandfonline.com/doi/10.1080/20002297.2024.2442245
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