Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression

Nicotinic acetylcholine receptors (nAChRs) are widely expressed in a variety of cell types and are involved in multiple physiological regulatory mechanisms in cells, tissues and systems. Increasing evidence suggests that the α5 nicotinic acetylcholine receptor (α5-nAChR), encoded by the CHRNA5 gene,...

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Main Authors: Jiaying Cai, Jingting Wang, Zengping Wang, Jing Wang, Yanfei Jia, Xiaoli Ma
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-03-01
Series:Frontiers in Cell and Developmental Biology
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Online Access:https://www.frontiersin.org/articles/10.3389/fcell.2025.1489958/full
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author Jiaying Cai
Jingting Wang
Zengping Wang
Jing Wang
Yanfei Jia
Xiaoli Ma
author_facet Jiaying Cai
Jingting Wang
Zengping Wang
Jing Wang
Yanfei Jia
Xiaoli Ma
author_sort Jiaying Cai
collection DOAJ
description Nicotinic acetylcholine receptors (nAChRs) are widely expressed in a variety of cell types and are involved in multiple physiological regulatory mechanisms in cells, tissues and systems. Increasing evidence suggests that the α5 nicotinic acetylcholine receptor (α5-nAChR), encoded by the CHRNA5 gene, is one of a key mediator involved in lung cancer development and immune responses. Several studies have shown that it is a regulator that stimulates processes via various signaling pathways, including STAT3 in lung cancer. In addition, α5-nAChR has a profound effect on lung immune response through multiple immune-related factor pathways. In this review, we focus on the perspectives on α5-nAChR in lung cancer progression, which indicates that targeting α5-nAChR could provide novel anticancer and immune therapy strategies for lung cancer.
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publisher Frontiers Media S.A.
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series Frontiers in Cell and Developmental Biology
spelling doaj-art-ad00a2d46f134264b5e227201006ad712025-08-20T02:04:28ZengFrontiers Media S.A.Frontiers in Cell and Developmental Biology2296-634X2025-03-011310.3389/fcell.2025.14899581489958Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progressionJiaying CaiJingting WangZengping WangJing WangYanfei JiaXiaoli MaNicotinic acetylcholine receptors (nAChRs) are widely expressed in a variety of cell types and are involved in multiple physiological regulatory mechanisms in cells, tissues and systems. Increasing evidence suggests that the α5 nicotinic acetylcholine receptor (α5-nAChR), encoded by the CHRNA5 gene, is one of a key mediator involved in lung cancer development and immune responses. Several studies have shown that it is a regulator that stimulates processes via various signaling pathways, including STAT3 in lung cancer. In addition, α5-nAChR has a profound effect on lung immune response through multiple immune-related factor pathways. In this review, we focus on the perspectives on α5-nAChR in lung cancer progression, which indicates that targeting α5-nAChR could provide novel anticancer and immune therapy strategies for lung cancer.https://www.frontiersin.org/articles/10.3389/fcell.2025.1489958/fullα5-nAChRgrowthepithelial-mesenchymal transition (EMT)immune escapelung cancer
spellingShingle Jiaying Cai
Jingting Wang
Zengping Wang
Jing Wang
Yanfei Jia
Xiaoli Ma
Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression
Frontiers in Cell and Developmental Biology
α5-nAChR
growth
epithelial-mesenchymal transition (EMT)
immune escape
lung cancer
title Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression
title_full Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression
title_fullStr Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression
title_full_unstemmed Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression
title_short Perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression
title_sort perspectives on the α5 nicotinic acetylcholine receptor in lung cancer progression
topic α5-nAChR
growth
epithelial-mesenchymal transition (EMT)
immune escape
lung cancer
url https://www.frontiersin.org/articles/10.3389/fcell.2025.1489958/full
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