Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cells

Background. Treatment of glioblastoma multiforme remains little effective due to the rapidly developing recurrence of the tumor, due to its high tumorigenic potential, resistance to chemoradiation therapy and increased dissemination of glioma stem cells (GSC). Molecular mechanisms of these cell inte...

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Main Authors: V. E. Shevchenko, I. S. Bryukhovetskiy, E. A. Savchenko, N. E. Arnotskaya
Format: Article
Language:Russian
Published: ABV-press 2019-04-01
Series:Успехи молекулярной онкологии
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Online Access:https://umo.abvpress.ru/jour/article/view/208
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author V. E. Shevchenko
I. S. Bryukhovetskiy
E. A. Savchenko
N. E. Arnotskaya
author_facet V. E. Shevchenko
I. S. Bryukhovetskiy
E. A. Savchenko
N. E. Arnotskaya
author_sort V. E. Shevchenko
collection DOAJ
description Background. Treatment of glioblastoma multiforme remains little effective due to the rapidly developing recurrence of the tumor, due to its high tumorigenic potential, resistance to chemoradiation therapy and increased dissemination of glioma stem cells (GSC). Molecular mechanisms of these cell interaction with extracellular matrix (ECM) are practically not studied. At present, it is also not clear the signaling of the ECM-receptor interaction (ECM-RI) differs for GSC and differentiated glioma cells (GDC).Objective: using high-resolution proteomic mass spectrometry to study the determinant expression of the ECM-receptor interaction signaling cascade in CD133+ GSC and CD133–    GDC.Results. 1990 proteins are identified, 18 of which are associated with the ECM-RI process. Positive regulation of 14 ECM-RI proteins was found in CD133+ GSC compared with CD133–    GDC, ten had more than 2 times increased expression. Increase in the CD133+ GSC level of 4 proteins activating the ECM-RI signaling cascade was noted.Conclusion. Important regularities are determined that could be used for the development of new approaches for detection of potential therapy targets of glioblastoma multiforme.
format Article
id doaj-art-acec70657b044d9a9b8f28cdb6ef069b
institution Kabale University
issn 2313-805X
2413-3787
language Russian
publishDate 2019-04-01
publisher ABV-press
record_format Article
series Успехи молекулярной онкологии
spelling doaj-art-acec70657b044d9a9b8f28cdb6ef069b2025-08-20T03:37:43ZrusABV-pressУспехи молекулярной онкологии2313-805X2413-37872019-04-0161637210.17650/2313-805X-2019-6-1-63-72157Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cellsV. E. Shevchenko0I. S. Bryukhovetskiy1E. A. Savchenko2N. E. Arnotskaya3Research Institute of Carcinogenesis, N.N. Blokhin National Medical Research Center of Oncology, Ministry of Health of RussiaSchool of Biomedicine, Far Eastern Federal University; National Scientific Center of Marine Biology, Far Eastern Branch of the Russian Academy of SciencesLimited Liability Company “APTO-FARM”Research Institute of Carcinogenesis, N.N. Blokhin National Medical Research Center of Oncology, Ministry of Health of RussiaBackground. Treatment of glioblastoma multiforme remains little effective due to the rapidly developing recurrence of the tumor, due to its high tumorigenic potential, resistance to chemoradiation therapy and increased dissemination of glioma stem cells (GSC). Molecular mechanisms of these cell interaction with extracellular matrix (ECM) are practically not studied. At present, it is also not clear the signaling of the ECM-receptor interaction (ECM-RI) differs for GSC and differentiated glioma cells (GDC).Objective: using high-resolution proteomic mass spectrometry to study the determinant expression of the ECM-receptor interaction signaling cascade in CD133+ GSC and CD133–    GDC.Results. 1990 proteins are identified, 18 of which are associated with the ECM-RI process. Positive regulation of 14 ECM-RI proteins was found in CD133+ GSC compared with CD133–    GDC, ten had more than 2 times increased expression. Increase in the CD133+ GSC level of 4 proteins activating the ECM-RI signaling cascade was noted.Conclusion. Important regularities are determined that could be used for the development of new approaches for detection of potential therapy targets of glioblastoma multiforme.https://umo.abvpress.ru/jour/article/view/208extracellular matrixglioma stem cellsglioblastoma multiformeproteomemass-spectrometry
spellingShingle V. E. Shevchenko
I. S. Bryukhovetskiy
E. A. Savchenko
N. E. Arnotskaya
Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cells
Успехи молекулярной онкологии
extracellular matrix
glioma stem cells
glioblastoma multiforme
proteome
mass-spectrometry
title Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cells
title_full Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cells
title_fullStr Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cells
title_full_unstemmed Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cells
title_short Study of interaction between extracellular matrix proteins and receptors of CD133+ stem cells and CD133– differentiated glioma cells
title_sort study of interaction between extracellular matrix proteins and receptors of cd133 stem cells and cd133 differentiated glioma cells
topic extracellular matrix
glioma stem cells
glioblastoma multiforme
proteome
mass-spectrometry
url https://umo.abvpress.ru/jour/article/view/208
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AT isbryukhovetskiy studyofinteractionbetweenextracellularmatrixproteinsandreceptorsofcd133stemcellsandcd133differentiatedgliomacells
AT easavchenko studyofinteractionbetweenextracellularmatrixproteinsandreceptorsofcd133stemcellsandcd133differentiatedgliomacells
AT nearnotskaya studyofinteractionbetweenextracellularmatrixproteinsandreceptorsofcd133stemcellsandcd133differentiatedgliomacells