Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or Aphasia

The clinical significance and characteristics of writing errors in bulbar-onset amyotrophic lateral sclerosis (ALS) are not clear. We retrospectively investigated writing samples in 19 patients with bulbar-onset ALS without preceding extra-motor symptoms. Co-development of dementia and/or aphasia wa...

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Main Authors: Hiroo Ichikawa, Nobuyoshi Takahashi, Soutaro Hieda, Hideki Ohno, Mitsuru Kawamura
Format: Article
Language:English
Published: Wiley 2008-01-01
Series:Behavioural Neurology
Online Access:http://dx.doi.org/10.3233/BEN-2008-0219
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author Hiroo Ichikawa
Nobuyoshi Takahashi
Soutaro Hieda
Hideki Ohno
Mitsuru Kawamura
author_facet Hiroo Ichikawa
Nobuyoshi Takahashi
Soutaro Hieda
Hideki Ohno
Mitsuru Kawamura
author_sort Hiroo Ichikawa
collection DOAJ
description The clinical significance and characteristics of writing errors in bulbar-onset amyotrophic lateral sclerosis (ALS) are not clear. We retrospectively investigated writing samples in 19 patients with bulbar-onset ALS without preceding extra-motor symptoms. Co-development of dementia and/or aphasia was also explored and single photon emission computed tomography (SPECT) images of the brain were reviewed. As a result, a high prevalence of writing errors (15 of the 19 patients) was found. Of note were isolated writing errors with neither dementia nor aphasia verified in 2 patients whose dysarthria was mild enough to evaluate spoken language. The remaining 13 patients also showed agraphia, but either dysarthria was too severe to evaluate aphasia or frontotemporal dementia (FTD)-like features co-existed. Of these patients, one who initially lacked dementia subsequently developed FTD-like features. The frequent writing errors were omission or substitution of kana letters and syntactic errors. SPECT images showed bilateral or left-side dominant hypoperfusion in the frontotemporal lobes as a consistent feature. These results show that patients with bulbar-onset ALS frequently exhibit agraphic writing errors and that these are not merely consequences of dementia or aphasia. However, these writing errors may indicate the involvement of frontotemporal language-related areas beyond the primary motor cortex.
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spelling doaj-art-ac3a1d16ce854dbb92f78aa2de0212722025-08-20T03:39:14ZengWileyBehavioural Neurology0953-41801875-85842008-01-01203-4919910.3233/BEN-2008-0219Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or AphasiaHiroo Ichikawa0Nobuyoshi Takahashi1Soutaro Hieda2Hideki Ohno3Mitsuru Kawamura4Department of Neurology, Showa University School of Medicine, Shinagawa-ku, Tokyo, JapanDepartment of Neurology, Showa University School of Medicine, Shinagawa-ku, Tokyo, JapanDepartment of Neurology, Showa University School of Medicine, Shinagawa-ku, Tokyo, JapanDepartment of Neurology, Showa University School of Medicine, Shinagawa-ku, Tokyo, JapanDepartment of Neurology, Showa University School of Medicine, Shinagawa-ku, Tokyo, JapanThe clinical significance and characteristics of writing errors in bulbar-onset amyotrophic lateral sclerosis (ALS) are not clear. We retrospectively investigated writing samples in 19 patients with bulbar-onset ALS without preceding extra-motor symptoms. Co-development of dementia and/or aphasia was also explored and single photon emission computed tomography (SPECT) images of the brain were reviewed. As a result, a high prevalence of writing errors (15 of the 19 patients) was found. Of note were isolated writing errors with neither dementia nor aphasia verified in 2 patients whose dysarthria was mild enough to evaluate spoken language. The remaining 13 patients also showed agraphia, but either dysarthria was too severe to evaluate aphasia or frontotemporal dementia (FTD)-like features co-existed. Of these patients, one who initially lacked dementia subsequently developed FTD-like features. The frequent writing errors were omission or substitution of kana letters and syntactic errors. SPECT images showed bilateral or left-side dominant hypoperfusion in the frontotemporal lobes as a consistent feature. These results show that patients with bulbar-onset ALS frequently exhibit agraphic writing errors and that these are not merely consequences of dementia or aphasia. However, these writing errors may indicate the involvement of frontotemporal language-related areas beyond the primary motor cortex.http://dx.doi.org/10.3233/BEN-2008-0219
spellingShingle Hiroo Ichikawa
Nobuyoshi Takahashi
Soutaro Hieda
Hideki Ohno
Mitsuru Kawamura
Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or Aphasia
Behavioural Neurology
title Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or Aphasia
title_full Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or Aphasia
title_fullStr Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or Aphasia
title_full_unstemmed Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or Aphasia
title_short Agraphia in Bulbar-Onset Amyotrophic Lateral Sclerosis: Not Merely a Consequence of Dementia or Aphasia
title_sort agraphia in bulbar onset amyotrophic lateral sclerosis not merely a consequence of dementia or aphasia
url http://dx.doi.org/10.3233/BEN-2008-0219
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