White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanism

As direct evaluation of a mouse model of human neurodevelopment, adolescent and young adult mice and humans underwent MR diffusion tensor imaging to quantify age-related differences in microstructural integrity of brain white matter fibers. Fractional anisotropy (FA) was greater in older than younge...

Full description

Saved in:
Bibliographic Details
Main Authors: David J. Piekarski, Natalie M. Zahr, Qingyu Zhao, Uran Ferizi, Kilian M. Pohl, Edith V. Sullivan, Adolf Pfefferbaum
Format: Article
Language:English
Published: Elsevier 2023-09-01
Series:NeuroImage: Reports
Subjects:
Online Access:http://www.sciencedirect.com/science/article/pii/S2666956023000247
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849472551736049664
author David J. Piekarski
Natalie M. Zahr
Qingyu Zhao
Uran Ferizi
Kilian M. Pohl
Edith V. Sullivan
Adolf Pfefferbaum
author_facet David J. Piekarski
Natalie M. Zahr
Qingyu Zhao
Uran Ferizi
Kilian M. Pohl
Edith V. Sullivan
Adolf Pfefferbaum
author_sort David J. Piekarski
collection DOAJ
description As direct evaluation of a mouse model of human neurodevelopment, adolescent and young adult mice and humans underwent MR diffusion tensor imaging to quantify age-related differences in microstructural integrity of brain white matter fibers. Fractional anisotropy (FA) was greater in older than younger mice and humans. Despite the cross-species commonality, the underlying developmental mechanism differed: whereas evidence for greater axonal extension contributed to higher FA in older mice, evidence for continuing myelination contributed to higher FA in human adolescent development. These differences occurred in the context of species distinctions in overall brain growth: whereas the continued growth of the brain and skull in the murine model can accommodate volume expansion into adulthood, human white matter volume and myelination continue growth into adulthood within a fixed intracranial volume. Appreciation of the similarities and differences in developmental mechanism can enhance the utility of animal models of brain white matter structure, function, and response to exogenous manipulation.
format Article
id doaj-art-ac04b80ce71d47cba820051e83fd1a03
institution Kabale University
issn 2666-9560
language English
publishDate 2023-09-01
publisher Elsevier
record_format Article
series NeuroImage: Reports
spelling doaj-art-ac04b80ce71d47cba820051e83fd1a032025-08-20T03:24:30ZengElsevierNeuroImage: Reports2666-95602023-09-013310017910.1016/j.ynirp.2023.100179White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanismDavid J. Piekarski0Natalie M. Zahr1Qingyu Zhao2Uran Ferizi3Kilian M. Pohl4Edith V. Sullivan5Adolf Pfefferbaum6Center for Health Science, SRI International, 333 Ravenswood Ave., Menlo Park, CA, 94015, USA; Corresponding author.Center for Health Science, SRI International, 333 Ravenswood Ave., Menlo Park, CA, 94015, USA; Department of Psychiatry and Behavioral Sciences, Stanford University School of, Medicine, 401 Quarry Rd., Stanford, CA, 94305, USADepartment of Psychiatry and Behavioral Sciences, Stanford University School of, Medicine, 401 Quarry Rd., Stanford, CA, 94305, USACenter for Health Science, SRI International, 333 Ravenswood Ave., Menlo Park, CA, 94015, USA; Department of Psychiatry and Behavioral Sciences, Stanford University School of, Medicine, 401 Quarry Rd., Stanford, CA, 94305, USADepartment of Psychiatry and Behavioral Sciences, Stanford University School of, Medicine, 401 Quarry Rd., Stanford, CA, 94305, USADepartment of Psychiatry and Behavioral Sciences, Stanford University School of, Medicine, 401 Quarry Rd., Stanford, CA, 94305, USACenter for Health Science, SRI International, 333 Ravenswood Ave., Menlo Park, CA, 94015, USA; Department of Psychiatry and Behavioral Sciences, Stanford University School of, Medicine, 401 Quarry Rd., Stanford, CA, 94305, USAAs direct evaluation of a mouse model of human neurodevelopment, adolescent and young adult mice and humans underwent MR diffusion tensor imaging to quantify age-related differences in microstructural integrity of brain white matter fibers. Fractional anisotropy (FA) was greater in older than younger mice and humans. Despite the cross-species commonality, the underlying developmental mechanism differed: whereas evidence for greater axonal extension contributed to higher FA in older mice, evidence for continuing myelination contributed to higher FA in human adolescent development. These differences occurred in the context of species distinctions in overall brain growth: whereas the continued growth of the brain and skull in the murine model can accommodate volume expansion into adulthood, human white matter volume and myelination continue growth into adulthood within a fixed intracranial volume. Appreciation of the similarities and differences in developmental mechanism can enhance the utility of animal models of brain white matter structure, function, and response to exogenous manipulation.http://www.sciencedirect.com/science/article/pii/S2666956023000247AdolescenceDevelopmentDiffusion tensor imagingHumanMouseTranslational
spellingShingle David J. Piekarski
Natalie M. Zahr
Qingyu Zhao
Uran Ferizi
Kilian M. Pohl
Edith V. Sullivan
Adolf Pfefferbaum
White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanism
NeuroImage: Reports
Adolescence
Development
Diffusion tensor imaging
Human
Mouse
Translational
title White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanism
title_full White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanism
title_fullStr White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanism
title_full_unstemmed White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanism
title_short White matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans: Same phenotype, different mechanism
title_sort white matter microstructural integrity continues to develop from adolescence to young adulthood in mice and humans same phenotype different mechanism
topic Adolescence
Development
Diffusion tensor imaging
Human
Mouse
Translational
url http://www.sciencedirect.com/science/article/pii/S2666956023000247
work_keys_str_mv AT davidjpiekarski whitemattermicrostructuralintegritycontinuestodevelopfromadolescencetoyoungadulthoodinmiceandhumanssamephenotypedifferentmechanism
AT nataliemzahr whitemattermicrostructuralintegritycontinuestodevelopfromadolescencetoyoungadulthoodinmiceandhumanssamephenotypedifferentmechanism
AT qingyuzhao whitemattermicrostructuralintegritycontinuestodevelopfromadolescencetoyoungadulthoodinmiceandhumanssamephenotypedifferentmechanism
AT uranferizi whitemattermicrostructuralintegritycontinuestodevelopfromadolescencetoyoungadulthoodinmiceandhumanssamephenotypedifferentmechanism
AT kilianmpohl whitemattermicrostructuralintegritycontinuestodevelopfromadolescencetoyoungadulthoodinmiceandhumanssamephenotypedifferentmechanism
AT edithvsullivan whitemattermicrostructuralintegritycontinuestodevelopfromadolescencetoyoungadulthoodinmiceandhumanssamephenotypedifferentmechanism
AT adolfpfefferbaum whitemattermicrostructuralintegritycontinuestodevelopfromadolescencetoyoungadulthoodinmiceandhumanssamephenotypedifferentmechanism