NHF-derived carbon dots: prevalidation approach in breast cancer treatment

Abstract Metastatic breast cancer dominates the female cancer-related mortality. Tumour-associated molecules represents a crucial for early disease detection and identification of novel therapeutic targets. Nanomaterial technologies provide promising novel approaches to disease diagnostics and thera...

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Main Authors: Crina Elena Tiron, Gabriel Luta, Mihail Butura, Florin Zugun-Eloae, Corneliu S. Stan, Adina Coroaba, Elena-Laura Ursu, Gabriela Dumitrita Stanciu, Adrian Tiron
Format: Article
Language:English
Published: Nature Portfolio 2020-07-01
Series:Scientific Reports
Online Access:https://doi.org/10.1038/s41598-020-69670-z
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author Crina Elena Tiron
Gabriel Luta
Mihail Butura
Florin Zugun-Eloae
Corneliu S. Stan
Adina Coroaba
Elena-Laura Ursu
Gabriela Dumitrita Stanciu
Adrian Tiron
author_facet Crina Elena Tiron
Gabriel Luta
Mihail Butura
Florin Zugun-Eloae
Corneliu S. Stan
Adina Coroaba
Elena-Laura Ursu
Gabriela Dumitrita Stanciu
Adrian Tiron
author_sort Crina Elena Tiron
collection DOAJ
description Abstract Metastatic breast cancer dominates the female cancer-related mortality. Tumour-associated molecules represents a crucial for early disease detection and identification of novel therapeutic targets. Nanomaterial technologies provide promising novel approaches to disease diagnostics and therapeutics. In the present study we extend the investigations of antitumoral properties of Carbon Dots prepared from N-hydroxyphthalimide (CD-NHF) precursor. We evaluate the effect of CD-NHF on tumour cell migration and invasion in vitro and their impact on tumour progression using an in vivo model. Furthermore, we investigate the molecular mechanisms involved in CD-NHF antitumour effects. In vivo mammary tumours were induced in Balb/c female mice by injecting 4T1 cells into the mammary fat pad. Conditional treatment with CD-NHF significantly impair both migration and invasion of metastatic breast cancer cells. The presence of CD-NHF within the 3D cell cultures strongly inhibited the malignant phenotype of MDA-MB-231, 4T1 and MCF-7 cells in 3D culture, resulting in culture colonies lacking invasive projections and reduction of mammospheres formation. Importantly, breast tumour growth and metastasis dissemination was significantly reduced upon CD-NHF treatments in a syngeneic mouse model and is associated with down-regulation of Ki67 and HSP90 expression. CD-NHF nanostructures provide exciting perspective for improving treatment outcome in breast cancer.
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spelling doaj-art-abf2f4bf241c4ae490a603bd6ba5174c2025-08-20T03:45:57ZengNature PortfolioScientific Reports2045-23222020-07-0110111010.1038/s41598-020-69670-zNHF-derived carbon dots: prevalidation approach in breast cancer treatmentCrina Elena Tiron0Gabriel Luta1Mihail Butura2Florin Zugun-Eloae3Corneliu S. Stan4Adina Coroaba5Elena-Laura Ursu6Gabriela Dumitrita Stanciu7Adrian Tiron8Regional Institute of Oncology, TRANSCEND CenterRegional Institute of Oncology, TRANSCEND CenterRegional Institute of Oncology, TRANSCEND CenterRegional Institute of Oncology, TRANSCEND CenterDepartment of Natural and Synthetic Polymers, “Gheorghe Asachi” Technical University of IasiDepartment of Chemistry, “Petru Poni” Institute of Macromolecular ChemistryDepartment of Chemistry, “Petru Poni” Institute of Macromolecular ChemistryCenter for Advanced Research and Development in Experimental Medicine (CEMEX), “Gr.T.Popa” University of Medicine and PharmacyRegional Institute of Oncology, TRANSCEND CenterAbstract Metastatic breast cancer dominates the female cancer-related mortality. Tumour-associated molecules represents a crucial for early disease detection and identification of novel therapeutic targets. Nanomaterial technologies provide promising novel approaches to disease diagnostics and therapeutics. In the present study we extend the investigations of antitumoral properties of Carbon Dots prepared from N-hydroxyphthalimide (CD-NHF) precursor. We evaluate the effect of CD-NHF on tumour cell migration and invasion in vitro and their impact on tumour progression using an in vivo model. Furthermore, we investigate the molecular mechanisms involved in CD-NHF antitumour effects. In vivo mammary tumours were induced in Balb/c female mice by injecting 4T1 cells into the mammary fat pad. Conditional treatment with CD-NHF significantly impair both migration and invasion of metastatic breast cancer cells. The presence of CD-NHF within the 3D cell cultures strongly inhibited the malignant phenotype of MDA-MB-231, 4T1 and MCF-7 cells in 3D culture, resulting in culture colonies lacking invasive projections and reduction of mammospheres formation. Importantly, breast tumour growth and metastasis dissemination was significantly reduced upon CD-NHF treatments in a syngeneic mouse model and is associated with down-regulation of Ki67 and HSP90 expression. CD-NHF nanostructures provide exciting perspective for improving treatment outcome in breast cancer.https://doi.org/10.1038/s41598-020-69670-z
spellingShingle Crina Elena Tiron
Gabriel Luta
Mihail Butura
Florin Zugun-Eloae
Corneliu S. Stan
Adina Coroaba
Elena-Laura Ursu
Gabriela Dumitrita Stanciu
Adrian Tiron
NHF-derived carbon dots: prevalidation approach in breast cancer treatment
Scientific Reports
title NHF-derived carbon dots: prevalidation approach in breast cancer treatment
title_full NHF-derived carbon dots: prevalidation approach in breast cancer treatment
title_fullStr NHF-derived carbon dots: prevalidation approach in breast cancer treatment
title_full_unstemmed NHF-derived carbon dots: prevalidation approach in breast cancer treatment
title_short NHF-derived carbon dots: prevalidation approach in breast cancer treatment
title_sort nhf derived carbon dots prevalidation approach in breast cancer treatment
url https://doi.org/10.1038/s41598-020-69670-z
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