Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent Manner

Chikungunya virus (CHIKV), the causative agent of the chikungunya fever, is an alphavirus widely transmitted by the bite of the female mosquito of the genus <i>Aedes</i> sp., especially in tropical and subtropical regions. Brazil is the country most affected by the microorganism. CHIKV c...

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Main Authors: Liziane C. M. da Silva, Andressa C. dos Santos Maia, Nágila C. F. de Sousa, Catielen P. Pavi, Beatriz P. Savi, Seigo Nagashima, Samara Damasceno, Ayda H. Schneider, Lucas Z. Mascarin, João F. S. Rodrigues, Cinara R. A. V. Monteiro, Izabella T. Silva, Gislaine Fongaro, Valério Monteiro-Neto, Maria R. Q. Bomfim, Thiago M. Cunha, João de Sousa Valente, João B. Calixto, Lúcia de Noronha, Susan D. Brain, Elizabeth S. Fernandes
Format: Article
Language:English
Published: MDPI AG 2025-01-01
Series:Biomolecules
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Online Access:https://www.mdpi.com/2218-273X/15/2/171
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author Liziane C. M. da Silva
Andressa C. dos Santos Maia
Nágila C. F. de Sousa
Catielen P. Pavi
Beatriz P. Savi
Seigo Nagashima
Samara Damasceno
Ayda H. Schneider
Lucas Z. Mascarin
João F. S. Rodrigues
Cinara R. A. V. Monteiro
Izabella T. Silva
Gislaine Fongaro
Valério Monteiro-Neto
Maria R. Q. Bomfim
Thiago M. Cunha
João de Sousa Valente
João B. Calixto
Lúcia de Noronha
Susan D. Brain
Elizabeth S. Fernandes
author_facet Liziane C. M. da Silva
Andressa C. dos Santos Maia
Nágila C. F. de Sousa
Catielen P. Pavi
Beatriz P. Savi
Seigo Nagashima
Samara Damasceno
Ayda H. Schneider
Lucas Z. Mascarin
João F. S. Rodrigues
Cinara R. A. V. Monteiro
Izabella T. Silva
Gislaine Fongaro
Valério Monteiro-Neto
Maria R. Q. Bomfim
Thiago M. Cunha
João de Sousa Valente
João B. Calixto
Lúcia de Noronha
Susan D. Brain
Elizabeth S. Fernandes
author_sort Liziane C. M. da Silva
collection DOAJ
description Chikungunya virus (CHIKV), the causative agent of the chikungunya fever, is an alphavirus widely transmitted by the bite of the female mosquito of the genus <i>Aedes</i> sp., especially in tropical and subtropical regions. Brazil is the country most affected by the microorganism. CHIKV classically induces articular pain, which can become long lasting for even years in a great number of the infected individuals, reducing their quality of life. The mechanisms of CHIKV-induced pain are poorly understood, but recent evidence indicated a role for the transient receptor potential vanilloid 1 (TRPV1) in this pathology. Herein, we assessed the ability of intra-articularly injected inactivated CHIKV or its RNA to trigger nociception in mice. Both stimuli induced bilateral secondary hyperalgesia to mechanical and heat stimuli. These responses were attenuated by TRPV1 ablation or antagonism. Joint structural alterations and increased cartilage TRPV1 protein expression were detected in the ipsilateral knee joints injected with either CHIKV or viral RNA. However, the lack of this receptor did not influence the histological changes triggered by CHIKV or RNA. The results further support the role of TRPV1 in CHIKV-induced pain and highlight its importance in the chronic phase of the disease.
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spelling doaj-art-ab982050d47e4fa490b6c5a85b4e32eb2025-08-20T02:44:36ZengMDPI AGBiomolecules2218-273X2025-01-0115217110.3390/biom15020171Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent MannerLiziane C. M. da Silva0Andressa C. dos Santos Maia1Nágila C. F. de Sousa2Catielen P. Pavi3Beatriz P. Savi4Seigo Nagashima5Samara Damasceno6Ayda H. Schneider7Lucas Z. Mascarin8João F. S. Rodrigues9Cinara R. A. V. Monteiro10Izabella T. Silva11Gislaine Fongaro12Valério Monteiro-Neto13Maria R. Q. Bomfim14Thiago M. Cunha15João de Sousa Valente16João B. Calixto17Lúcia de Noronha18Susan D. Brain19Elizabeth S. Fernandes20Programa de Pós-Graduação em Biotecnologia Aplicada à Saúde da Criança e do Adolescente, Faculdades Pequeno Príncipe, Curitiba 80230-020, PR, BrazilPrograma de Pós-Graduação em Biotecnologia Aplicada à Saúde da Criança e do Adolescente, Faculdades Pequeno Príncipe, Curitiba 80230-020, PR, BrazilPrograma de Pós-Graduação, Universidade CEUMA, São Luís 65075-120, MA, BrazilDepartamento de Microbiologia, Imunologia e Parasitologia, Centro de Ciências Biológicas, Universidade Federal de Santa Catarina, Florianópolis 88040-900, SC, BrazilDepartamento de Microbiologia, Imunologia e Parasitologia, Centro de Ciências Biológicas, Universidade Federal de Santa Catarina, Florianópolis 88040-900, SC, BrazilEscola de Medicina e Ciências da Vida, Pontifícia Universidade Católica do Paraná, Curitiba 80215-901, PR, BrazilFaculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, BrazilFaculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, BrazilDepartamento de Microbiologia, Imunologia e Parasitologia, Centro de Ciências Biológicas, Universidade Federal de Santa Catarina, Florianópolis 88040-900, SC, BrazilPrograma de Pós-Graduação, Universidade CEUMA, São Luís 65075-120, MA, BrazilPrograma de Pós-Graduação em Ciências da Saúde, Universidade Federal do Maranhão, São Luís 65085-040, MA, BrazilDepartamento de Microbiologia, Imunologia e Parasitologia, Centro de Ciências Biológicas, Universidade Federal de Santa Catarina, Florianópolis 88040-900, SC, BrazilDepartamento de Microbiologia, Imunologia e Parasitologia, Centro de Ciências Biológicas, Universidade Federal de Santa Catarina, Florianópolis 88040-900, SC, BrazilPrograma de Pós-Graduação em Ciências da Saúde, Universidade Federal do Maranhão, São Luís 65085-040, MA, BrazilPrograma de Pós-Graduação, Universidade CEUMA, São Luís 65075-120, MA, BrazilFaculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, Ribeirão Preto 14049-900, SP, BrazilThe School of Cardiovascular and Metabolic Medicine (South Bank), King’s College London, London SE1 9NH, UKCentro de Inovação e Ensaios Pré-Clínicos, Florianópolis 88056-000, SC, BrazilEscola de Medicina e Ciências da Vida, Pontifícia Universidade Católica do Paraná, Curitiba 80215-901, PR, BrazilThe School of Cardiovascular and Metabolic Medicine (South Bank), King’s College London, London SE1 9NH, UKPrograma de Pós-Graduação em Biotecnologia Aplicada à Saúde da Criança e do Adolescente, Faculdades Pequeno Príncipe, Curitiba 80230-020, PR, BrazilChikungunya virus (CHIKV), the causative agent of the chikungunya fever, is an alphavirus widely transmitted by the bite of the female mosquito of the genus <i>Aedes</i> sp., especially in tropical and subtropical regions. Brazil is the country most affected by the microorganism. CHIKV classically induces articular pain, which can become long lasting for even years in a great number of the infected individuals, reducing their quality of life. The mechanisms of CHIKV-induced pain are poorly understood, but recent evidence indicated a role for the transient receptor potential vanilloid 1 (TRPV1) in this pathology. Herein, we assessed the ability of intra-articularly injected inactivated CHIKV or its RNA to trigger nociception in mice. Both stimuli induced bilateral secondary hyperalgesia to mechanical and heat stimuli. These responses were attenuated by TRPV1 ablation or antagonism. Joint structural alterations and increased cartilage TRPV1 protein expression were detected in the ipsilateral knee joints injected with either CHIKV or viral RNA. However, the lack of this receptor did not influence the histological changes triggered by CHIKV or RNA. The results further support the role of TRPV1 in CHIKV-induced pain and highlight its importance in the chronic phase of the disease.https://www.mdpi.com/2218-273X/15/2/171Chikungunyaviral RNAarthralgiaTRPV1nociception
spellingShingle Liziane C. M. da Silva
Andressa C. dos Santos Maia
Nágila C. F. de Sousa
Catielen P. Pavi
Beatriz P. Savi
Seigo Nagashima
Samara Damasceno
Ayda H. Schneider
Lucas Z. Mascarin
João F. S. Rodrigues
Cinara R. A. V. Monteiro
Izabella T. Silva
Gislaine Fongaro
Valério Monteiro-Neto
Maria R. Q. Bomfim
Thiago M. Cunha
João de Sousa Valente
João B. Calixto
Lúcia de Noronha
Susan D. Brain
Elizabeth S. Fernandes
Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent Manner
Biomolecules
Chikungunya
viral RNA
arthralgia
TRPV1
nociception
title Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent Manner
title_full Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent Manner
title_fullStr Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent Manner
title_full_unstemmed Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent Manner
title_short Chikungunya Particle and RNA Induce Mechanical and Heat Hypersensitivities in a TRPV1-Dependent Manner
title_sort chikungunya particle and rna induce mechanical and heat hypersensitivities in a trpv1 dependent manner
topic Chikungunya
viral RNA
arthralgia
TRPV1
nociception
url https://www.mdpi.com/2218-273X/15/2/171
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