Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2

Abstract Background Post-prostatectomy urinary incontinence (PPUI) is a common complication for patients with prostate cancer after surgery. MicroRNA-330-3p (miR-330-3p) is down-regulated in stress urinary incontinence patients. However, its clinical role and regulatory mechanism in PPUI remain unkn...

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Main Authors: Xiaoying Feng, Yuanyuan Mi, Mengye Weng
Format: Article
Language:English
Published: BMC 2025-06-01
Series:Hereditas
Subjects:
Online Access:https://doi.org/10.1186/s41065-025-00475-8
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author Xiaoying Feng
Yuanyuan Mi
Mengye Weng
author_facet Xiaoying Feng
Yuanyuan Mi
Mengye Weng
author_sort Xiaoying Feng
collection DOAJ
description Abstract Background Post-prostatectomy urinary incontinence (PPUI) is a common complication for patients with prostate cancer after surgery. MicroRNA-330-3p (miR-330-3p) is down-regulated in stress urinary incontinence patients. However, its clinical role and regulatory mechanism in PPUI remain unknown. Objective To assess the clinical significance of miR-330-3p in PPUI and to explore the potential mechanisms via matrix metalloproteinase 2 (MMP2) regulation. Methods This study enrolled 135 ageing prostate cancer patients (86 without PPUI, 49 with PPUI). Reverse transcription PCR (RT-qPCR) was utilized to measure the levels of miR-330-3p, while Receiver operating characteristic (ROC) analysis was conducted to evaluate the predictive significance of miR-330-3p for PPUI. The proliferative of human urethral fibroblasts (HUFs) was assessed by Cell Counting Kit-8 (CCK-8) assay, while inflammatory cytokines were quantified via enzyme-linked immunosorbent assay (ELISA) kits. Western blot assay was employed to examine the protein levels of extracellular matrix (ECM) remodeling-related markers. The miR-330-3p/MMP2 interaction was validated by dual-luciferase assay. Result miR-330-3p was significantly downregulated in PPUI patients, with low expression predicting PPUI. In HUFs, miR-330-3p overexpression inhibited IL-1β-induced hyperproliferation, inflammation, and ECM degradation. Overexpression of MMP2 counteracted the influence of miR-330-3p mimic on HUFs. Conclusion miR-330-3p is a potential biomarker for PPUI and regulates the function of urethral fibroblasts by targeting MMP2.
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spelling doaj-art-ab71f0e75c5647e8b0e275d2a92a80632025-08-20T02:10:31ZengBMCHereditas1601-52232025-06-01162111110.1186/s41065-025-00475-8Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2Xiaoying Feng0Yuanyuan Mi1Mengye Weng2Department of Urology Surgery, Affiliated Hospital of Jiangnan UniversityDepartment of Urology Surgery, Affiliated Hospital of Jiangnan UniversityDepartment of Orthopedics, Affiliated Hospital of Jiangnan UniversityAbstract Background Post-prostatectomy urinary incontinence (PPUI) is a common complication for patients with prostate cancer after surgery. MicroRNA-330-3p (miR-330-3p) is down-regulated in stress urinary incontinence patients. However, its clinical role and regulatory mechanism in PPUI remain unknown. Objective To assess the clinical significance of miR-330-3p in PPUI and to explore the potential mechanisms via matrix metalloproteinase 2 (MMP2) regulation. Methods This study enrolled 135 ageing prostate cancer patients (86 without PPUI, 49 with PPUI). Reverse transcription PCR (RT-qPCR) was utilized to measure the levels of miR-330-3p, while Receiver operating characteristic (ROC) analysis was conducted to evaluate the predictive significance of miR-330-3p for PPUI. The proliferative of human urethral fibroblasts (HUFs) was assessed by Cell Counting Kit-8 (CCK-8) assay, while inflammatory cytokines were quantified via enzyme-linked immunosorbent assay (ELISA) kits. Western blot assay was employed to examine the protein levels of extracellular matrix (ECM) remodeling-related markers. The miR-330-3p/MMP2 interaction was validated by dual-luciferase assay. Result miR-330-3p was significantly downregulated in PPUI patients, with low expression predicting PPUI. In HUFs, miR-330-3p overexpression inhibited IL-1β-induced hyperproliferation, inflammation, and ECM degradation. Overexpression of MMP2 counteracted the influence of miR-330-3p mimic on HUFs. Conclusion miR-330-3p is a potential biomarker for PPUI and regulates the function of urethral fibroblasts by targeting MMP2.https://doi.org/10.1186/s41065-025-00475-8miR-330-3pMMP2Post-prostatectomy urinary incontinenceInflammationECM remodeling
spellingShingle Xiaoying Feng
Yuanyuan Mi
Mengye Weng
Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2
Hereditas
miR-330-3p
MMP2
Post-prostatectomy urinary incontinence
Inflammation
ECM remodeling
title Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2
title_full Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2
title_fullStr Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2
title_full_unstemmed Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2
title_short Abnormal expression of miR-330-3p predicts post-prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting MMP2
title_sort abnormal expression of mir 330 3p predicts post prostatectomy urinary incontinence and regulates the function of urethral fibroblasts by targeting mmp2
topic miR-330-3p
MMP2
Post-prostatectomy urinary incontinence
Inflammation
ECM remodeling
url https://doi.org/10.1186/s41065-025-00475-8
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AT yuanyuanmi abnormalexpressionofmir3303ppredictspostprostatectomyurinaryincontinenceandregulatesthefunctionofurethralfibroblastsbytargetingmmp2
AT mengyeweng abnormalexpressionofmir3303ppredictspostprostatectomyurinaryincontinenceandregulatesthefunctionofurethralfibroblastsbytargetingmmp2