Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviors

Abstract Tinnitus has been identified as a potential contributor to anxiety. Thalamo-cortical pathway plays a crucial role in the transmission of auditory and emotional information, but its casual link to tinnitus-associated anxiety remains unclear. In this study, we explore the neural activities in...

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Main Authors: Jingyu Chen, Xueru Wang, Zijie Li, Hui Yuan, Xuejiao Wang, Yang Yun, Xu Wu, Pingting Yang, Ling Qin
Format: Article
Language:English
Published: Nature Portfolio 2024-10-01
Series:Communications Biology
Online Access:https://doi.org/10.1038/s42003-024-07040-5
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author Jingyu Chen
Xueru Wang
Zijie Li
Hui Yuan
Xuejiao Wang
Yang Yun
Xu Wu
Pingting Yang
Ling Qin
author_facet Jingyu Chen
Xueru Wang
Zijie Li
Hui Yuan
Xuejiao Wang
Yang Yun
Xu Wu
Pingting Yang
Ling Qin
author_sort Jingyu Chen
collection DOAJ
description Abstract Tinnitus has been identified as a potential contributor to anxiety. Thalamo-cortical pathway plays a crucial role in the transmission of auditory and emotional information, but its casual link to tinnitus-associated anxiety remains unclear. In this study, we explore the neural activities in the thalamus and cortex of the sodium salicylate (NaSal)-treated mice, which exhibit both hyperacusis and anxiety-like behaviors. We find an increase in gamma band oscillations (GBO) in both auditory cortex (AC) and prefrontal cortex (PFC), as well as phase-locking between cortical GBO and thalamic neural activity. These changes are attributable to a suppression of GABAergic neuron activity in thalamic reticular nucleus (TRN), and optogenetic activation of TRN reduces NaSal-induced hyperacusis and anxiety-like behaviors. The elevation of endocannabinoid (eCB)/ cannabinoid receptor 1 (CB1R) transmission in TRN contributes to the NaSal-induced abnormalities. Our results highlight the regulative role of TRN in the auditory and limbic thalamic-cortical pathways.
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issn 2399-3642
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spelling doaj-art-aabaa1b579dc4bcaad3fe5886b785a0c2025-08-20T02:17:46ZengNature PortfolioCommunications Biology2399-36422024-10-017111710.1038/s42003-024-07040-5Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviorsJingyu Chen0Xueru Wang1Zijie Li2Hui Yuan3Xuejiao Wang4Yang Yun5Xu Wu6Pingting Yang7Ling Qin8Department of Physiology, School of Life Sciences, China Medical UniversityLaboratory of Hearing Research, School of Life Sciences, China Medical UniversityDepartment of Physiology, School of Life Sciences, China Medical UniversityLaboratory of Hearing Research, School of Life Sciences, China Medical UniversityDepartment of Physiology, School of Life Sciences, China Medical UniversityDepartment of Nephrology, Shengjing Hospital of China Medical UniversityDepartment of Forensic Pathology, China Medical University School of Forensic MedicineDepartment of Rheumatology and Immunology, The First Hospital of China Medical UniversityLaboratory of Hearing Research, School of Life Sciences, China Medical UniversityAbstract Tinnitus has been identified as a potential contributor to anxiety. Thalamo-cortical pathway plays a crucial role in the transmission of auditory and emotional information, but its casual link to tinnitus-associated anxiety remains unclear. In this study, we explore the neural activities in the thalamus and cortex of the sodium salicylate (NaSal)-treated mice, which exhibit both hyperacusis and anxiety-like behaviors. We find an increase in gamma band oscillations (GBO) in both auditory cortex (AC) and prefrontal cortex (PFC), as well as phase-locking between cortical GBO and thalamic neural activity. These changes are attributable to a suppression of GABAergic neuron activity in thalamic reticular nucleus (TRN), and optogenetic activation of TRN reduces NaSal-induced hyperacusis and anxiety-like behaviors. The elevation of endocannabinoid (eCB)/ cannabinoid receptor 1 (CB1R) transmission in TRN contributes to the NaSal-induced abnormalities. Our results highlight the regulative role of TRN in the auditory and limbic thalamic-cortical pathways.https://doi.org/10.1038/s42003-024-07040-5
spellingShingle Jingyu Chen
Xueru Wang
Zijie Li
Hui Yuan
Xuejiao Wang
Yang Yun
Xu Wu
Pingting Yang
Ling Qin
Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviors
Communications Biology
title Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviors
title_full Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviors
title_fullStr Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviors
title_full_unstemmed Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviors
title_short Thalamo-cortical neural mechanism of sodium salicylate-induced hyperacusis and anxiety-like behaviors
title_sort thalamo cortical neural mechanism of sodium salicylate induced hyperacusis and anxiety like behaviors
url https://doi.org/10.1038/s42003-024-07040-5
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