ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axis

Abstract OTU deubiquitinase 6B (OTUD6B) study in tumors is gradually increasing; however, studies on the role of OTUD6B in colorectal cancer (CRC) are rare. OTUD6B was overexpressed in some human CRC and liver metastasis samples. Although OTUD6B facilitated migration and invasion in CRC cells, it ex...

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Main Authors: Jinglei Li, Kunpeng Huang, Bing Yang, Xia Hu, Bosheng Mei, Xiang Cheng, Xin Zhong, Chuyi Cao, Zihan Chen, Hui Wang, Jinxiang Zhang
Format: Article
Language:English
Published: Nature Publishing Group 2025-07-01
Series:Cell Death and Disease
Online Access:https://doi.org/10.1038/s41419-025-07837-0
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author Jinglei Li
Kunpeng Huang
Bing Yang
Xia Hu
Bosheng Mei
Xiang Cheng
Xin Zhong
Chuyi Cao
Zihan Chen
Hui Wang
Jinxiang Zhang
author_facet Jinglei Li
Kunpeng Huang
Bing Yang
Xia Hu
Bosheng Mei
Xiang Cheng
Xin Zhong
Chuyi Cao
Zihan Chen
Hui Wang
Jinxiang Zhang
author_sort Jinglei Li
collection DOAJ
description Abstract OTU deubiquitinase 6B (OTUD6B) study in tumors is gradually increasing; however, studies on the role of OTUD6B in colorectal cancer (CRC) are rare. OTUD6B was overexpressed in some human CRC and liver metastasis samples. Although OTUD6B facilitated migration and invasion in CRC cells, it exhibited opposite effects on liver metastasis in immunodeficient and immunocompetent mice. We demonstrated that Otud6b enhanced metastasis in nude mice, but it recruited more CD8+ T cell infiltration in colorectal liver metastasis (CRLM) mouse model of C57BL/6J to inhibit CRLM through upregulating Cxcl11. Furthermore, we demonstrated that OTUD6B deubiquitinated and stabilized DDX5. Ectopically expressed DDX5 facilitated transcription factor STAT3 activation by resolving the RNA G-quadruplex structure of STAT3, resulting in a higher level of CXCL11 transcription and an increase in tumor-infiltrating CD8+ T cells. All-trans retinoic acid inhibited CRLM by upregulating OTUD6B.
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language English
publishDate 2025-07-01
publisher Nature Publishing Group
record_format Article
series Cell Death and Disease
spelling doaj-art-a9fb06b5bf7943f9a4dd50c7a73cd9912025-08-20T03:06:10ZengNature Publishing GroupCell Death and Disease2041-48892025-07-0116111410.1038/s41419-025-07837-0ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axisJinglei Li0Kunpeng Huang1Bing Yang2Xia Hu3Bosheng Mei4Xiang Cheng5Xin Zhong6Chuyi Cao7Zihan Chen8Hui Wang9Jinxiang Zhang10Department of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Gastroenterology, Second Affiliated Hospital of Nanchang UniversityDepartment of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyCancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Medical Genetics, Basic School of Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Medical Genetics, Basic School of Tongji Medical College, Huazhong University of Science and TechnologyDepartment of Emergency Surgery, Union Hospital, Tongji Medical College, Huazhong University of Science and TechnologyAbstract OTU deubiquitinase 6B (OTUD6B) study in tumors is gradually increasing; however, studies on the role of OTUD6B in colorectal cancer (CRC) are rare. OTUD6B was overexpressed in some human CRC and liver metastasis samples. Although OTUD6B facilitated migration and invasion in CRC cells, it exhibited opposite effects on liver metastasis in immunodeficient and immunocompetent mice. We demonstrated that Otud6b enhanced metastasis in nude mice, but it recruited more CD8+ T cell infiltration in colorectal liver metastasis (CRLM) mouse model of C57BL/6J to inhibit CRLM through upregulating Cxcl11. Furthermore, we demonstrated that OTUD6B deubiquitinated and stabilized DDX5. Ectopically expressed DDX5 facilitated transcription factor STAT3 activation by resolving the RNA G-quadruplex structure of STAT3, resulting in a higher level of CXCL11 transcription and an increase in tumor-infiltrating CD8+ T cells. All-trans retinoic acid inhibited CRLM by upregulating OTUD6B.https://doi.org/10.1038/s41419-025-07837-0
spellingShingle Jinglei Li
Kunpeng Huang
Bing Yang
Xia Hu
Bosheng Mei
Xiang Cheng
Xin Zhong
Chuyi Cao
Zihan Chen
Hui Wang
Jinxiang Zhang
ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axis
Cell Death and Disease
title ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axis
title_full ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axis
title_fullStr ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axis
title_full_unstemmed ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axis
title_short ATRA upregulates OTUD6B to recruit CD8+ T cells to suppress colorectal liver metastasis by stabilizing DDX5/STAT3/CXCL11 axis
title_sort atra upregulates otud6b to recruit cd8 t cells to suppress colorectal liver metastasis by stabilizing ddx5 stat3 cxcl11 axis
url https://doi.org/10.1038/s41419-025-07837-0
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