Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD Mice

Recent evidence has highlighted the role of histamine in inflammation. Since this monoamine has also been strongly implicated in the pathogenesis of type-1 diabetes, we assessed its effect in the nonobese diabetic (NOD) mouse model. To this end, we used mice (inactivated) knocked out for the gene en...

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Main Authors: Manal Alkan, François Machavoine, Rachel Rignault, Julie Dam, Michel Dy, Nathalie Thieblemont
Format: Article
Language:English
Published: Wiley 2015-01-01
Series:Journal of Diabetes Research
Online Access:http://dx.doi.org/10.1155/2015/965056
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author Manal Alkan
François Machavoine
Rachel Rignault
Julie Dam
Michel Dy
Nathalie Thieblemont
author_facet Manal Alkan
François Machavoine
Rachel Rignault
Julie Dam
Michel Dy
Nathalie Thieblemont
author_sort Manal Alkan
collection DOAJ
description Recent evidence has highlighted the role of histamine in inflammation. Since this monoamine has also been strongly implicated in the pathogenesis of type-1 diabetes, we assessed its effect in the nonobese diabetic (NOD) mouse model. To this end, we used mice (inactivated) knocked out for the gene encoding histidine decarboxylase, the unique histamine-forming enzyme, backcrossed on a NOD genetic background. We found that the lack of endogenous histamine in NOD HDC−/− mice decreased the incidence of diabetes in relation to their wild-type counterpart. Whereas the proportion of regulatory T and myeloid-derived suppressive cells was similar in both strains, histamine deficiency was associated with increased levels of immature macrophages, as compared with wild-type NOD mice. Concerning the cytokine pattern, we found a decrease in circulating IL-12 and IFN-γ in HDC−/− mice, while IL-6 or leptin remained unchanged, suggesting that histamine primarily modulates the inflammatory environment. Paradoxically, exogenous histamine given to NOD HDC−/− mice provided also protection against T1D. Our study supports the notion that histamine is involved in the pathogenesis of diabetes, thus providing additional evidence for its role in the regulation of the immune response.
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series Journal of Diabetes Research
spelling doaj-art-a9e5ec5bd9b143638d8229a926e4eec92025-08-20T02:20:40ZengWileyJournal of Diabetes Research2314-67452314-67532015-01-01201510.1155/2015/965056965056Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD MiceManal Alkan0François Machavoine1Rachel Rignault2Julie Dam3Michel Dy4Nathalie Thieblemont5Université Paris Descartes, 75014 Paris, FranceUniversité Paris Descartes, 75014 Paris, FranceUniversité Paris Descartes, 75014 Paris, FranceUniversité Paris Descartes, 75014 Paris, FranceUniversité Paris Descartes, 75014 Paris, FranceUniversité Paris Descartes, 75014 Paris, FranceRecent evidence has highlighted the role of histamine in inflammation. Since this monoamine has also been strongly implicated in the pathogenesis of type-1 diabetes, we assessed its effect in the nonobese diabetic (NOD) mouse model. To this end, we used mice (inactivated) knocked out for the gene encoding histidine decarboxylase, the unique histamine-forming enzyme, backcrossed on a NOD genetic background. We found that the lack of endogenous histamine in NOD HDC−/− mice decreased the incidence of diabetes in relation to their wild-type counterpart. Whereas the proportion of regulatory T and myeloid-derived suppressive cells was similar in both strains, histamine deficiency was associated with increased levels of immature macrophages, as compared with wild-type NOD mice. Concerning the cytokine pattern, we found a decrease in circulating IL-12 and IFN-γ in HDC−/− mice, while IL-6 or leptin remained unchanged, suggesting that histamine primarily modulates the inflammatory environment. Paradoxically, exogenous histamine given to NOD HDC−/− mice provided also protection against T1D. Our study supports the notion that histamine is involved in the pathogenesis of diabetes, thus providing additional evidence for its role in the regulation of the immune response.http://dx.doi.org/10.1155/2015/965056
spellingShingle Manal Alkan
François Machavoine
Rachel Rignault
Julie Dam
Michel Dy
Nathalie Thieblemont
Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD Mice
Journal of Diabetes Research
title Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD Mice
title_full Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD Mice
title_fullStr Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD Mice
title_full_unstemmed Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD Mice
title_short Histidine Decarboxylase Deficiency Prevents Autoimmune Diabetes in NOD Mice
title_sort histidine decarboxylase deficiency prevents autoimmune diabetes in nod mice
url http://dx.doi.org/10.1155/2015/965056
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AT rachelrignault histidinedecarboxylasedeficiencypreventsautoimmunediabetesinnodmice
AT juliedam histidinedecarboxylasedeficiencypreventsautoimmunediabetesinnodmice
AT micheldy histidinedecarboxylasedeficiencypreventsautoimmunediabetesinnodmice
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