Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease

Complications of short bowel syndrome (SBS) include malabsorption and bacterial overgrowth, requiring prolonged dependence on parenteral nutrition (PN). We hypothesized that the intolerance of whole food in some SBS patients might be due to the effect of dietary fiber on the gut microbiome. Shotgun...

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Main Authors: Arthur Kastl, Wenjing Zong, Victoria M. Gershuni, Elliot S. Friedman, Ceylan Tanes, Adoma Boateng, William J. Mitchell, Kathleen O’Connor, Kyle Bittinger, Natalie A. Terry, Christina Bales, Lindsey Albenberg, Gary D. Wu
Format: Article
Language:English
Published: Taylor & Francis Group 2022-12-01
Series:Gut Microbes
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Online Access:https://www.tandfonline.com/doi/10.1080/19490976.2022.2083417
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author Arthur Kastl
Wenjing Zong
Victoria M. Gershuni
Elliot S. Friedman
Ceylan Tanes
Adoma Boateng
William J. Mitchell
Kathleen O’Connor
Kyle Bittinger
Natalie A. Terry
Christina Bales
Lindsey Albenberg
Gary D. Wu
author_facet Arthur Kastl
Wenjing Zong
Victoria M. Gershuni
Elliot S. Friedman
Ceylan Tanes
Adoma Boateng
William J. Mitchell
Kathleen O’Connor
Kyle Bittinger
Natalie A. Terry
Christina Bales
Lindsey Albenberg
Gary D. Wu
author_sort Arthur Kastl
collection DOAJ
description Complications of short bowel syndrome (SBS) include malabsorption and bacterial overgrowth, requiring prolonged dependence on parenteral nutrition (PN). We hypothesized that the intolerance of whole food in some SBS patients might be due to the effect of dietary fiber on the gut microbiome. Shotgun metagenomic sequencing and targeted metabolomics were performed using biospecimens collected from 55 children with SBS and a murine dietary fiber model. Bioinformatic analyses were performed on these datasets as well as from a healthy human dietary intervention study. Compared to healthy controls, the gut microbiota in SBS had lower diversity and increased Proteobacteria, a pattern most pronounced in children on PN and inversely correlated with whole food consumption. Whole food intake correlated with increased glycoside hydrolases (GH) and bile salt hydrolases (BSH) with reduced fecal conjugated bile acids suggesting that dietary fiber regulates BSH activity via GHs. Mechanistic evidence supporting this notion was generated via fecal and plasma bile acid profiling in a healthy human fiber-free dietary intervention study as well as in a dietary fiber mouse experiment. Gaussian mixture modeling of fecal bile acids was used to identify three clinically relevant SBS phenotypes. Dietary fiber is associated with bile acid deconjugation likely via an interaction between gut microbiota BSHs and GHs in the small intestine, which may lead to whole food intolerance in patients with SBS. This mechanism not only has potential utility in clinical phenotyping and targeted therapeutics in SBS based on bile acid metabolism but may have relevance to other intestinal disease states.
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spelling doaj-art-a9b52c19c2ba4c99bafd0df8dcf7a6da2025-08-20T03:05:25ZengTaylor & Francis GroupGut Microbes1949-09761949-09842022-12-0114110.1080/19490976.2022.2083417Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human diseaseArthur Kastl0Wenjing Zong1Victoria M. Gershuni2Elliot S. Friedman3Ceylan Tanes4Adoma Boateng5William J. Mitchell6Kathleen O’Connor7Kyle Bittinger8Natalie A. Terry9Christina Bales10Lindsey Albenberg11Gary D. Wu12Division of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADepartment of Surgery, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USADivision of Gastroenterology and Hepatology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology and Hepatology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology, Hepatology, and Nutrition, the Children’s Hospital of Philadelphia, Philadelphia, PA, USADivision of Gastroenterology and Hepatology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USAComplications of short bowel syndrome (SBS) include malabsorption and bacterial overgrowth, requiring prolonged dependence on parenteral nutrition (PN). We hypothesized that the intolerance of whole food in some SBS patients might be due to the effect of dietary fiber on the gut microbiome. Shotgun metagenomic sequencing and targeted metabolomics were performed using biospecimens collected from 55 children with SBS and a murine dietary fiber model. Bioinformatic analyses were performed on these datasets as well as from a healthy human dietary intervention study. Compared to healthy controls, the gut microbiota in SBS had lower diversity and increased Proteobacteria, a pattern most pronounced in children on PN and inversely correlated with whole food consumption. Whole food intake correlated with increased glycoside hydrolases (GH) and bile salt hydrolases (BSH) with reduced fecal conjugated bile acids suggesting that dietary fiber regulates BSH activity via GHs. Mechanistic evidence supporting this notion was generated via fecal and plasma bile acid profiling in a healthy human fiber-free dietary intervention study as well as in a dietary fiber mouse experiment. Gaussian mixture modeling of fecal bile acids was used to identify three clinically relevant SBS phenotypes. Dietary fiber is associated with bile acid deconjugation likely via an interaction between gut microbiota BSHs and GHs in the small intestine, which may lead to whole food intolerance in patients with SBS. This mechanism not only has potential utility in clinical phenotyping and targeted therapeutics in SBS based on bile acid metabolism but may have relevance to other intestinal disease states.https://www.tandfonline.com/doi/10.1080/19490976.2022.2083417Short bowel syndromeparenteral nutritionmicrobiotabile acids
spellingShingle Arthur Kastl
Wenjing Zong
Victoria M. Gershuni
Elliot S. Friedman
Ceylan Tanes
Adoma Boateng
William J. Mitchell
Kathleen O’Connor
Kyle Bittinger
Natalie A. Terry
Christina Bales
Lindsey Albenberg
Gary D. Wu
Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease
Gut Microbes
Short bowel syndrome
parenteral nutrition
microbiota
bile acids
title Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease
title_full Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease
title_fullStr Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease
title_full_unstemmed Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease
title_short Dietary fiber-based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease
title_sort dietary fiber based regulation of bile salt hydrolase activity in the gut microbiota and its relevance to human disease
topic Short bowel syndrome
parenteral nutrition
microbiota
bile acids
url https://www.tandfonline.com/doi/10.1080/19490976.2022.2083417
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