The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast Cancer

The microenvironment at the site of tumor metastasis plays a key role in determining the fate of the metastasizing tumor cells. This ultimately has a direct impact on the progression of cancer. Bone is the preferred site of metastasis of breast cancer. Painful, debilitating osteolytic lesions are fo...

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Main Authors: Shamik Das, Rajeev S. Samant, Lalita A. Shevde
Format: Article
Language:English
Published: Wiley 2012-01-01
Series:International Journal of Breast Cancer
Online Access:http://dx.doi.org/10.1155/2012/298623
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author Shamik Das
Rajeev S. Samant
Lalita A. Shevde
author_facet Shamik Das
Rajeev S. Samant
Lalita A. Shevde
author_sort Shamik Das
collection DOAJ
description The microenvironment at the site of tumor metastasis plays a key role in determining the fate of the metastasizing tumor cells. This ultimately has a direct impact on the progression of cancer. Bone is the preferred site of metastasis of breast cancer. Painful, debilitating osteolytic lesions are formed as a result of crosstalk between breast cancer cells and cells in the bone, predominantly the osteoblasts and osteoclasts. In this paper, we have discussed the temporal and spatial role of hedgehog (Hh) signaling in influencing the fate of metastatic breast cancer cells in bone. By virtue of its secreted ligands, the Hh pathway is capable of homotypic and heterotypic signaling and consequently altering the microenvironment in the bone. We also have put into perspective the therapeutic implications of using Hh inhibitors to prevent and/or treat bone metastases of breast cancer.
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publishDate 2012-01-01
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series International Journal of Breast Cancer
spelling doaj-art-a991d596ce9d4988a817af1c01b0c4ef2025-08-20T03:39:10ZengWileyInternational Journal of Breast Cancer2090-31702090-31892012-01-01201210.1155/2012/298623298623The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast CancerShamik Das0Rajeev S. Samant1Lalita A. Shevde2Department of Oncologic Sciences, USA Mitchell Cancer Institute, Mobile, AL 36608, USADepartment of Oncologic Sciences, USA Mitchell Cancer Institute, Mobile, AL 36608, USADepartment of Oncologic Sciences, USA Mitchell Cancer Institute, Mobile, AL 36608, USAThe microenvironment at the site of tumor metastasis plays a key role in determining the fate of the metastasizing tumor cells. This ultimately has a direct impact on the progression of cancer. Bone is the preferred site of metastasis of breast cancer. Painful, debilitating osteolytic lesions are formed as a result of crosstalk between breast cancer cells and cells in the bone, predominantly the osteoblasts and osteoclasts. In this paper, we have discussed the temporal and spatial role of hedgehog (Hh) signaling in influencing the fate of metastatic breast cancer cells in bone. By virtue of its secreted ligands, the Hh pathway is capable of homotypic and heterotypic signaling and consequently altering the microenvironment in the bone. We also have put into perspective the therapeutic implications of using Hh inhibitors to prevent and/or treat bone metastases of breast cancer.http://dx.doi.org/10.1155/2012/298623
spellingShingle Shamik Das
Rajeev S. Samant
Lalita A. Shevde
The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast Cancer
International Journal of Breast Cancer
title The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast Cancer
title_full The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast Cancer
title_fullStr The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast Cancer
title_full_unstemmed The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast Cancer
title_short The Hedgehog Pathway Conditions the Bone Microenvironment for Osteolytic Metastasis of Breast Cancer
title_sort hedgehog pathway conditions the bone microenvironment for osteolytic metastasis of breast cancer
url http://dx.doi.org/10.1155/2012/298623
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