Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)

Triple negative breast cancer (TNBC) is the most aggressive molecular subtype and it lacks targetable receptors. Patients have an increased risk of recurrence and poor prognosis. Little is known concerning the characteristics of disseminated tumor cells (DTCs) and their role in TNBC patients. We ana...

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Main Authors: Anne Eckardt, Ivonne Nel, Laura Weydandt, Elisa Brochwitz, Anne Kathrin Höhn, Karsten Winter, Bahriye Aktas
Format: Article
Language:English
Published: MDPI AG 2025-06-01
Series:Cells
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Online Access:https://www.mdpi.com/2073-4409/14/12/857
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author Anne Eckardt
Ivonne Nel
Laura Weydandt
Elisa Brochwitz
Anne Kathrin Höhn
Karsten Winter
Bahriye Aktas
author_facet Anne Eckardt
Ivonne Nel
Laura Weydandt
Elisa Brochwitz
Anne Kathrin Höhn
Karsten Winter
Bahriye Aktas
author_sort Anne Eckardt
collection DOAJ
description Triple negative breast cancer (TNBC) is the most aggressive molecular subtype and it lacks targetable receptors. Patients have an increased risk of recurrence and poor prognosis. Little is known concerning the characteristics of disseminated tumor cells (DTCs) and their role in TNBC patients. We analyzed the bone marrow aspirates of 80 patients with primary (<i>n</i> = 67) or recurrent (<i>n</i> = 13) TNBC, using a multi-parameter immunofluorescence staining procedure, including Pan-CK as an epithelial marker, vimentin (vim) as a marker of epithelial–mesenchymal transition, Ki67 for cell proliferation, and HER2 as well as PD-L1 as therapy-related markers. The DTC positive rate was 56% (<i>n</i>= 45) among the cohort. We found 20 different DTC subpopulations. The most frequently detected profile was CK+Vim+Ki67+ (<i>n</i> = 75 cells). The occurrence of CK- DTCs (<i>n</i> = 69) was significantly correlated to PD-L1 (r = −0.305, <i>p</i> < 0.01) and HER2 positivity (r = −0.234, <i>p</i> < 0.001). DTC positive patients that received neoadjuvant chemotherapy (NACT) and did not reach pathologic complete response were more likely to have CK- DTCs. Our data indicate that the occurrence of DTC subpopulations positive for Vim, Ki67, and HER2 appear to be markers for bad prognosis and could be therapeutically relevant. Furthermore, our results raise the question of whether DTCs are dormant in TNBC patients and persistent towards chemotherapy.
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spelling doaj-art-a95b1261d9ff4dd19e3ea97bb060d9c82025-08-20T02:24:37ZengMDPI AGCells2073-44092025-06-01141285710.3390/cells14120857Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)Anne Eckardt0Ivonne Nel1Laura Weydandt2Elisa Brochwitz3Anne Kathrin Höhn4Karsten Winter5Bahriye Aktas6Department of Gynecology, Medical Center, Leipzig University, 04103 Leipzig, GermanyDepartment of Gynecology, Medical Center, Leipzig University, 04103 Leipzig, GermanyDepartment of Gynecology, Medical Center, Leipzig University, 04103 Leipzig, GermanyDepartment of Gynecology, Medical Center, Leipzig University, 04103 Leipzig, GermanyDepartment of Pathology, Medical Center, Leipzig University, 04103 Leipzig, GermanyInstitute of Anatomy, Medical Faculty, Leipzig University, 04103 Leipzig, GermanyDepartment of Gynecology, Medical Center, Leipzig University, 04103 Leipzig, GermanyTriple negative breast cancer (TNBC) is the most aggressive molecular subtype and it lacks targetable receptors. Patients have an increased risk of recurrence and poor prognosis. Little is known concerning the characteristics of disseminated tumor cells (DTCs) and their role in TNBC patients. We analyzed the bone marrow aspirates of 80 patients with primary (<i>n</i> = 67) or recurrent (<i>n</i> = 13) TNBC, using a multi-parameter immunofluorescence staining procedure, including Pan-CK as an epithelial marker, vimentin (vim) as a marker of epithelial–mesenchymal transition, Ki67 for cell proliferation, and HER2 as well as PD-L1 as therapy-related markers. The DTC positive rate was 56% (<i>n</i>= 45) among the cohort. We found 20 different DTC subpopulations. The most frequently detected profile was CK+Vim+Ki67+ (<i>n</i> = 75 cells). The occurrence of CK- DTCs (<i>n</i> = 69) was significantly correlated to PD-L1 (r = −0.305, <i>p</i> < 0.01) and HER2 positivity (r = −0.234, <i>p</i> < 0.001). DTC positive patients that received neoadjuvant chemotherapy (NACT) and did not reach pathologic complete response were more likely to have CK- DTCs. Our data indicate that the occurrence of DTC subpopulations positive for Vim, Ki67, and HER2 appear to be markers for bad prognosis and could be therapeutically relevant. Furthermore, our results raise the question of whether DTCs are dormant in TNBC patients and persistent towards chemotherapy.https://www.mdpi.com/2073-4409/14/12/857triple negativebreast cancer (TNBC)disseminated tumor cells (DTCs)bone marrowneoadjuvant chemotherapypathologic complete remission (pCR)
spellingShingle Anne Eckardt
Ivonne Nel
Laura Weydandt
Elisa Brochwitz
Anne Kathrin Höhn
Karsten Winter
Bahriye Aktas
Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)
Cells
triple negative
breast cancer (TNBC)
disseminated tumor cells (DTCs)
bone marrow
neoadjuvant chemotherapy
pathologic complete remission (pCR)
title Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)
title_full Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)
title_fullStr Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)
title_full_unstemmed Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)
title_short Characterization of Disseminated Tumor Cells (DTCs) in Patients with Triple-Negative Breast Cancer (TNBC)
title_sort characterization of disseminated tumor cells dtcs in patients with triple negative breast cancer tnbc
topic triple negative
breast cancer (TNBC)
disseminated tumor cells (DTCs)
bone marrow
neoadjuvant chemotherapy
pathologic complete remission (pCR)
url https://www.mdpi.com/2073-4409/14/12/857
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