Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels

Background. Cisplatin induces nephrotoxicity and this study evaluated the protective effect of fosfomycin on it in 11 gynecological cancer patients. Methods. The N‐acetyl‐β‐D‐glucosaminidase (NAG), β2‐microglobulin (β2MG), creatinine (uCr) and total protein (TP) levels in a 24‐hour urine specimen as...

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Main Authors: Masatoshi Hayashi, Masahide Numaguchi, Hideki Watabe, Hideo Enomoto, Yoshimasa Yaoi
Format: Article
Language:English
Published: Wiley 1997-06-01
Series:Acta Obstetricia et Gynecologica Scandinavica
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Online Access:https://doi.org/10.3109/00016349709024590
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author Masatoshi Hayashi
Masahide Numaguchi
Hideki Watabe
Hideo Enomoto
Yoshimasa Yaoi
author_facet Masatoshi Hayashi
Masahide Numaguchi
Hideki Watabe
Hideo Enomoto
Yoshimasa Yaoi
author_sort Masatoshi Hayashi
collection DOAJ
description Background. Cisplatin induces nephrotoxicity and this study evaluated the protective effect of fosfomycin on it in 11 gynecological cancer patients. Methods. The N‐acetyl‐β‐D‐glucosaminidase (NAG), β2‐microglobulin (β2MG), creatinine (uCr) and total protein (TP) levels in a 24‐hour urine specimen as well as the blood urea nitrogen (BUN) and serum creatinine (sCr) were measured before and after CAPF chemotherapy alone (control) or with fosfomycin. Results. The results were statistically analyzed by using the t‐test. NAG, β2MG, uCr and TP levels increased significantly after chemotherapy in the control patients, but BUN and sCr levels did not change significantly. The NAG level in the control group was twice as high as in the fosfomycin group 8 days after chemotherapy (p<0.01). The uCr and TP in control patients increased significantly after chemotherapy when compared to those in patients coadministered fosfomycin. There were no significant changes in β2MG, BUN and sCr levels. Conclusions. Cisplatin affected the levels of NAG, β2MG, uCr and TP without influencing BUN and sCr levels. Fosfomycin, therefore, may be useful as a supplemental treatment for reducing cisplatin nephrotoxicity, especially proximal tubular damage.
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spelling doaj-art-a83425adcc8540aba9d0750ef178cca52025-08-20T02:37:32ZengWileyActa Obstetricia et Gynecologica Scandinavica0001-63491600-04121997-06-0176659059510.3109/00016349709024590Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levelsMasatoshi Hayashi0Masahide Numaguchi1Hideki Watabe2Hideo Enomoto3Yoshimasa Yaoi4Department of Obstetrics and Gynecology, Koshigaya Hospital, Dokkyo University School of Medicine, Minami‐Koshigaya, Koshigaya‐shi, Saitama, 343, JapanDepartment of Obstetrics and Gynecology, Koshigaya Hospital, Dokkyo University School of Medicine, Minami‐Koshigaya, Koshigaya‐shi, Saitama, 343, JapanDepartment of Obstetrics and Gynecology, Koshigaya Hospital, Dokkyo University School of Medicine, Minami‐Koshigaya, Koshigaya‐shi, Saitama, 343, JapanDepartment of Obstetrics and Gynecology, Koshigaya Hospital, Dokkyo University School of Medicine, Minami‐Koshigaya, Koshigaya‐shi, Saitama, 343, JapanDepartment of Obstetrics and Gynecology, Koshigaya Hospital, Dokkyo University School of Medicine, Minami‐Koshigaya, Koshigaya‐shi, Saitama, 343, JapanBackground. Cisplatin induces nephrotoxicity and this study evaluated the protective effect of fosfomycin on it in 11 gynecological cancer patients. Methods. The N‐acetyl‐β‐D‐glucosaminidase (NAG), β2‐microglobulin (β2MG), creatinine (uCr) and total protein (TP) levels in a 24‐hour urine specimen as well as the blood urea nitrogen (BUN) and serum creatinine (sCr) were measured before and after CAPF chemotherapy alone (control) or with fosfomycin. Results. The results were statistically analyzed by using the t‐test. NAG, β2MG, uCr and TP levels increased significantly after chemotherapy in the control patients, but BUN and sCr levels did not change significantly. The NAG level in the control group was twice as high as in the fosfomycin group 8 days after chemotherapy (p<0.01). The uCr and TP in control patients increased significantly after chemotherapy when compared to those in patients coadministered fosfomycin. There were no significant changes in β2MG, BUN and sCr levels. Conclusions. Cisplatin affected the levels of NAG, β2MG, uCr and TP without influencing BUN and sCr levels. Fosfomycin, therefore, may be useful as a supplemental treatment for reducing cisplatin nephrotoxicity, especially proximal tubular damage.https://doi.org/10.3109/00016349709024590cisplatinfosfomycinnephrotoxicityurinary metabolites
spellingShingle Masatoshi Hayashi
Masahide Numaguchi
Hideki Watabe
Hideo Enomoto
Yoshimasa Yaoi
Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels
Acta Obstetricia et Gynecologica Scandinavica
cisplatin
fosfomycin
nephrotoxicity
urinary metabolites
title Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels
title_full Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels
title_fullStr Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels
title_full_unstemmed Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels
title_short Cisplatin‐induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels
title_sort cisplatin induced nephrotoxicity and the protective effect of fosfomycin on it as demonstrated by using a crossover study of urinary metabolite levels
topic cisplatin
fosfomycin
nephrotoxicity
urinary metabolites
url https://doi.org/10.3109/00016349709024590
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