A crucial role of miR-155 in the pathomechanism of acute kidney injury

Acute kidney injury (AKI) is one of the nonnegligible causes of mortality worldwide. It is important to understand the underlying molecular mechanism of AKI to effective therapeutic targets. miR-155 has been found to play a pivotal role in the development of AKI, while a comprehensive review on this...

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Main Authors: Hui Wang, Jian Liao, Wei Wang, Jianhua Zhang
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-04-01
Series:Frontiers in Pharmacology
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Online Access:https://www.frontiersin.org/articles/10.3389/fphar.2025.1570000/full
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author Hui Wang
Jian Liao
Wei Wang
Jianhua Zhang
author_facet Hui Wang
Jian Liao
Wei Wang
Jianhua Zhang
author_sort Hui Wang
collection DOAJ
description Acute kidney injury (AKI) is one of the nonnegligible causes of mortality worldwide. It is important to understand the underlying molecular mechanism of AKI to effective therapeutic targets. miR-155 has been found to play a pivotal role in the development of AKI, while a comprehensive review on this topic is currently still lacking. Based on this review, we found that miR-155and is strongly correlated with the pathophysiological development of AKI by modulating cell apoptosis, inflammation, and proliferation. Mechanistically, miR-155 exerts a promoting function in multiple types of AKI by regulating multiple proteins or signaling pathways, such as SOCS-1, ERRFI1, SOCS-1, TRF1, CDK12, and TCF4/Wnt/β-catenin pathway. The inhibition of miR-155 has a renoprotective effect in drug- or substance-induced AKI. Therefore, drugs or biological compounds targeted by miR-155 and its pathways may recover the process of AKI by altering apoptosis, inflammation, and pyroptosis. A miRNA nanocarrier system that has already been developed could offer a novel approach to treat AKI, providing a direction for future research. Further large-scale studies are necessary to elucidate the clinical significance of miR-155 as a potential therapeutic target for multiple types of AKI.
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spelling doaj-art-a82bbef50b88404d8b9ea12b3f9215622025-08-20T02:26:24ZengFrontiers Media S.A.Frontiers in Pharmacology1663-98122025-04-011610.3389/fphar.2025.15700001570000A crucial role of miR-155 in the pathomechanism of acute kidney injuryHui Wang0Jian Liao1Wei Wang2Jianhua Zhang3Department of Urology, The First People’s Hospital of Linhai, Linhai, Zhejiang, ChinaDepartment of Nephrology, Jiaxing Hospital of Traditional Chinese Medicine, Jiaxing, ChinaDepartment of Urology, Tiantai People’s Hospital of Zhejiang Province (Tiantai Branch of Zhejiang Provincial People’s Hospital), Hangzhou Medical College, Taizhou, Zhejiang, ChinaDepartment of Urology, Tiantai People’s Hospital of Zhejiang Province (Tiantai Branch of Zhejiang Provincial People’s Hospital), Hangzhou Medical College, Taizhou, Zhejiang, ChinaAcute kidney injury (AKI) is one of the nonnegligible causes of mortality worldwide. It is important to understand the underlying molecular mechanism of AKI to effective therapeutic targets. miR-155 has been found to play a pivotal role in the development of AKI, while a comprehensive review on this topic is currently still lacking. Based on this review, we found that miR-155and is strongly correlated with the pathophysiological development of AKI by modulating cell apoptosis, inflammation, and proliferation. Mechanistically, miR-155 exerts a promoting function in multiple types of AKI by regulating multiple proteins or signaling pathways, such as SOCS-1, ERRFI1, SOCS-1, TRF1, CDK12, and TCF4/Wnt/β-catenin pathway. The inhibition of miR-155 has a renoprotective effect in drug- or substance-induced AKI. Therefore, drugs or biological compounds targeted by miR-155 and its pathways may recover the process of AKI by altering apoptosis, inflammation, and pyroptosis. A miRNA nanocarrier system that has already been developed could offer a novel approach to treat AKI, providing a direction for future research. Further large-scale studies are necessary to elucidate the clinical significance of miR-155 as a potential therapeutic target for multiple types of AKI.https://www.frontiersin.org/articles/10.3389/fphar.2025.1570000/fullAKImir-155mechanismtherapeutic targetsapoptosisinflammation
spellingShingle Hui Wang
Jian Liao
Wei Wang
Jianhua Zhang
A crucial role of miR-155 in the pathomechanism of acute kidney injury
Frontiers in Pharmacology
AKI
mir-155
mechanism
therapeutic targets
apoptosis
inflammation
title A crucial role of miR-155 in the pathomechanism of acute kidney injury
title_full A crucial role of miR-155 in the pathomechanism of acute kidney injury
title_fullStr A crucial role of miR-155 in the pathomechanism of acute kidney injury
title_full_unstemmed A crucial role of miR-155 in the pathomechanism of acute kidney injury
title_short A crucial role of miR-155 in the pathomechanism of acute kidney injury
title_sort crucial role of mir 155 in the pathomechanism of acute kidney injury
topic AKI
mir-155
mechanism
therapeutic targets
apoptosis
inflammation
url https://www.frontiersin.org/articles/10.3389/fphar.2025.1570000/full
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